Robert Opoka

912 total citations
7 papers, 740 citations indexed

About

Robert Opoka is a scholar working on Molecular Biology, Genetics and Immunology. According to data from OpenAlex, Robert Opoka has authored 7 papers receiving a total of 740 indexed citations (citations by other indexed papers that have themselves been cited), including 4 papers in Molecular Biology, 3 papers in Genetics and 3 papers in Immunology. Recurrent topics in Robert Opoka's work include Immune Cell Function and Interaction (3 papers), Diabetes and associated disorders (2 papers) and Epigenetics and DNA Methylation (2 papers). Robert Opoka is often cited by papers focused on Immune Cell Function and Interaction (3 papers), Diabetes and associated disorders (2 papers) and Epigenetics and DNA Methylation (2 papers). Robert Opoka collaborates with scholars based in United States, Germany and Switzerland. Robert Opoka's co-authors include James M. Wells, Jennifer J. Kordich, Yan Dong, Hongzhu Liu, Chun Han, Tatyana Y. Belenkaya, Xinhua Lin, Marat Khodoun, Scott A. Rankin and Jason R. Spence and has published in prestigious journals such as Cell, The Journal of Immunology and Molecular and Cellular Biology.

In The Last Decade

Robert Opoka

7 papers receiving 732 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Robert Opoka United States	6	569	167	159	140	73	7	740
Heather P. Ostendorff Germany	11	590 1.0×	118 0.7×	160 1.0×	50 0.4×	50 0.7×	14	778
Madeline N. Hayes Canada	10	463 0.8×	201 1.2×	109 0.7×	69 0.5×	40 0.5×	16	677
Tomohisa Sekimoto Japan	13	314 0.6×	54 0.3×	116 0.7×	133 0.9×	87 1.2×	33	589
Shuyi Nie United States	15	764 1.3×	122 0.7×	121 0.8×	58 0.4×	33 0.5×	27	914
Ursula Zimber‐Strobl Switzerland	8	672 1.2×	123 0.7×	134 0.8×	214 1.5×	124 1.7×	9	1.0k
Gunther Wuytens Belgium	9	639 1.1×	146 0.9×	75 0.5×	88 0.6×	44 0.6×	9	802
Karine Sii-Felice France	14	418 0.7×	53 0.3×	99 0.6×	80 0.6×	41 0.6×	17	589
Junko Hyodo‐Miura Japan	8	727 1.3×	105 0.6×	104 0.7×	46 0.3×	76 1.0×	8	837
N Shimizu Japan	14	451 0.8×	107 0.6×	133 0.8×	55 0.4×	97 1.3×	25	756
Jawahir Y. Mohamed Saudi Arabia	15	512 0.9×	90 0.5×	239 1.5×	74 0.5×	28 0.4×	20	849

Countries citing papers authored by Robert Opoka

Since Specialization

Citations

This map shows the geographic impact of Robert Opoka's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert Opoka with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert Opoka more than expected).

Fields of papers citing papers by Robert Opoka

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert Opoka. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert Opoka. The network helps show where Robert Opoka may publish in the future.

Co-authorship network of co-authors of Robert Opoka

This figure shows the co-authorship network connecting the top 25 collaborators of Robert Opoka. A scholar is included among the top collaborators of Robert Opoka based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Robert Opoka. Robert Opoka is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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7 of 7 papers shown

1.

Mattner, Jochen, Javid P. Mohammed, Michael E. Fusakio, et al.. (2019). Genetic and functional data identifying Cd101 as a type 1 diabetes (T1D) susceptibility gene in nonobese diabetic (NOD) mice. PLoS Genetics. 15(6). e1008178–e1008178. 10 indexed citations

2.

Opoka, Robert, Stephanie Schmidt, Sidonia Mihai, et al.. (2016). A mutation within the SH2 domain of slp‐76 regulates the tissue distribution and cytokine production of iNKT cells in mice. European Journal of Immunology. 46(9). 2121–2136. 1 indexed citations

3.

Katz, Jonathan D., et al.. (2010). Cutting Edge: Merocytic Dendritic Cells Break T Cell Tolerance to β Cell Antigens in Nonobese Diabetic Mouse Diabetes. The Journal of Immunology. 185(4). 1999–2003. 33 indexed citations

4.

Opoka, Robert, et al.. (2007). Identification of molecular markers that are expressed in discrete anterior–posterior domains of the endoderm from the gastrula stage to mid‐gestation. Developmental Dynamics. 236(7). 1997–2003. 35 indexed citations

5.

Sinner, Débora, Jennifer J. Kordich, Jason R. Spence, et al.. (2007). Sox17 and Sox4 Differentially Regulate β-Catenin/T-Cell Factor Activity and Proliferation of Colon Carcinoma Cells. Molecular and Cellular Biology. 27(22). 7802–7815. 274 indexed citations

6.

Sordet‐Dessimoz, Jessica, Robert Opoka, Jennifer J. Kordich, Anne Grapin‐Botton, & James M. Wells. (2005). FGF signaling is necessary for establishing gut tube domains alongthe anterior–posterior axis in vivo. Mechanisms of Development. 123(1). 42–55. 136 indexed citations

7.

Belenkaya, Tatyana Y., Chun Han, Yan Dong, et al.. (2004). Drosophila Dpp Morphogen Movement Is Independent of Dynamin-Mediated Endocytosis but Regulated by the Glypican Members of Heparan Sulfate Proteoglycans. Cell. 119(2). 231–244. 251 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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