Rita A. Avelar

444 total citations
8 papers, 85 citations indexed

About

Rita A. Avelar is a scholar working on Molecular Biology, Cell Biology and Cancer Research. According to data from OpenAlex, Rita A. Avelar has authored 8 papers receiving a total of 85 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 2 papers in Cell Biology and 2 papers in Cancer Research. Recurrent topics in Rita A. Avelar's work include Ubiquitin and proteasome pathways (3 papers), Cell death mechanisms and regulation (2 papers) and Autophagy in Disease and Therapy (1 paper). Rita A. Avelar is often cited by papers focused on Ubiquitin and proteasome pathways (3 papers), Cell death mechanisms and regulation (2 papers) and Autophagy in Disease and Therapy (1 paper). Rita A. Avelar collaborates with scholars based in United States, Australia and Singapore. Rita A. Avelar's co-authors include Analisa DiFeo, Sreeja C. Sekhar, Stephanie L. Skala, Goutham Narla, Stefanie Avril, Ronny Drapkin, Caitlin M. O’Connor, Sarah E. Taylor, Michele Dziubinski and Peronne Joseph and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and Nature Communications.

In The Last Decade

Rita A. Avelar

7 papers receiving 85 citations

Peers

Countries citing papers authored by Rita A. Avelar

Since Specialization

Citations

This map shows the geographic impact of Rita A. Avelar's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Rita A. Avelar with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Rita A. Avelar more than expected).

Fields of papers citing papers by Rita A. Avelar

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Rita A. Avelar. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Rita A. Avelar. The network helps show where Rita A. Avelar may publish in the future.

Co-authorship network of co-authors of Rita A. Avelar

This figure shows the co-authorship network connecting the top 25 collaborators of Rita A. Avelar. A scholar is included among the top collaborators of Rita A. Avelar based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Rita A. Avelar. Rita A. Avelar is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

#	Work	Indexed citations
1	Understanding the Functional Dependence and Inhibition of the Bcl-2 Pro-Survival Proteins in a Wide Spectrum of Cancers toward Precision Medicine 2025 ·ACS Pharmacology & Translational Science ·(unknown), (unknown), (unknown), Rita A. Avelar, Carlos Murga‐Zamalloa, (unknown), Malathi Kandarpa, Ahmed S.A. Mady, Analisa DiFeo, Lin Zhang, Sami N. Malek, Tycel Phillips, (unknown), Dale L. Bixby, Moshe Talpaz, Zaneta Nikolovska‐Coleska	1
2	Small-molecule modulators of B56-PP2A restore 4E-BP function to suppress eIF4E-dependent translation in cancer cells 2025 ·Journal of Clinical Investigation ·Michelle A. Lum, (unknown), Surendra Parmar, Adrian R. Black, Caitlin M. O’Connor, Stephanie Dobersch, Naomi Yamamoto, Thomas Robertson, (unknown), (unknown), Rita A. Avelar, Analisa DiFeo, Nicholas T. Woods, Sita Kugel, Goutham Narla, Jennifer D. Black	0
3	Integrated stress response plasticity governs normal cell adaptation to chronic stress via the PP2A-TFE3-ATF4 pathway 2024 ·Cell Death and Differentiation ·Rita A. Avelar, (unknown), (unknown), (unknown), (unknown), (unknown), (unknown), Alexey I. Nesvizhskii, Caitlin M. O’Connor, (unknown), Shirish Shenolikar, Peter Arvan, (unknown), Analisa DiFeo	3
4	miR-181a initiates and perpetuates oncogenic transformation through the regulation of innate immune signaling 2020 ·Nature Communications ·(unknown), Rita A. Avelar, Sreeja C. Sekhar, (unknown), Michele Dziubinski, (unknown), Stephanie L. Skala, Stefanie Avril, Ronny Drapkin, Analisa DiFeo	30
5	Inactivation of PP2A by a recurrent mutation drives resistance to MEK inhibitors 2019 ·Oncogene ·Caitlin M. O’Connor, Daniel Léonard, Danica Wiredja, Rita A. Avelar, Zhizhi Wang, Daniela Schlatzer, Benjamin L. Bryson, (unknown), Sarah E. Taylor, Aditya Upadhyay, Jaya Sangodkar, Anne‐Claude Gingras, Jukka Westermarck, Wenqing Xu, Analisa DiFeo, David L. Brautigan, Shozeb Haider, Mark W. Jackson, Goutham Narla	27
6	Protein phosphatase 2A Aα regulates Aβ protein expression and stability 2019 ·Journal of Biological Chemistry ·Caitlin M. O’Connor, (unknown), Sarah E. Taylor, Rita A. Avelar, Goutham Narla	9
7	Mitotic Exit Dysfunction through the Deregulation of APC/C Characterizes Cisplatin-Resistant State in Epithelial Ovarian Cancer 2018 ·Clinical Cancer Research ·Anil Belur Nagaraj, Olga Kovalenko, Rita A. Avelar, Peronne Joseph, (unknown), (unknown), (unknown), Analisa DiFeo	13
8	Abstract C132: Therapeutic reactivation of PP2A for prostate cancer treatment 2015 ·Molecular Cancer Therapeutics ·(unknown), Rita A. Avelar, (unknown), David B. Kastrinsky, Michael Ohlmeyer, Stephen R. Plymate, Matthew D. Galsky, Goutham Narla	2

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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