Matthew Tate

15 papers receiving 274 citations

Peers

Matthew Tate
Comparison fields: 5 of 63
  • Chemical Health and Safety 9
  • Cancer Research 140
  • Health, Toxicology and Mutagenesis 114
  • Small Animals 44
  • Pollution 33
Replace Robert W. Rees with:
Robert W. Rees United Kingdom
Paul W. Hastwell United Kingdom
Albrecht Poth Germany
Tatyana Y. Doktorova Belgium
Laura Jeffrey United Kingdom
Darren Kidd United Kingdom
Katie Smith United Kingdom
Ulla Plappert‐Helbig Switzerland
Julia Kenny United Kingdom
Werner Bomann United States
Matthew Tate relative to Robert W. Rees United Kingdom Robert W. Rees's profile →
Citations per field
00.5×
Robert W. Rees · 1×
Citations per year

Countries citing papers authored by Matthew Tate

Since Specialization
Citations

This map shows the geographic impact of Matthew Tate's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew Tate with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew Tate more than expected).

Fields of papers citing papers by Matthew Tate

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Matthew Tate. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew Tate. The network helps show where Matthew Tate may publish in the future.

Co-authors

The 25 scholars most cited alongside Matthew Tate, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Matthew Tate Line = papers co-authored together Matthew Tate links everyone, so they are left out of the graph.

All Works

15 of 15 papers shown
#Work
1 20234
2 20223
3 20192
4 20173
5 201615
6 201551
7 201331
8 201235
9 201121
10 201012
11 200936
12 200943
13 20091
14 200831
15
Biochemical studies on lymphoblastoid cells with inherited N-acetyl-glucosamine 1-phosphotransferase deficiency (I-cell disease).
19883

About Matthew Tate

Matthew Tate is a scholar working on Small Animals, Cancer Research, Health, Toxicology and Mutagenesis, Biophysics and Pollution, having authored 15 papers that have together received 291 indexed citations. Recurring topics across this work include Carcinogens and Genotoxicity Assessment (9 papers), Effects and risks of endocrine disrupting chemicals (5 papers), Pesticide Exposure and Toxicity (5 papers), Animal testing and alternatives (5 papers), DNA Repair Mechanisms (2 papers), Acute Lymphoblastic Leukemia research (2 papers), Carbohydrate Chemistry and Synthesis (1 paper) and Air Quality and Health Impacts (1 paper). The work is most often cited by research in Chemical Health and Safety (9 citations), Cancer Research (140 citations), Health, Toxicology and Mutagenesis (114 citations), Small Animals (44 citations) and Pollution (33 citations). Matthew Tate has collaborated with scholars based in United Kingdom, Switzerland and United States. Frequent co-authors include Richard M. Walmsley, Nicholas Billinton, Paul A. Cahill, Andrew C. Povey, Rhoderick H. Elder, Anthony M. Lynch, Adam Rabinowitz, Andrew W. Knight, James Harvey and Paul W. Hastwell. Their work appears in journals such as Mutagenesis, Mutation Research/Genetic Toxicology and Environmental Mutagenesis, SLAS DISCOVERY, Environmental and Molecular Mutagenesis and Toxicology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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