Richard W. Gross
About
Richard W. Gross is a scholar working on Molecular Biology, Biochemistry and Physiology.
According to data from OpenAlex, Richard W. Gross has authored 223 papers receiving a total of 21.0k indexed citations (citations by other indexed papers that have themselves been cited), including 179 papers in Molecular Biology, 51 papers in Biochemistry and 49 papers in Physiology. Recurrent topics in Richard W. Gross's work include Metabolomics and Mass Spectrometry Studies (55 papers), Mitochondrial Function and Pathology (46 papers) and Lipid metabolism and biosynthesis (39 papers). Richard W. Gross is often cited by papers focused on Metabolomics and Mass Spectrometry Studies (55 papers), Mitochondrial Function and Pathology (46 papers) and Lipid metabolism and biosynthesis (39 papers). Richard W. Gross collaborates with scholars based in United States, Belarus and Brazil. Richard W. Gross's co-authors include Xianlin Han, Kui Yang, Christopher M. Jenkins, David J. Mancuso, Stanley L. Hazen, Burton E. Sobel, David A. Ford, Daniel P. Kelly, Harold F. Sims and Brian N. Finck and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of the American Chemical Society and Journal of Biological Chemistry.
In The Last Decade
Richard W. Gross
221 papers receiving 20.6k citations
Hit Papers
Peers — A (Enhanced Table)
Peers by citation overlap · career bar shows stage (early→late) cites · hero ref
| Name | h | Career | Trend | Papers | Cites | |||||
|---|---|---|---|---|---|---|---|---|---|---|
| Richard W. Gross United States | 79 | 14.7k | 4.7k | 3.7k | 3.4k | 2.6k | 223 | 21.0k | ||
| Xianlin Han United States | 87 | 19.2k 1.3× | 7.7k 1.7× | 4.1k 1.1× | 5.0k 1.5× | 2.7k 1.1× | 363 | 28.8k | ||
| Takao Shimizu Japan | 87 | 14.5k 1.0× | 5.8k 1.2× | 4.0k 1.1× | 1.5k 0.4× | 803 0.3× | 494 | 28.4k | ||
| Andrew P. Halestrap United Kingdom | 97 | 22.5k 1.5× | 6.7k 1.4× | 2.5k 0.7× | 599 0.2× | 2.0k 0.8× | 228 | 35.3k | ||
| Charles L. Hoppel United States | 77 | 14.4k 1.0× | 5.8k 1.3× | 1.5k 0.4× | 623 0.2× | 2.2k 0.8× | 350 | 23.0k | ||
| Sidney Fleischer United States | 77 | 17.6k 1.2× | 2.8k 0.6× | 2.1k 0.6× | 715 0.2× | 5.2k 2.0× | 301 | 23.8k | ||
| Ronald J. A. Wanders Netherlands | 102 | 32.0k 2.2× | 8.2k 1.8× | 4.3k 1.2× | 575 0.2× | 998 0.4× | 828 | 42.4k | ||
| John Turk United States | 73 | 8.5k 0.6× | 2.9k 0.6× | 2.0k 0.5× | 2.2k 0.6× | 306 0.1× | 267 | 15.5k | ||
| J. David Lambeth United States | 81 | 11.5k 0.8× | 8.4k 1.8× | 1.4k 0.4× | 503 0.1× | 1.3k 0.5× | 184 | 25.1k | ||
| António Zorzano Spain | 78 | 14.6k 1.0× | 5.8k 1.3× | 4.9k 1.3× | 319 0.1× | 1.1k 0.4× | 345 | 23.0k | ||
| Manuel Palacı́n Spain | 67 | 10.7k 0.7× | 3.6k 0.8× | 5.8k 1.6× | 421 0.1× | 645 0.2× | 255 | 17.1k |
Countries citing papers authored by Richard W. Gross
Since
Specialization
Citations
This map shows the geographic impact of Richard W. Gross's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Richard W. Gross with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Richard W. Gross more than expected).
Fields of papers citing papers by Richard W. Gross
Since
Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences
This network shows the impact of papers produced by Richard W. Gross. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Richard W. Gross. The network helps show where Richard W. Gross may publish in the future.
Co-authorship network of co-authors of Richard W. Gross
This figure shows the co-authorship network connecting the top 25 collaborators of Richard W. Gross. A scholar is included among the top collaborators of Richard W. Gross based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Richard W. Gross. Richard W. Gross is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
1.
Jenkins, Christopher M., et al.. (2018). Cytochrome c is an oxidative stress–activated plasmalogenase that cleaves plasmenylcholine and plasmenylethanolamine at the sn-1 vinyl ether linkage. Journal of Biological Chemistry. 293(22). 8693–8709.
2.
Moon, Sung Ho, Xinping Liu, Ari Cedars, et al.. (2017). Heart failure–induced activation of phospholipase iPLA2γ generates hydroxyeicosatetraenoic acids opening the mitochondrial permeability transition pore. Journal of Biological Chemistry. 293(1). 115–129.
3.
Beirowski, Bogdan, Elisabetta Babetto, Judith P. Golden, et al.. (2014). Metabolic regulator LKB1 is crucial for Schwann cell–mediated axon maintenance. Nature Neuroscience. 17(10). 1351–1361.
4.
Viader, Andreu, Yo Sasaki, Sung‐Su Kim, et al.. (2013). Aberrant Schwann Cell Lipid Metabolism Linked to Mitochondrial Deficits Leads to Axon Degeneration and Neuropathy. Neuron. 77(5). 886–898.
5.
Gross, Richard W. & Xianlin Han. (2011). Lipidomics at the Interface of Structure and Function in Systems Biology. Chemistry & Biology. 18(3). 284–291.
6.
Basantani, Mahesh Kumar, Lingzhi Cai, Daniel Brenner, et al.. (2010). Pnpla3/Adiponutrin deficiency in mice does not contribute to fatty liver disease or metabolic syndrome. Journal of Lipid Research. 52(2). 318–329.
7.
Malik, Ibrahim, John Turk, David J. Mancuso, et al.. (2008). Disrupted Membrane Homeostasis and Accumulation of Ubiquitinated Proteins in a Mouse Model of Infantile Neuroaxonal Dystrophy Caused by PLA2G6 Mutations. American Journal Of Pathology. 172(2). 406–416.
8.
Burkart, Eileen M., Nandakumar Sambandam, Xianlin Han, et al.. (2007). Nuclear receptors PPARβ/δ and PPARα direct distinct metabolic regulatory programs in the mouse heart. Journal of Clinical Investigation. 117(12). 3930–9.
9.
Mancuso, David J., Xianlin Han, Christopher M. Jenkins, et al.. (2007). Dramatic Accumulation of Triglycerides and Precipitation of Cardiac Hemodynamic Dysfunction during Brief Caloric Restriction in Transgenic Myocardium Expressing Human Calcium-independent Phospholipase A2γ. Journal of Biological Chemistry. 282(12). 9216–9227.
10.
Yang, Kui, Zhongdan Zhao, Richard W. Gross, & Xianlin Han. (2007). Shotgun Lipidomics Identifies a Paired Rule for the Presence of Isomeric Ether Phospholipid Molecular Species. PLoS ONE. 2(12). e1368–e1368.
11.
Han, Xianlin, Kui Yang, Jingyue Yang, Hua Cheng, & Richard W. Gross. (2006). Shotgun lipidomics of cardiolipin molecular species in lipid extracts of biological samples. Journal of Lipid Research. 47(4). 864–879.
12.
Jenkins, Christopher M., Wei Yan, David J. Mancuso, & Richard W. Gross. (2006). Highly Selective Hydrolysis of Fatty Acyl-CoAs by Calcium-independent Phospholipase A2β. Journal of Biological Chemistry. 281(23). 15615–15624.
13.
Su, Xiong, Xianlin Han, David J. Mancuso, Dana R. Abendschein, & Richard W. Gross. (2005). Accumulation of Long-Chain Acylcarnitine and 3-Hydroxy Acylcarnitine Molecular Species in Diabetic Myocardium: Identification of Alterations in Mitochondrial Fatty Acid Processing in Diabetic Myocardium by Shotgun Lipidomics. Biochemistry. 44(13). 5234–5245.
14.
Han, Xianlin & Richard W. Gross. (2004). Shotgun lipidomics: Electrospray ionization mass spectrometric analysis and quantitation of cellular lipidomes directly from crude extracts of biological samples. Mass Spectrometry Reviews. 24(3). 367–412.
15.
Gross, Richard W., Christopher M. Jenkins, Jingyue Yang, David J. Mancuso, & Xianlin Han. (2004). Functional lipidomics: the roles of specialized lipids and lipid–protein interactions in modulating neuronal function. Prostaglandins & Other Lipid Mediators. 77(1-4). 52–64.
16.
Finck, Brian N., John J. Lehman, Teresa C. Leone, et al.. (2002). The cardiac phenotype induced by PPARα overexpression mimics that caused by diabetes mellitus. Journal of Clinical Investigation. 109(1). 121–130.
17.
Finck, Brian N., John J. Lehman, Teresa C. Leone, et al.. (2002). The cardiac phenotype induced by PPARα overexpression mimics that caused by diabetes mellitus. Journal of Clinical Investigation. 109(1). 121–130.
18.
Jenkins, Christopher M., Xianlin Han, David J. Mancuso, & Richard W. Gross. (2002). Identification of Calcium-independent Phospholipase A2 (iPLA2) β, and Not iPLA2γ, as the Mediator of Arginine Vasopressin-induced Arachidonic Acid Release in A-10 Smooth Muscle Cells. Journal of Biological Chemistry. 277(36). 32807–32814.
19.
Han, Xianlin & Richard W. Gross. (2001). Quantitative Analysis and Molecular Species Fingerprinting of Triacylglyceride Molecular Species Directly from Lipid Extracts of Biological Samples by Electrospray Ionization Tandem Mass Spectrometry. Analytical Biochemistry. 295(1). 88–100.
20.
Ford, David A. & Richard W. Gross. (1989). Differential accumulation of diacyl and plasmalogenic diglycerides during myocardial ischemia.. Circulation Research. 64(1). 173–177.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.
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