Robert W. Rees

488 total citations
17 papers, 343 citations indexed

About

Robert W. Rees is a scholar working on Cancer Research, Molecular Biology and Small Animals. According to data from OpenAlex, Robert W. Rees has authored 17 papers receiving a total of 343 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Cancer Research, 8 papers in Molecular Biology and 5 papers in Small Animals. Recurrent topics in Robert W. Rees's work include Carcinogens and Genotoxicity Assessment (11 papers), Effects and risks of endocrine disrupting chemicals (5 papers) and Animal testing and alternatives (5 papers). Robert W. Rees is often cited by papers focused on Carcinogens and Genotoxicity Assessment (11 papers), Effects and risks of endocrine disrupting chemicals (5 papers) and Animal testing and alternatives (5 papers). Robert W. Rees collaborates with scholars based in United Kingdom, United States and Germany. Robert W. Rees's co-authors include Paul W. Hastwell, Richard M. Walmsley, James Harvey, Kevin J. Roberts, Anthony M. Lynch, Nicholas Billinton, Jennifer Southgate, Matthew Tate, Nicola J. MacLaine and Julie C. Holder and has published in prestigious journals such as Molecular Cancer Research, Toxicology in Vitro and Mutation Research/Genetic Toxicology and Environmental Mutagenesis.

In The Last Decade

Robert W. Rees

17 papers receiving 329 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Robert W. Rees United Kingdom 9 192 152 95 77 75 17 343
Abby Jacobs United States 7 207 1.1× 137 0.9× 110 1.2× 61 0.8× 48 0.6× 7 401
Albrecht Poth Germany 11 188 1.0× 165 1.1× 158 1.7× 81 1.1× 64 0.9× 21 439
Paul W. Hastwell United Kingdom 7 132 0.7× 109 0.7× 62 0.7× 49 0.6× 57 0.8× 7 230
Tatyana Y. Doktorova Belgium 13 142 0.7× 213 1.4× 87 0.9× 56 0.7× 45 0.6× 22 389
V. Thybaud France 8 297 1.5× 165 1.1× 171 1.8× 39 0.5× 125 1.7× 8 438
Rodger Curren United States 12 143 0.7× 116 0.8× 142 1.5× 209 2.7× 66 0.9× 21 498
Mick D. Fellows United Kingdom 10 245 1.3× 292 1.9× 104 1.1× 51 0.7× 68 0.9× 19 465
Matthew Tate United Kingdom 9 140 0.7× 123 0.8× 114 1.2× 44 0.6× 57 0.8× 15 291
Ulla Plappert‐Helbig Switzerland 10 156 0.8× 113 0.7× 86 0.9× 56 0.7× 65 0.9× 13 298
Katie Smith United Kingdom 7 246 1.3× 122 0.8× 133 1.4× 67 0.9× 98 1.3× 13 348

Countries citing papers authored by Robert W. Rees

Since Specialization
Citations

This map shows the geographic impact of Robert W. Rees's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert W. Rees with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert W. Rees more than expected).

Fields of papers citing papers by Robert W. Rees

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert W. Rees. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert W. Rees. The network helps show where Robert W. Rees may publish in the future.

Co-authorship network of co-authors of Robert W. Rees

This figure shows the co-authorship network connecting the top 25 collaborators of Robert W. Rees. A scholar is included among the top collaborators of Robert W. Rees based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Robert W. Rees. Robert W. Rees is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Billinton, Nicholas, Paul W. Hastwell, Christopher Jagger, et al.. (2010). A pre-validation transferability study of the GreenScreen HC GADD45a-GFP assay with a metabolic activation system (S9). Mutation Research/Genetic Toxicology and Environmental Mutagenesis. 700(1-2). 44–50. 12 indexed citations
2.
Hastwell, Paul W., Matthew Tate, Nicholas Billinton, et al.. (2009). Analysis of 75 marketed pharmaceuticals using the GADD45a-GFP ‘GreenScreen HC’ genotoxicity assay. Mutagenesis. 24(5). 455–463. 36 indexed citations
3.
Lynch, Anthony M., Mark D. Smith, Sharon A. Robinson, et al.. (2009). An evaluation of chemical photoreactivity and the relationship to photogenotoxicity. Regulatory Toxicology and Pharmacology. 58(2). 219–223. 6 indexed citations
4.
Billinton, Nicholas, Paul W. Hastwell, D. Beerens, et al.. (2008). Interlaboratory assessment of the GreenScreen HC GADD45a-GFP genotoxicity screening assay: An enabling study for independent validation as an alternative method. Mutation Research/Genetic Toxicology and Environmental Mutagenesis. 653(1-2). 23–33. 23 indexed citations
5.
Lynch, Anthony M., et al.. (2008). Is Muta™Mouse insensitive to clastogens?. Mutation Research/Genetic Toxicology and Environmental Mutagenesis. 652(2). 145–150. 4 indexed citations
6.
Lynch, Anthony M., Sharon A. Robinson, Phillip Wilcox, et al.. (2008). Cycloheximide and disulfoton are positive in the photoclastogencity assay but do not absorb UV irradiation: another example of pseudophotoclastogenicity?. Mutagenesis. 23(2). 111–118. 18 indexed citations
7.
Collins, J, et al.. (2008). Evaluation of the Litron In Vitro MicroFlow® Kit for the flow cytometric enumeration of micronuclei (MN) in mammalian cells. Mutation Research/Genetic Toxicology and Environmental Mutagenesis. 654(1). 76–81. 14 indexed citations
8.
MacLaine, Nicola J., et al.. (2008). Sensitivity of Normal, Paramalignant, and Malignant Human Urothelial Cells to Inhibitors of the Epidermal Growth Factor Receptor Signaling Pathway. Molecular Cancer Research. 6(1). 53–63. 27 indexed citations
9.
Thybaud, Véronique, Marilyn J. Aardema, Daniel A. Casciano, et al.. (2007). Relevance and follow-up of positive results in in vitro genetic toxicity assays: An ILSI-HESI initiative. Mutation Research/Genetic Toxicology and Environmental Mutagenesis. 633(2). 67–79. 27 indexed citations
10.
Hastwell, Paul W., et al.. (2006). High-specificity and high-sensitivity genotoxicity assessment in a human cell line: Validation of the GreenScreen HC GADD45a-GFP genotoxicity assay. Mutation Research/Genetic Toxicology and Environmental Mutagenesis. 607(2). 160–175. 118 indexed citations
11.
Lord, Peter G., et al.. (2002). Inter-individual variation in urothelial DNA repair gene expression in vitro. Toxicology in Vitro. 16(4). 383–387. 5 indexed citations
12.
Smith, Catherine C., et al.. (1999). Analysis of gene mutations and clastogenicity following short-term treatment with azathioprine in Muta?Mouse. Environmental and Molecular Mutagenesis. 34(2-3). 131–139. 25 indexed citations
13.
14.
15.
Rees, Robert W., et al.. (1989). Optimization of metabolic activation for four mutagens in a bacterial, fungal and two mammalian cell mutagenesis assays. Mutagenesis. 4(5). 335–342. 8 indexed citations
16.
Rees, Robert W., et al.. (1982). 4CMB: Investigation of activity in a mouse micronucleus test. Mutation Research/Genetic Toxicology. 100(1-4). 361–364. 3 indexed citations
17.
Fox, Mary E., Robert W. Rees, Daimark Bennett, & Laurie Henry. (1971). Investigations into the lymphopaenic and immunosuppressive properties of the antitumour agent, mitoclomine.. PubMed. 4(2). 35–47. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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