Saiyan Bian

432 total citations
18 papers, 292 citations indexed

About

Saiyan Bian is a scholar working on Molecular Biology, Cancer Research and Cell Biology. According to data from OpenAlex, Saiyan Bian has authored 18 papers receiving a total of 292 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 6 papers in Cancer Research and 4 papers in Cell Biology. Recurrent topics in Saiyan Bian's work include Ubiquitin and proteasome pathways (7 papers), RNA modifications and cancer (4 papers) and Cancer-related Molecular Pathways (3 papers). Saiyan Bian is often cited by papers focused on Ubiquitin and proteasome pathways (7 papers), RNA modifications and cancer (4 papers) and Cancer-related Molecular Pathways (3 papers). Saiyan Bian collaborates with scholars based in China and United States. Saiyan Bian's co-authors include Wenkai Ni, Mengqi Zhu, Mingbing Xiao, Qianqian Song, Jianping Zhang, Wenjie Zheng, Wenjie Zheng, Runzhou Ni, Lishuai Qu and Yihui Fan and has published in prestigious journals such as Cell Death and Differentiation, International Journal of Biological Macromolecules and Cell Death and Disease.

In The Last Decade

Saiyan Bian

15 papers receiving 290 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Saiyan Bian China 10 231 109 51 45 38 18 292
Sang Yean Kim South Korea 9 218 0.9× 134 1.2× 34 0.7× 46 1.0× 32 0.8× 12 297
Feiye Liu China 9 213 0.9× 148 1.4× 44 0.9× 23 0.5× 29 0.8× 13 294
Juan L López-Cánovas Spain 9 182 0.8× 81 0.7× 24 0.5× 26 0.6× 36 0.9× 13 250
Qiangnu Zhang China 12 277 1.2× 217 2.0× 50 1.0× 65 1.4× 35 0.9× 24 372
Zixi Liang China 7 304 1.3× 218 2.0× 93 1.8× 29 0.6× 19 0.5× 15 412
Miljana Nenkov Germany 10 171 0.7× 125 1.1× 64 1.3× 53 1.2× 20 0.5× 12 267
Zining Jin China 11 186 0.8× 136 1.2× 74 1.5× 61 1.4× 18 0.5× 28 309
Yaozhen Pan China 7 176 0.8× 92 0.8× 91 1.8× 44 1.0× 24 0.6× 22 284
Xiwu Ouyang China 12 247 1.1× 173 1.6× 37 0.7× 50 1.1× 23 0.6× 16 321
Min Cheng China 10 218 0.9× 209 1.9× 47 0.9× 54 1.2× 26 0.7× 19 326

Countries citing papers authored by Saiyan Bian

Since Specialization
Citations

This map shows the geographic impact of Saiyan Bian's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Saiyan Bian with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Saiyan Bian more than expected).

Fields of papers citing papers by Saiyan Bian

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Saiyan Bian. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Saiyan Bian. The network helps show where Saiyan Bian may publish in the future.

Co-authorship network of co-authors of Saiyan Bian

This figure shows the co-authorship network connecting the top 25 collaborators of Saiyan Bian. A scholar is included among the top collaborators of Saiyan Bian based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Saiyan Bian. Saiyan Bian is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
2.
Xu, Nuo, Saiyan Bian, Pin Lyu, Xinlin He, & Wenjie Zheng. (2025). Dynamic interplay of neuroendocrine signaling and immuno-surveillance in tumor niche remodeling. Critical Reviews in Oncology/Hematology. 216. 104958–104958.
3.
Xu, Nuo, et al.. (2024). Ubiquitination-specific protease 7 enhances stemness of hepatocellular carcinoma by stabilizing basic transcription factor 3. Functional & Integrative Genomics. 24(1). 28–28. 1 indexed citations
4.
Bian, Saiyan, Wenkai Ni, Xuying Zhao, et al.. (2024). Ubiquitin-specific protease 1 facilitates hepatocellular carcinoma progression by modulating mitochondrial fission and metabolic reprogramming via cyclin-dependent kinase 5 stabilization. Cell Death and Differentiation. 31(9). 1202–1218. 10 indexed citations
5.
Zhang, Xue, Saiyan Bian, Yao Ni, et al.. (2023). Minichromosome maintenance protein family member 6 mediates hepatocellular carcinoma progression by recruiting UBE3A to induce P53 ubiquitination. International Journal of Biological Macromolecules. 248. 125854–125854. 4 indexed citations
6.
Zheng, Wenjie, Saiyan Bian, Colin E. Bishop, et al.. (2023). Placenta mesenchymal stem cell-derived extracellular vesicles alleviate liver fibrosis by inactivating hepatic stellate cells through a miR-378c/SKP2 axis. Inflammation and Regeneration. 43(1). 47–47. 20 indexed citations
7.
Xu, Nuo, et al.. (2023). Identifying Dental Pulp Stem Cell as a Novel Therapeutic trategy for Digestive Diseases. Current Stem Cell Research & Therapy. 19(10). 1293–1302.
8.
Bian, Saiyan, Hui Wang, Jiahang Mo, et al.. (2022). CRMP2 derived from cancer associated fibroblasts facilitates progression of ovarian cancer via HIF-1α-glycolysis signaling pathway. Cell Death and Disease. 13(8). 675–675. 22 indexed citations
9.
Zhu, Mengqi, Jianping Zhang, Saiyan Bian, et al.. (2022). Circadian gene CSNK1D promoted the progression of hepatocellular carcinoma by activating Wnt/β-catenin pathway via stabilizing Dishevelled Segment Polarity Protein 3. Biological Procedures Online. 24(1). 21–21. 9 indexed citations
10.
Bian, Saiyan, Wenkai Ni, Mengqi Zhu, et al.. (2022). Flap endonuclease 1 Facilitated Hepatocellular Carcinoma Progression by Enhancing USP7/MDM2-mediated P53 Inactivation. International Journal of Biological Sciences. 18(3). 1022–1038. 38 indexed citations
11.
Ni, Wenkai, Saiyan Bian, Mengqi Zhu, et al.. (2021). Identification and Validation of Ubiquitin-Specific Proteases as a Novel Prognostic Signature for Hepatocellular Carcinoma. Frontiers in Oncology. 11. 629327–629327. 15 indexed citations
12.
Zhu, Mengqi, Mengna Wu, Saiyan Bian, et al.. (2021). DNA primase subunit 1 deteriorated progression of hepatocellular carcinoma by activating AKT/mTOR signaling and UBE2C-mediated P53 ubiquitination. Cell & Bioscience. 11(1). 42–42. 36 indexed citations
13.
14.
Zhou, Bin, et al.. (2021). The lncRNA B3GALT5-AS1 Functions as an HCC Suppressor by Regulating the miR-934/UFM1 Axis. Journal of Oncology. 2021. 1–18. 10 indexed citations
15.
Zhang, Wei, Jingxin Zhang, Shiqing Zhang, et al.. (2020). Ubiquitin-specific protease 7 is a drug-able target that promotes hepatocellular carcinoma and chemoresistance. Cancer Cell International. 20(1). 28–28. 33 indexed citations
16.
Bian, Saiyan, Wenkai Ni, Mengqi Zhu, et al.. (2020). Identification and Validation of the N6-Methyladenosine RNA Methylation Regulator YTHDF1 as a Novel Prognostic Marker and Potential Target for Hepatocellular Carcinoma. Frontiers in Molecular Biosciences. 7. 604766–604766. 49 indexed citations
17.
Zhu, Mengqi, Yuting Huang, Saiyan Bian, et al.. (2020). Organoids: Current Implications and Pharmaceutical Applications in Liver Diseases. Current Molecular Pharmacology. 14(4). 498–508. 2 indexed citations
18.
Ni, Wenkai, Shengli Lin, Saiyan Bian, et al.. (2020). USP7 mediates pathological hepatic de novo lipogenesis through promoting stabilization and transcription of ZNF638. Cell Death and Disease. 11(10). 843–843. 36 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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