Ronald E. Cannon

1.8k citations
49 papers · 1.5k indexed · h-index 25

Ronald E. Cannon

49 papers receiving 1.5k citations

Peers

Ronald E. Cannon
Comparison fields: 5 of 107
  • Neurology 132
  • Oncology 422
  • Molecular Biology 783
  • Health, Toxicology and Mutagenesis 156
  • Cancer Research 156
Replace Michael C. Velarde with:
Michael C. Velarde United States
Yung‐Feng Lin Taiwan
Françoise Miot Belgium
Keisuke Yamashita Japan
Lun Song China
Sandy Schwemberger United States
Rong Yu China
José Martín‐Nieto Spain
Rikio Tokunaga Japan
Pearl P.Y. Lie United States
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Citations per field
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Citations per year

Countries citing papers authored by Ronald E. Cannon

Since Specialization
Citations

This map shows the geographic impact of Ronald E. Cannon's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ronald E. Cannon with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ronald E. Cannon more than expected).

Fields of papers citing papers by Ronald E. Cannon

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ronald E. Cannon. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ronald E. Cannon. The network helps show where Ronald E. Cannon may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Ronald E. Cannon, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Ronald E. Cannon Line = papers co-authored together Ronald E. Cannon links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 20242
2 202313
3 202034
4 201920
5 201724
6 201633
7 201419
8 2014133
9 200988
10 200837
11 20065
12 200428
13 200318
14 200331
15 20019
16 20005
17 200015
18 199814
19 199832
20 199740

About Ronald E. Cannon

Ronald E. Cannon is a scholar working on Oncology, Neurology, Molecular Biology, Cancer Research and Nutrition and Dietetics, having authored 49 papers that have together received 1.5k indexed citations. Recurring topics across this work include Drug Transport and Resistance Mechanisms (18 papers), Virus-based gene therapy research (6 papers), Trace Elements in Health (5 papers), Sphingolipid Metabolism and Signaling (4 papers), Barrier Structure and Function Studies (4 papers), Pharmacological Effects and Toxicity Studies (4 papers), Cancer therapeutics and mechanisms (3 papers) and Pregnancy and Medication Impact (3 papers). The work is most often cited by research in Neurology (132 citations), Oncology (422 citations), Molecular Biology (783 citations), Health, Toxicology and Mutagenesis (156 citations) and Cancer Research (156 citations). Ronald E. Cannon has collaborated with scholars based in United States, Australia and Canada. Frequent co-authors include David S. Miller, Christopher Campos, John G. Scandalios, Raymond W. Tennant, Guang‐Jer Wu, Carol S. Trempus, Birandra K. Sinha, Joseph White, Brian T. Hawkins and Lindsay K Smith. Their work appears in journals such as Molecular Carcinogenesis, Journal of Cerebral Blood Flow & Metabolism, Proceedings of the National Academy of Sciences, Toxicological Sciences and Environmental Health Perspectives.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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