Ron Skurray
Impact in
- Molecular Medicine top 5%
- Antibiotic Resistance in Bacteria
- Endocrinology top 5%
Papers in
-
- DNA Repair Mechanisms 6
- RNA modifications and cancer 4
- DNA and Nucleic Acid Chemistry 3
- Genetics 11
- Bacterial Genetics and Biotechnology 11
- Co-authors
- Neil Willetts (2 shared papers)Animesh Ray (8 shared papers)David S. Cram (6 shared papers)Neville Firth (2 shared papers)U. Rahmsdorf (1 shared paper)Nicholas A. Kennedy (1 shared paper)Lothar Beutin (1 shared paper)Peter Herrlich (1 shared paper)
- Journals
- Gene (2 papers)Journal of Molecular Biology (1 paper)Annual Review of Genetics (1 paper)Nature (1 paper)Plasmid (8 papers)
- Partner nations
- AustraliaUnited KingdomGermany
In The Last Decade
Ron Skurray
18 papers receiving 613 citations
Peers
Comparison fields: 5 of 58
- Molecular Medicine 144
- Endocrinology 100
- Genetics 423
- Ecology 245
- Molecular Biology 436
Countries citing papers authored by Ron Skurray
This map shows the geographic impact of Ron Skurray's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ron Skurray with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ron Skurray more than expected).
Fields of papers citing papers by Ron Skurray
This network shows the impact of papers produced by Ron Skurray. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ron Skurray. The network helps show where Ron Skurray may publish in the future.
Co-authors
The 13 scholars most cited alongside Ron Skurray, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 1980 | 246 | |
| 2 | 1977 | 140 | |
| 3 | 1983 | 68 | |
| 4 | 1992 | 48 | |
| 5 | 1983 | 39 | |
| 6 | 1989 | 25 | |
| 7 | 1984 | 20 | |
| 8 | 1989 | 16 | |
| 9 | 1984 | 16 | |
| 10 | 1984 | 16 | |
| 11 | 1986 | 15 | |
| 12 | 1989 | 14 | |
| 13 | 1984 | 11 | |
| 14 | 1984 | 11 | |
| 15 | 1984 | 10 | |
| 16 | 1989 | 8 | |
| 17 | 1986 | 6 | |
| 18 | 1987 | 2 |
About Ron Skurray
Ron Skurray is a scholar working on Molecular Biology, Genetics, Ecology, Molecular Medicine and Plant Science, having authored 18 papers that have together received 711 indexed citations. Recurring topics across this work include Bacterial Genetics and Biotechnology (11 papers), Bacteriophages and microbial interactions (7 papers), DNA Repair Mechanisms (6 papers), RNA modifications and cancer (4 papers), DNA and Nucleic Acid Chemistry (3 papers), Legume Nitrogen Fixing Symbiosis (3 papers), Antibiotic Resistance in Bacteria (3 papers) and Plant Micronutrient Interactions and Effects (2 papers). The work is most often cited by research in Molecular Medicine (144 citations), Endocrinology (100 citations), Genetics (423 citations), Ecology (245 citations) and Molecular Biology (436 citations). Ron Skurray has collaborated with scholars based in Australia, United Kingdom and Germany. Frequent co-authors include Neil Willetts, Animesh Ray, David S. Cram, Neville Firth, U. Rahmsdorf, Nicholas A. Kennedy, Lothar Beutin, Peter Herrlich, Mark Achtman and Dairena Gaffney. Their work appears in journals such as Gene, Journal of Molecular Biology, Annual Review of Genetics, Nature and Plasmid.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.