S. Rafael

514 total citations
17 papers, 431 citations indexed

About

S. Rafael is a scholar working on Oncology, Molecular Biology and Pathology and Forensic Medicine. According to data from OpenAlex, S. Rafael has authored 17 papers receiving a total of 431 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Oncology, 5 papers in Molecular Biology and 5 papers in Pathology and Forensic Medicine. Recurrent topics in S. Rafael's work include Cancer Cells and Metastasis (7 papers), Genetic factors in colorectal cancer (5 papers) and Cancer Genomics and Diagnostics (4 papers). S. Rafael is often cited by papers focused on Cancer Cells and Metastasis (7 papers), Genetic factors in colorectal cancer (5 papers) and Cancer Genomics and Diagnostics (4 papers). S. Rafael collaborates with scholars based in Spain. S. Rafael's co-authors include Marta Vidaurreta, S. Veganzones, Manuel Arroyo, María Teresa Sanz Casla, Eduardo Díaz‐Rubio, Javier Sastre, José Á. García-Sáenz, Miguel Martín, Rosario Alfonso and M.L. Maestro and has published in prestigious journals such as Journal of Clinical Oncology, Annals of Oncology and Annals of Surgical Oncology.

In The Last Decade

S. Rafael

16 papers receiving 420 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
S. Rafael Spain 10 331 184 126 101 87 17 431
Albin Rudisch Austria 6 151 0.5× 90 0.5× 42 0.3× 31 0.3× 178 2.0× 7 325
Stéphane Pouderoux France 12 249 0.8× 139 0.8× 133 1.1× 70 0.7× 94 1.1× 31 403
María Teresa Sanz Casla Spain 10 301 0.9× 154 0.8× 107 0.8× 110 1.1× 119 1.4× 23 425
Zongming Lin China 12 134 0.4× 99 0.5× 132 1.0× 25 0.2× 226 2.6× 19 396
J. H. Finke United States 6 175 0.5× 145 0.8× 65 0.5× 22 0.2× 97 1.1× 9 395
Roger Stenling Sweden 8 233 0.7× 98 0.5× 165 1.3× 93 0.9× 210 2.4× 8 474
Valeria Pusceddu Italy 12 238 0.7× 103 0.6× 72 0.6× 62 0.6× 135 1.6× 45 382
Richard Wharton United Kingdom 7 319 1.0× 138 0.8× 77 0.6× 89 0.9× 67 0.8× 11 389
C. Rimkus Germany 5 148 0.4× 55 0.3× 66 0.5× 54 0.5× 169 1.9× 10 365
Ya-Chin Hou Taiwan 8 175 0.5× 104 0.6× 48 0.4× 9 0.1× 173 2.0× 10 393

Countries citing papers authored by S. Rafael

Since Specialization
Citations

This map shows the geographic impact of S. Rafael's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by S. Rafael with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites S. Rafael more than expected).

Fields of papers citing papers by S. Rafael

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by S. Rafael. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by S. Rafael. The network helps show where S. Rafael may publish in the future.

Co-authorship network of co-authors of S. Rafael

This figure shows the co-authorship network connecting the top 25 collaborators of S. Rafael. A scholar is included among the top collaborators of S. Rafael based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with S. Rafael. S. Rafael is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Veganzones, S., M. L. Maestro, S. Rafael, et al.. (2015). Combined methylation of p16 and hMLH1 (CMETH2) discriminates a subpopulation with better prognosis in colorectal cancer patients with microsatellite instability tumors. Tumor Biology. 36(5). 3853–3861. 12 indexed citations
2.
Rafael, S., et al.. (2014). Effect of β-catenin alterations in the prognosis of patients with sporadic colorectal cancer. Journal of Cancer Research and Therapeutics. 10(3). 591–591. 4 indexed citations
3.
Rafael, S., et al.. (2013). A9 region in EPHB2 mutation is frequent in tumors with microsatellite instability. Analysis of prognosis.. PubMed. 33(11). 5159–63. 2 indexed citations
4.
Sánchez‐Fructuoso, Ana, M. L. Maestro, Isabel Pérez-Flores, et al.. (2012). Serum level of fibroblast growth factor 23 in maintenance renal transplant patients. Nephrology Dialysis Transplantation. 27(11). 4227–4235. 28 indexed citations
5.
Vidaurreta, Marta, S. Rafael, S. Veganzones, et al.. (2010). Influence of A9 Region Mutation in EphB2 Gene in the Prognosis of Patients with Colorectal Adenocarcinoma. Annals of Surgical Oncology. 18(5). 1501–1505. 2 indexed citations
6.
Vidaurreta, Marta, S. Veganzones, S. Rafael, et al.. (2010). Vascular endothelial growth factor gene polymorphisms in patients with colorectal cancer. Revista Española de Enfermedades Digestivas. 102(1). 20–31. 21 indexed citations
7.
García-Sáenz, José Á., Miguel Martín, M. L. Maestro, et al.. (2009). Circulating tumour cells in locally advanced breast cancer. Clinical & Translational Oncology. 11(8). 544–547. 10 indexed citations
8.
Resel, Luis, Marta Vidaurreta, S. Veganzones, et al.. (2009). 902 CIRCULATING TUMOR CELLS (CTC) IN THE THREE TUMOR TYPES OF EPITHELIAL ORIGIN: PROSTATE, BREAST AND COLORECTAL, IN PATIENTS WITH AND WITHOUT METASTASIS. European Urology Supplements. 8(4). 346–346.
9.
Sastre, Javier, S. Rafael, S. Veganzones, et al.. (2009). Circulating tumor cells in solid tumor in metastatic and localized stages.. PubMed. 29(11). 4839–43. 69 indexed citations
10.
García-Sáenz, José Á., Miguel Martín, M. L. Maestro, et al.. (2009). Circulating tumor cells in locally advanced breast cancer. Journal of Clinical Oncology. 27(15_suppl). e22159–e22159. 1 indexed citations
11.
Sastre, Javier, M.L. Maestro, Javier Puente, et al.. (2008). Circulating tumor cells in colorectal cancer: correlation with clinical and pathological variables. Annals of Oncology. 19(5). 935–938. 185 indexed citations
12.
Vidaurreta, Marta, M. L. Maestro, María Teresa Sanz Casla, et al.. (2008). Colorectal carcinoma prognosis can be predicted by alterations in gene p53 exons 5 and 8. International Journal of Colorectal Disease. 23(6). 581–586. 17 indexed citations
13.
Vidaurreta, Marta, Javier Sastre, María Teresa Sanz Casla, et al.. (2007). Detección y cuantificación de células tumorales en sangre periférica en pacientes con cáncer de colon. Medicina Clínica. 129(9). 333–334. 7 indexed citations
14.
Vidaurreta, Marta, M.L. Maestro, María Teresa Sanz Casla, et al.. (2007). Inactivation of p16 by CpG hypermethylation in renal cell carcinoma. Urologic Oncology Seminars and Original Investigations. 26(3). 239–245. 19 indexed citations
15.
García-Sáenz, José Á., Miguel Martín, M.L. Maestro, et al.. (2006). Circulating tumoral cells lack circadian-rhythm in hospitalized metastasic breast cancer patients. Clinical & Translational Oncology. 8(11). 826–829. 9 indexed citations
16.
Maestro, M. L., Marta Vidaurreta, María Teresa Sanz Casla, et al.. (2006). Role of the BRAF Mutations in the Microsatellite Instability Genetic Pathway in Sporadic Colorectal Cancer. Annals of Surgical Oncology. 14(3). 1229–1236. 43 indexed citations
17.
Vidaurreta, Marta, María Teresa Sanz Casla, M. L. Maestro, et al.. (2005). La inestabilidad de microsatélites predice un mejor pronóstico en los pacientes intervenidos de carcinoma colorrectal. Medicina Clínica. 124(4). 121–125. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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