Rolf A.K. Stahl

16.6k total citations · 2 hit papers
257 papers, 12.2k citations indexed

About

Rolf A.K. Stahl is a scholar working on Nephrology, Immunology and Molecular Biology. According to data from OpenAlex, Rolf A.K. Stahl has authored 257 papers receiving a total of 12.2k indexed citations (citations by other indexed papers that have themselves been cited), including 106 papers in Nephrology, 67 papers in Immunology and 59 papers in Molecular Biology. Recurrent topics in Rolf A.K. Stahl's work include Renal Diseases and Glomerulopathies (84 papers), Renin-Angiotensin System Studies (33 papers) and Chronic Kidney Disease and Diabetes (32 papers). Rolf A.K. Stahl is often cited by papers focused on Renal Diseases and Glomerulopathies (84 papers), Renin-Angiotensin System Studies (33 papers) and Chronic Kidney Disease and Diabetes (32 papers). Rolf A.K. Stahl collaborates with scholars based in Germany, United States and Switzerland. Rolf A.K. Stahl's co-authors include Günter Wolf, Friedrich Thaiss, Gunther Zahner, Elion Hoxha, Udo Helmchen, Ulf Panzer, Fuad N. Ziyadeh, Ulrich Wenzel, Oliver M. Steinmetz and Günter Wolf and has published in prestigious journals such as New England Journal of Medicine, The Lancet and Journal of Biological Chemistry.

In The Last Decade

Rolf A.K. Stahl

252 papers receiving 12.0k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Mechanisms Underlying Endothelial Dysfunction in Diabetes Mellitus

2001 965 citations Friedrich Thaiss, Rolf A.K. Stahl et al. profile →
Thrombospondin Type-1 Domain-Containing 7A in Idiopathic Membranous Nephropathy

2014 641 citations Nicola M. Tomas, Laurence H. Beck et al. New England Journal of Medicine profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Rolf A.K. Stahl Germany	59	4.8k	3.4k	2.9k	1.9k	1.7k	257	12.2k
David J. Nikolic‐Paterson Australia	70	6.0k 1.3×	4.7k 1.4×	5.9k 2.1×	2.2k 1.2×	1.1k 0.6×	244	17.6k
Carla Zoja Italy	69	4.8k 1.0×	2.4k 0.7×	3.4k 1.2×	1.2k 0.6×	2.4k 1.4×	192	13.3k
George F. Schreiner United States	56	3.1k 0.6×	1.5k 0.4×	4.2k 1.5×	1.4k 0.7×	2.2k 1.3×	139	11.3k
Jeremy Hughes United Kingdom	45	3.0k 0.6×	2.3k 0.7×	2.8k 1.0×	1.3k 0.7×	464 0.3×	112	8.8k
Yusuke Suzuki Japan	45	3.9k 0.8×	2.0k 0.6×	1.7k 0.6×	849 0.4×	1.1k 0.6×	320	8.0k
Peter W. Mathieson United Kingdom	53	5.1k 1.1×	1.9k 0.5×	3.1k 1.1×	1.1k 0.6×	464 0.3×	178	9.4k
Xiao‐Ming Meng China	58	3.2k 0.7×	2.0k 0.6×	8.1k 2.8×	1.8k 0.9×	773 0.4×	256	15.3k
Clemens D. Cohen Germany	57	3.7k 0.8×	2.1k 0.6×	3.7k 1.3×	1.0k 0.5×	424 0.2×	146	9.8k
Emile de Heer Netherlands	47	2.6k 0.5×	1.3k 0.4×	2.9k 1.0×	1.1k 0.6×	451 0.3×	204	8.5k
Hirokazu Okada Japan	36	2.8k 0.6×	956 0.3×	3.4k 1.2×	1.7k 0.9×	1.2k 0.7×	220	8.9k

Countries citing papers authored by Rolf A.K. Stahl

Since Specialization

Citations

This map shows the geographic impact of Rolf A.K. Stahl's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Rolf A.K. Stahl with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Rolf A.K. Stahl more than expected).

Fields of papers citing papers by Rolf A.K. Stahl

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Rolf A.K. Stahl. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Rolf A.K. Stahl. The network helps show where Rolf A.K. Stahl may publish in the future.

Co-authorship network of co-authors of Rolf A.K. Stahl

This figure shows the co-authorship network connecting the top 25 collaborators of Rolf A.K. Stahl. A scholar is included among the top collaborators of Rolf A.K. Stahl based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Rolf A.K. Stahl. Rolf A.K. Stahl is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Reinhard, Linda, et al.. (2021). Netrin G1 Is a Novel Target Antigen in Membranous Nephropathy. Journal of the American Society of Nephrology. 32(10S). 463–463. 2 indexed citations

Rovin, Brad H., Sharon G. Adler, Elion Hoxha, et al.. (2021). Felzartamab in Patients with Anti-Phospholipase A2 Receptor Autoantibody Positive (Anti-PLA2R+) Membranous Nephropathy (MN): Interim Results from the M-PLACE Study. Journal of the American Society of Nephrology. 32(10S). 513–514. 1 indexed citations

Nosko, Anna, Malte A. Kluger, Claudia Wegscheid, et al.. (2018). IL-10 Receptor Signaling Empowers Regulatory T Cells to Control Th17 Responses and Protect from GN. Journal of the American Society of Nephrology. 29(7). 1825–1837. 43 indexed citations

Wiech, Thorsten, Ulrich Wenzel, Marlies Sachs, et al.. (2017). Ubiquitin C-Terminal Hydrolase L1 is required for regulated protein degradation through the ubiquitin proteasome system in kidney. Kidney International. 93(1). 110–127. 27 indexed citations

Hoxha, Elion, Laurence H. Beck, Thorsten Wiech, et al.. (2016). An Indirect Immunofluorescence Method Facilitates Detection of Thrombospondin Type 1 Domain–Containing 7A–Specific Antibodies in Membranous Nephropathy. Journal of the American Society of Nephrology. 28(2). 520–531. 152 indexed citations

Krebs, Christian F., Jan‐Eric Turner, Hans‐Joachim Paust, et al.. (2016). Plasticity of Th17 Cells in Autoimmune Kidney Diseases. The Journal of Immunology. 197(2). 449–457. 28 indexed citations

Krebs, Christian F., Sascha Lange, Catherine Meyer‐Schwesinger, et al.. (2013). Deficiency of the Interleukin 17/23 Axis Accelerates Renal Injury in Mice With Deoxycorticosterone Acetate+Angiotensin II–Induced Hypertension. Hypertension. 63(3). 565–571. 76 indexed citations

Kluger, Malte A., Gunther Zahner, Hans‐Joachim Paust, et al.. (2013). Leukocyte-derived MMP9 is crucial for the recruitment of proinflammatory macrophages in experimental glomerulonephritis. Kidney International. 83(5). 865–877. 38 indexed citations

Stahl, Rolf A.K., et al.. (2010). Stenting versus Endarterectomy for Treatment of Carotid-Artery Stenosis. New England Journal of Medicine. 363(5). 498–498. 42 indexed citations

10.

Turner, Jan‐Eric, Hans‐Joachim Paust, Oliver M. Steinmetz, et al.. (2008). CCR5 Deficiency Aggravates Crescentic Glomerulonephritis in Mice. The Journal of Immunology. 181(9). 6546–6556. 45 indexed citations

11.

Nabah, Yafa Naim Abu, May Abu‐Taha, Manuel Mata, et al.. (2006). Angiotensin II-Induced Mononuclear Leukocyte Interactions with Arteriolar and Venular Endothelium Are Mediated by the Release of Different CC Chemokines. The Journal of Immunology. 176(9). 5577–5586. 88 indexed citations

12.

Wenzel, Ulrich, Matthias Janneck, Christoph Weber, Rolf A.K. Stahl, & Winfried Brenner. (2006). A patient with autosomal dominant polycystic kidney and liver disease presenting with urinary tract infection…‘Unable to see the wood for the trees’. Nephrology Dialysis Transplantation. 21(8). 2333–2334. 1 indexed citations

13.

Wenzel, Ulrich, U. Helmchen, Rolf A.K. Stahl, & C Nolte-Ernsting. (2006). Massive bleeding after biopsy of a renal allograft. Kidney International. 69(7). 1101–1101. 2 indexed citations

14.

Harendza, Sigrid, David H. Lovett, Ulf Panzer, et al.. (2003). Linked Common Polymorphisms in the Gelatinase A Promoter Are Associated with Diminished Transcriptional Response to Estrogen and Genetic Fitness. Journal of Biological Chemistry. 278(23). 20490–20499. 71 indexed citations

15.

Gómez‐Guerrero, Carmen, Sigrid Harendza, Purificación Hernández-Vargas, et al.. (2003). Differential Activation of NF-κB, AP-1, and C/EBP in Endotoxin-Tolerant Rats: Mechanisms for In Vivo Regulation of Glomerular RANTES/CCL5 Expression. The Journal of Immunology. 170(12). 6280–6291. 34 indexed citations

16.

Wenzel, Ulrich, Friedrich Thaiss, U. Helmchen, Rolf A.K. Stahl, & Günter Wolf. (2002). Angiotensin II infusion ameliorates the early phase of a mesangioproliferative glomerulonephritis11See Editorial by de Zeeuw, p. 1176.. Kidney International. 61(3). 1020–1029. 18 indexed citations

17.

Wolf, Günter, Ulf Panzer, & Rolf A.K. Stahl. (2000). Regulatory peptides and their antagonists in nephropathies. Current Opinion in Nephrology & Hypertension. 9(3). 233–239. 5 indexed citations

18.

Panzer, Ulf, André Schneider, Jill Wilken, et al.. (1999). The chemokine receptor antagonist AOP-RANTES reduces monocyte infiltration in experimental glomerulonephritis. Kidney International. 56(6). 2107–2115. 59 indexed citations

19.

Assmann, Karel J.M., et al.. (1997). Overexpression of aminopeptidase A abolishes the growth promoting effects of angiotensin II in cultured mouse mesangial cells. Kidney International. 52(5). 1250–1260. 14 indexed citations

20.

Luettichau, Irene von, Peter J. Nelson, James Pattison, et al.. (1996). RANTES chemokine expression in diseased and normal human tissues. Cytokine. 8(1). 89–98. 75 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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