Rotraud Wieser

7.4k total citations · 3 hit papers
63 papers, 6.0k citations indexed

About

Rotraud Wieser is a scholar working on Molecular Biology, Hematology and Genetics. According to data from OpenAlex, Rotraud Wieser has authored 63 papers receiving a total of 6.0k indexed citations (citations by other indexed papers that have themselves been cited), including 51 papers in Molecular Biology, 31 papers in Hematology and 10 papers in Genetics. Recurrent topics in Rotraud Wieser's work include Acute Myeloid Leukemia Research (28 papers), TGF-β signaling in diseases (10 papers) and Retinoids in leukemia and cellular processes (8 papers). Rotraud Wieser is often cited by papers focused on Acute Myeloid Leukemia Research (28 papers), TGF-β signaling in diseases (10 papers) and Retinoids in leukemia and cellular processes (8 papers). Rotraud Wieser collaborates with scholars based in Austria, United States and Germany. Rotraud Wieser's co-authors include Joan Massagué, Jeffrey L. Wrana, Liliana Attisano, Francesc Ventura, Hongyan Zou, Lee Niswander, G R Mundy, Katri S. Selander, Mark Dallas and Juan Juan Yin and has published in prestigious journals such as Nature, Journal of Biological Chemistry and Journal of Clinical Investigation.

In The Last Decade

Rotraud Wieser

62 papers receiving 5.9k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Mechanism of activation of the TGF-β receptor

1994 2.0k citations Jeffrey L. Wrana, Liliana Attisano et al. profile →
TGF-β signaling blockade inhibits PTHrP secretion by breast cancer cells and bone metastases development

1999 766 citations Rotraud Wieser, Joan Massagué et al. profile →
GS domain mutations that constitutively activate T beta R-I, the downstream signaling component in the TGF-beta receptor complex.

1995 596 citations Rotraud Wieser, Jeffrey L. Wrana et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Rotraud Wieser Austria	26	4.8k	1.6k	689	546	498	63	6.0k
Dennis K. Watson United States	41	3.6k 0.7×	1.3k 0.8×	591 0.9×	1.0k 1.9×	567 1.1×	106	5.8k
Carl‐Henrik Heldin Sweden	23	4.0k 0.8×	1.3k 0.8×	274 0.4×	664 1.2×	417 0.8×	29	5.7k
Minhong Yan United States	33	3.9k 0.8×	1.4k 0.9×	346 0.5×	1.5k 2.8×	363 0.7×	51	7.6k
Florian Otto Germany	24	3.1k 0.6×	1.4k 0.9×	269 0.4×	814 1.5×	581 1.2×	35	4.9k
Kosei Ito Japan	37	3.6k 0.7×	1.3k 0.8×	478 0.7×	1.1k 2.0×	448 0.9×	94	5.0k
Agnieszka E. Gorska United States	31	3.2k 0.7×	3.0k 1.9×	284 0.4×	799 1.5×	278 0.6×	48	5.5k
Kevin D. Bunting United States	39	3.0k 0.6×	3.0k 1.9×	1.2k 1.7×	811 1.5×	490 1.0×	111	6.5k
Reuben Kapur United States	41	2.8k 0.6×	932 0.6×	1.3k 1.9×	568 1.0×	299 0.6×	172	6.0k
D Delia Italy	31	2.5k 0.5×	1.1k 0.7×	729 1.1×	430 0.8×	312 0.6×	62	4.6k
Georges Uzan France	36	2.6k 0.5×	629 0.4×	1.1k 1.6×	539 1.0×	270 0.5×	134	5.0k

Countries citing papers authored by Rotraud Wieser

Since Specialization

Citations

This map shows the geographic impact of Rotraud Wieser's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Rotraud Wieser with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Rotraud Wieser more than expected).

Fields of papers citing papers by Rotraud Wieser

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Rotraud Wieser. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Rotraud Wieser. The network helps show where Rotraud Wieser may publish in the future.

Co-authorship network of co-authors of Rotraud Wieser

This figure shows the co-authorship network connecting the top 25 collaborators of Rotraud Wieser. A scholar is included among the top collaborators of Rotraud Wieser based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Rotraud Wieser. Rotraud Wieser is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Witalisz‐Siepracka, Agnieszka, Thomas Eder, Omar Sharif, et al.. (2024). A novel function of STAT3β in suppressing interferon response improves outcome in acute myeloid leukemia. Cell Death and Disease. 15(5). 369–369. 6 indexed citations

Nguyen, Chi Huu, Hubert Hackl, Heinz Sill, et al.. (2021). Downregulation of MTSS1 in acute myeloid leukemia is associated with a poor prognosis, chemotherapy resistance, and disease aggressiveness. Leukemia. 35(10). 2827–2839. 13 indexed citations

Nguyen, Chi Huu, et al.. (2020). All-trans retinoic acid in non-promyelocytic acute myeloid leukemia: driver lesion dependent effects on leukemic stem cells. Cell Cycle. 19(20). 2573–2588. 13 indexed citations

Nguyen, Chi Huu, Elisabeth Koller, Emiel van der Kouwe, et al.. (2020). IL2RA Promotes Aggressiveness and Stem Cell–Related Properties of Acute Myeloid Leukemia. Cancer Research. 80(20). 4527–4539. 23 indexed citations

Nguyen, Chi Huu, et al.. (2020). Evi1 Counteracts Anti-Leukemic and Stem Cell Inhibitory Effects of All-Trans Retinoic Acid on Flt3-ITD/Npm1c-Driven Acute Myeloid Leukemia Cells. Biomedicines. 8(10). 385–385. 4 indexed citations

Lee, Anne W.M., Sai Wah Tsao, Kwok Wai Lo, et al.. (2020). Role of miR-96/EVI1/miR-449a Axis in the Nasopharyngeal Carcinoma Cell Migration and Tumor Sphere Formation. International Journal of Molecular Sciences. 21(15). 5495–5495. 9 indexed citations

Nguyen, Chi Huu, Katharina Bauer, Hubert Hackl, et al.. (2019). SOCS2 is part of a highly prognostic 4-gene signature in AML and promotes disease aggressiveness. Scientific Reports. 9(1). 9139–9139. 36 indexed citations

Hatzl, Stefan, Rotraud Wieser, Martin Pichler, et al.. (2016). Increased Expression of miR-23a Mediates a Loss of Expression in the RAF Kinase Inhibitor Protein RKIP. Cancer Research. 76(12). 3644–3654. 39 indexed citations

Heller, Gerwin, Julia Etzler, Hubert Hackl, et al.. (2015). EVI1 promotes tumor growth via transcriptional repression of MS4A3. Journal of Hematology & Oncology. 8(1). 28–28. 24 indexed citations

10.

Hackl, Hubert, Christine Schneckenleithner, Marcel Scheideler, et al.. (2013). Overexpression of primary microRNA 221/222 in acute myeloid leukemia. BMC Cancer. 13(1). 364–364. 43 indexed citations

11.

Hackl, Hubert, et al.. (2010). Tetracycline Regulator Expression Alters the Transcriptional Program of Mammalian Cells. PLoS ONE. 5(9). e13013–e13013. 6 indexed citations

12.

Kundi, Michael, Wolfgang R. Sperr, Harald Esterbauer, et al.. (2008). Expression and prognostic significance of different mRNA 5′‐end variants of the oncogene EVI1 in 266 patients with de novo AML: EVI1 and MDS1/EVI1 overexpression both predict short remission duration. Genes Chromosomes and Cancer. 47(4). 288–298. 52 indexed citations

13.

Wieser, Rotraud. (2005). Cystic fibrosis – the most frequent life-shortening autosomal recessive disease. Wiener Medizinische Wochenschrift. 155(11-12). 268–272. 3 indexed citations

14.

Aytekin, Metin, Ursula Vinatzer, Mónica Musteanu, Sophie Raynaud, & Rotraud Wieser. (2005). Regulation of the expression of the oncogene EVI1 through the use of alternative mRNA 5′-ends. Gene. 356. 160–168. 31 indexed citations

15.

Kaufmann, H., J Ackermann, Thomas Nösslinger, et al.. (2004). Both IGH translocations and chromosome 13q deletions are early events in monoclonal gammopathy of undetermined significance and do not evolve during transition to multiple myeloma. Leukemia. 18(11). 1879–1882. 59 indexed citations

16.

Wimmer, Katharina, et al.. (1998). Comparative Expression Analysis of the Antagonistic Transcription Factors EVI1 and MDS1-EVI1 in Murine Tissues and duringin VitroHematopoietic Differentiation. Biochemical and Biophysical Research Communications. 252(3). 691–696. 16 indexed citations

17.

Wieser, Rotraud, Beata Gruber, Harald Rieder, & C. Fonatsch. (1998). Mutational analysis of the tumor suppressor Smad2 in acute lymphoid and myeloid leukemia. Leukemia. 12(7). 1114–1118. 7 indexed citations

18.

Zou, Hongyan, Rotraud Wieser, Joan Massagué, & Lee Niswander. (1997). Distinct roles of type I bone morphogenetic protein receptors in the formation and differentiation of cartilage. Genes & Development. 11(17). 2191–2203. 440 indexed citations

19.

Cárcamo, Juan M., Francesc Ventura, Rotraud Wieser, et al.. (1994). Type I receptors specify growth-inhibitory and transcriptional responses to transforming growth factor beta and activin.. Molecular and Cellular Biology. 14(6). 3810–3821. 335 indexed citations

20.

Wieser, Rotraud, Gerhard Adam, Christoph Schüller, et al.. (1991). Heat shock factor-independent heat control of transcription of the CTT1 gene encoding the cytosolic catalase T of Saccharomyces cerevisiae. Journal of Biological Chemistry. 266(19). 12406–12411. 124 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact