David J. Salant

16.1k total citations · 4 hit papers
163 papers, 11.6k citations indexed

About

David J. Salant is a scholar working on Nephrology, Immunology and Molecular Biology. According to data from OpenAlex, David J. Salant has authored 163 papers receiving a total of 11.6k indexed citations (citations by other indexed papers that have themselves been cited), including 117 papers in Nephrology, 59 papers in Immunology and 51 papers in Molecular Biology. Recurrent topics in David J. Salant's work include Renal Diseases and Glomerulopathies (112 papers), Complement system in diseases (36 papers) and Chronic Kidney Disease and Diabetes (25 papers). David J. Salant is often cited by papers focused on Renal Diseases and Glomerulopathies (112 papers), Complement system in diseases (36 papers) and Chronic Kidney Disease and Diabetes (25 papers). David J. Salant collaborates with scholars based in United States, Japan and United Kingdom. David J. Salant's co-authors include Laurence H. Beck, William G. Couser, Andrey V. Cybulsky, Ramon Bonegio, Gérard Lambeau, Jon B. Klein, Richard J. Quigg, Timothy D. Cummins, David W. Powell and Helmut G. Rennke and has published in prestigious journals such as New England Journal of Medicine, Circulation and Journal of Clinical Investigation.

In The Last Decade

David J. Salant

159 papers receiving 11.3k citations

Hit Papers

M-Type Phospholipase A 2 Rece... 1978 2026 1994 2010 2009 2014 2010 1978 500 1000 1.5k

Peers

David J. Salant
Sanjeev Sethi United States
Samih H. Nasr United States
Fernando C. Fervenza United States
Ingeborg M. Bajema Netherlands
Jan A. Bruijn Netherlands
Lynn D. Cornell United States
Surya V. Seshan United States
Sanjeev Sethi United States
David J. Salant
Citations per year, relative to David J. Salant David J. Salant (= 1×) peers Sanjeev Sethi

Countries citing papers authored by David J. Salant

Since Specialization
Citations

This map shows the geographic impact of David J. Salant's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David J. Salant with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David J. Salant more than expected).

Fields of papers citing papers by David J. Salant

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David J. Salant. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David J. Salant. The network helps show where David J. Salant may publish in the future.

Co-authorship network of co-authors of David J. Salant

This figure shows the co-authorship network connecting the top 25 collaborators of David J. Salant. A scholar is included among the top collaborators of David J. Salant based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David J. Salant. David J. Salant is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Haddad, George, Johan M. Lorenzen, Hong Ma, et al.. (2020). Altered glycosylation of IgG4 promotes lectin complement pathway activation in anti-PLA2R1–associated membranous nephropathy. Journal of Clinical Investigation. 131(5). 125 indexed citations
2.
Braden, Gregory L., Arlene B. Chapman, David H. Ellison, et al.. (2020). Advancing Nephrology. Clinical Journal of the American Society of Nephrology. 16(2). 319–327. 7 indexed citations
3.
Caster, Dawn J., Min Tan, Michelle T. Barati, et al.. (2018). Neutrophil exocytosis induces podocyte cytoskeletal reorganization and proteinuria in experimental glomerulonephritis. American Journal of Physiology-Renal Physiology. 315(3). F595–F606. 12 indexed citations
4.
Estrada, Chelsea C., Yiqing Guo, Mónica P. Revelo, et al.. (2018). Krüppel-like factor 4 is a negative regulator of STAT3-induced glomerular epithelial cell proliferation. JCI Insight. 3(12). 31 indexed citations
5.
Kasinath, Vivek, Mayuko Uehara, Liwei Jiang, et al.. (2018). Activation of fibroblastic reticular cells in kidney lymph node during crescentic glomerulonephritis. Kidney International. 95(2). 310–320. 27 indexed citations
6.
Dai, Yan, Anqun Chen, Ruijie Liu, et al.. (2017). Retinoic acid improves nephrotoxic serum–induced glomerulonephritis through activation of podocyte retinoic acid receptor α. Kidney International. 92(6). 1444–1457. 29 indexed citations
7.
Hoxha, Elion, Laurence H. Beck, Thorsten Wiech, et al.. (2016). An Indirect Immunofluorescence Method Facilitates Detection of Thrombospondin Type 1 Domain–Containing 7A–Specific Antibodies in Membranous Nephropathy. Journal of the American Society of Nephrology. 28(2). 520–531. 152 indexed citations
8.
Braun, Gerald S., Yuichi Maruta, Felix Heymann, et al.. (2015). IL-6 Trans-Signaling Drives Murine Crescentic GN. Journal of the American Society of Nephrology. 27(1). 132–142. 49 indexed citations
9.
Cravedi, Paolo, Mauro Abbate, Elena Gagliardini, et al.. (2011). Membranous Nephropathy Associated With IgG4-Related Disease. American Journal of Kidney Diseases. 58(2). 272–275. 54 indexed citations
10.
Beck, Laurence H. & David J. Salant. (2010). Membranous nephropathy: recent travels and new roads ahead. Kidney International. 77(9). 765–770. 139 indexed citations
11.
Salant, David J., Vaishali Sanchorawala, & Vivette D. D’Agati. (2007). A Case of Atypical Light Chain Deposition Disease—Diagnosis and Treatment. Clinical Journal of the American Society of Nephrology. 2(4). 858–867. 9 indexed citations
12.
Lin, Feng, David J. Salant, Howard Meyerson, et al.. (2004). Respective Roles of Decay-Accelerating Factor and CD59 in Circumventing Glomerular Injury in Acute Nephrotoxic Serum Nephritis. The Journal of Immunology. 172(4). 2636–2642. 33 indexed citations
13.
Shinozaki, Michiya, Junichi Hirahashi, Tatiana Lebedeva, et al.. (2002). IL-15, a survival factor for kidney epithelial cells, counteracts apoptosis and inflammation during nephritis. Journal of Clinical Investigation. 109(7). 951–960. 64 indexed citations
14.
Shinozaki, Michiya, Junichi Hirahashi, Tatiana Lebedeva, et al.. (2002). IL-15, a survival factor for kidney epithelial cells, counteracts apoptosis and inflammation during nephritis. Journal of Clinical Investigation. 109(7). 951–960. 7 indexed citations
15.
Yanagita, Motoko, Yoshikazu Ishimoto, Hidenori Arai, et al.. (2002). Essential role of Gas6 for glomerular injury in nephrotoxic nephritis. Journal of Clinical Investigation. 110(2). 239–246. 5 indexed citations
16.
Topham, Peter, et al.. (1999). Complement-mediated injury reversibly disrupts glomerular epithelial cell actin microfilaments and focal adhesions. Kidney International. 55(5). 1763–1775. 66 indexed citations
17.
Bailey, Elaine M., Steven J. Harper, James H. Pringle, et al.. (1998). Visceral Glomerular Epithelial Cell DNA Synthesis in Experimental and Human Membranous Disease. Nephron Experimental Nephrology. 6(4). 352–358. 7 indexed citations
18.
Goldstein, Daniel J., David C. Wheeler, & David J. Salant. (1996). Effects of omega-3 fatty acids on complement-mediated glomerular epithelial cell injury. Kidney International. 50(6). 1863–1871. 6 indexed citations
19.
Salant, David J.. (1987). Immunopathogenesis of crescentic glomerulonephritis and lung purpura. Kidney International. 32(3). 408–425. 45 indexed citations
20.
Ed, Gomperts, et al.. (1977). Antithrombin functional activity after a fatty meal in normal subjects, familial hyperlipidemia, and nephrotic syndrome.. Munich Personal RePEc Archive (Ludwig Maximilian University of Munich). 90(3). 529–35. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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