Mark W. Jackson

6.9k total citations · 1 hit paper
127 papers, 5.0k citations indexed

About

Mark W. Jackson is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Mark W. Jackson has authored 127 papers receiving a total of 5.0k indexed citations (citations by other indexed papers that have themselves been cited), including 62 papers in Molecular Biology, 53 papers in Oncology and 20 papers in Cancer Research. Recurrent topics in Mark W. Jackson's work include Cancer-related Molecular Pathways (26 papers), Cancer Cells and Metastasis (16 papers) and Ubiquitin and proteasome pathways (14 papers). Mark W. Jackson is often cited by papers focused on Cancer-related Molecular Pathways (26 papers), Cancer Cells and Metastasis (16 papers) and Ubiquitin and proteasome pathways (14 papers). Mark W. Jackson collaborates with scholars based in United States, United Kingdom and China. Mark W. Jackson's co-authors include George R. Stark, Damian J. Junk, Rocky Cipriano, Steven J. Berberich, Perry J. Hardin, Jacob Smigiel, Benjamin L. Bryson, Courtney A. Bartel, Lindsey D. Mayo and Neetha Parameswaran and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Journal of Clinical Investigation.

In The Last Decade

Mark W. Jackson

124 papers receiving 4.9k citations

Hit Papers

IFNβ-dependent increases in STAT1, STAT2, and IRF9 mediat... 2013 2026 2017 2021 2013 50 100 150 200 250

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Mark W. Jackson United States 42 2.7k 1.8k 879 635 440 127 5.0k
Hiroshi Nakagawa United States 49 3.5k 1.3× 2.3k 1.3× 1.1k 1.3× 712 1.1× 358 0.8× 159 6.8k
Dalong Ma China 38 2.6k 1.0× 1.3k 0.7× 638 0.7× 933 1.5× 296 0.7× 169 4.7k
Masahiro Nakashima Japan 34 1.7k 0.6× 1.1k 0.6× 751 0.9× 1.0k 1.6× 399 0.9× 208 5.3k
Shinji Yamamoto Japan 39 2.4k 0.9× 1.1k 0.6× 651 0.7× 529 0.8× 407 0.9× 261 6.0k
Harry P. Erba United States 42 3.3k 1.2× 1.6k 0.9× 475 0.5× 635 1.0× 411 0.9× 252 6.6k
Hitoshi Ichikawa Japan 42 2.9k 1.1× 1.2k 0.7× 804 0.9× 367 0.6× 365 0.8× 142 5.0k
Michael J. Atkinson Germany 45 3.9k 1.4× 1.4k 0.8× 1.6k 1.8× 487 0.8× 383 0.9× 213 7.2k
Jing Huang United States 42 4.8k 1.8× 1.7k 0.9× 1.1k 1.3× 749 1.2× 664 1.5× 135 7.2k
Lars L. Vindeløv Denmark 31 2.3k 0.8× 1.9k 1.0× 1.2k 1.4× 776 1.2× 571 1.3× 113 6.1k
Guidalberto Manfioletti Italy 43 4.4k 1.6× 862 0.5× 1.7k 2.0× 1000 1.6× 693 1.6× 107 6.9k

Countries citing papers authored by Mark W. Jackson

Since Specialization
Citations

This map shows the geographic impact of Mark W. Jackson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mark W. Jackson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mark W. Jackson more than expected).

Fields of papers citing papers by Mark W. Jackson

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mark W. Jackson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mark W. Jackson. The network helps show where Mark W. Jackson may publish in the future.

Co-authorship network of co-authors of Mark W. Jackson

This figure shows the co-authorship network connecting the top 25 collaborators of Mark W. Jackson. A scholar is included among the top collaborators of Mark W. Jackson based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mark W. Jackson. Mark W. Jackson is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Huang, Wei, Derek J. Taylor, Sarah E. Taylor, et al.. (2024). Mutant PP2A Induces IGFBP2 Secretion to Promote Development of High-Grade Uterine Cancer. Cancer Research. 85(3). 442–461. 3 indexed citations
2.
Tamagno, Ilaria, et al.. (2024). Toll-Like receptor 3-mediated interferon-β production is suppressed by oncostatin m and a broader epithelial-mesenchymal transition program. Breast Cancer Research. 26(1). 167–167. 1 indexed citations
3.
Tamagno, Ilaria, Neetha Parameswaran, Jacob Smigiel, et al.. (2023). Oncostatin-M and OSM-Receptor Feed-Forward Activation of MAPK Induces Separable Stem-like and Mesenchymal Programs. Molecular Cancer Research. 21(9). 975–990. 4 indexed citations
4.
Bryson, Benjamin L., et al.. (2020). Aberrant Induction of a Mesenchymal/Stem Cell Program Engages Senescence in Normal Mammary Epithelial Cells. Molecular Cancer Research. 19(4). 651–666. 6 indexed citations
5.
Taylor, Sarah E., Caitlin M. O’Connor, Zhizhi Wang, et al.. (2019). The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis. Cancer Research. 79(16). 4242–4257. 42 indexed citations
6.
Parameswaran, Neetha, Courtney A. Bartel, Kristy Miskimen, et al.. (2019). A FAM83A Positive Feed-back Loop Drives Survival and Tumorigenicity of Pancreatic Ductal Adenocarcinomas. Scientific Reports. 9(1). 13396–13396. 28 indexed citations
7.
O’Connor, Caitlin M., Daniel Léonard, Danica Wiredja, et al.. (2019). Inactivation of PP2A by a recurrent mutation drives resistance to MEK inhibitors. Oncogene. 39(3). 703–717. 27 indexed citations
8.
Doherty, Mary R. & Mark W. Jackson. (2018). The Critical, Clinical Role of Interferon-Beta in Regulating Cancer Stem Cell Properties in Triple-Negative Breast Cancer. DNA and Cell Biology. 37(6). 513–516. 14 indexed citations
9.
Smigiel, Jacob, Neetha Parameswaran, & Mark W. Jackson. (2017). Potent EMT and CSC Phenotypes Are Induced By Oncostatin-M in Pancreatic Cancer. Molecular Cancer Research. 15(4). 478–488. 67 indexed citations
10.
Bartel, Courtney A. & Mark W. Jackson. (2017). HER2-positive breast cancer cells expressing elevated FAM83A are sensitive to FAM83A loss. PLoS ONE. 12(5). e0176778–e0176778. 32 indexed citations
11.
Karan, Goutam, Amit Chakrabarti, Sukanya Karan, et al.. (2016). Identification of a Small Molecule That Overcomes HdmX-Mediated Suppression of p53. Molecular Cancer Therapeutics. 15(4). 574–582. 17 indexed citations
12.
Merry, Callie R., Megan E. Forrest, Lydia Beard, et al.. (2015). DNMT1-associated long non-coding RNAs regulate global gene expression and DNA methylation in colon cancer. Human Molecular Genetics. 24(21). 6240–6253. 135 indexed citations
13.
Cipriano, Rocky, et al.. (2014). Conserved Oncogenic Behavior of the FAM83 Family Regulates MAPK Signaling in Human Cancer. Molecular Cancer Research. 12(8). 1156–1165. 90 indexed citations
14.
Cipriano, Rocky, et al.. (2011). c-MYC Functions as a Molecular Switch to Alter the Response of Human Mammary Epithelial Cells to Oncostatin M. Cancer Research. 71(22). 6930–6939. 31 indexed citations
15.
Helm, Jenny, et al.. (2009). A case of canine Angiostrongylus vasorum in Scotland confirmed by PCR and sequence analysis. Journal of Small Animal Practice. 50(5). 255–259. 45 indexed citations
16.
Jackson, Richard H. & Mark W. Jackson. (2009). Students Assessment of a Semidirected Internship Program. Journal of Geography. 108(2). 57–67. 10 indexed citations
17.
Davis, James, Mark W. Jackson, & Richard H. Jackson. (2009). Heritage tourism and group identity: Polynesians in the American West. Journal of Heritage Tourism. 4(1). 3–17. 1 indexed citations
18.
Patton, John T., et al.. (2007). p53-Mediated Growth Suppression in Response to Nutlin-3 in Cyclin D1–Transformed Cells Occurs Independently of p21. Cancer Research. 67(20). 9862–9868. 22 indexed citations
19.
Patton, John T., Lindsey D. Mayo, Aatur D. Singhi, et al.. (2006). Levels of HdmX Expression Dictate the Sensitivity of Normal and Transformed Cells to Nutlin-3. Cancer Research. 66(6). 3169–3176. 147 indexed citations
20.
Fincher, Ruth‐Marie E., Mark W. Jackson, & Asma Q. Fischer. (1990). Case Report: Citrobacter Freundii: A Newly Reported Cause of Pyomyositis. The American Journal of the Medical Sciences. 299(5). 331–333. 16 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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