Jost I. Preis

1.8k total citations
13 papers, 1.3k citations indexed

About

Jost I. Preis is a scholar working on Molecular Biology, Genetics and Pediatrics, Perinatology and Child Health. According to data from OpenAlex, Jost I. Preis has authored 13 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 6 papers in Genetics and 3 papers in Pediatrics, Perinatology and Child Health. Recurrent topics in Jost I. Preis's work include Epigenetics and DNA Methylation (7 papers), Congenital heart defects research (6 papers) and Genetic Syndromes and Imprinting (3 papers). Jost I. Preis is often cited by papers focused on Epigenetics and DNA Methylation (7 papers), Congenital heart defects research (6 papers) and Genetic Syndromes and Imprinting (3 papers). Jost I. Preis collaborates with scholars based in Australia, United Kingdom and Japan. Jost I. Preis's co-authors include Emma Whitelaw, Vardhman K. Rakyan, Marnie E. Blewitt, Hugh D. Morgan, Sally L. Dunwoodie, Duncan B. Sparrow, Sarah L. Withington, Richard P. Harvey, Juan Pedro Martı́nez-Barberá and Milena B. Furtado and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Genes & Development and Molecular and Cellular Biology.

In The Last Decade

Jost I. Preis

13 papers receiving 1.3k citations

Peers

Jost I. Preis
Jost I. Preis
Citations per year, relative to Jost I. Preis Jost I. Preis (= 1×) peers Evica Rajcan‐Separovic

Countries citing papers authored by Jost I. Preis

Since Specialization
Citations

This map shows the geographic impact of Jost I. Preis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jost I. Preis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jost I. Preis more than expected).

Fields of papers citing papers by Jost I. Preis

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jost I. Preis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jost I. Preis. The network helps show where Jost I. Preis may publish in the future.

Co-authorship network of co-authors of Jost I. Preis

This figure shows the co-authorship network connecting the top 25 collaborators of Jost I. Preis. A scholar is included among the top collaborators of Jost I. Preis based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jost I. Preis. Jost I. Preis is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

13 of 13 papers shown
1.
O׳Reilly, Victoria C., Hongjun Shi, Bogdan E. Chapman, et al.. (2014). Gene–environment interaction demonstrates the vulnerability of the embryonic heart. Developmental Biology. 391(1). 99–110. 12 indexed citations
2.
Artap, Stanley T, Jost I. Preis, Diane Fatkin, et al.. (2010). Loss of Cited2 causes congenital heart disease by perturbing left–right patterning of the body axis. Human Molecular Genetics. 20(6). 1097–1110. 49 indexed citations
3.
Furtado, Milena B., Mark J. Solloway, Vanessa Jones, et al.. (2008). BMP/SMAD1 signaling sets a threshold for the left/right pathway in lateral plate mesoderm and limits availability of SMAD4. Genes & Development. 22(21). 3037–3049. 57 indexed citations
4.
Withington, Sarah L., Annabelle Scott, Darren N. Saunders, et al.. (2006). Loss of Cited2 affects trophoblast formation and vascularization of the mouse placenta. Developmental Biology. 294(1). 67–82. 90 indexed citations
5.
Preis, Jost I., et al.. (2006). Generation of conditionalCited2 null alleles. genesis. 44(12). 579–583. 20 indexed citations
6.
Stennard, Fiona A., Mauro W. Costa, Donna Lai, et al.. (2005). Murine T-box transcription factor Tbx20 acts as a repressor during heart development, and is essential for adult heart integrity, function and adaptation. Development. 132(10). 2451–2462. 171 indexed citations
7.
Weninger, Wolfgang J., M. B. Bennett, Sarah L. Withington, et al.. (2005). Cited2is required both for heart morphogenesis and establishment of the left-right axis in mouse development. Development. 132(6). 1337–1348. 109 indexed citations
8.
Blewitt, Marnie E., Nicola Vickaryous, Sarah J. Hemley, et al.. (2005). AnN-ethyl-N-nitrosourea screen for genes involved in variegation in the mouse. Proceedings of the National Academy of Sciences. 102(21). 7629–7634. 141 indexed citations
9.
Preis, Jost I., Meredith Downes, Nathan Oates, John E.J. Rasko, & Emma Whitelaw. (2003). Sensitive Flow Cytometric Analysis Reveals a Novel Type of Parent-of-Origin Effect in the Mouse Genome. Current Biology. 13(11). 955–959. 29 indexed citations
10.
Rodríguez, Tristan A., Duncan B. Sparrow, Annabelle Scott, et al.. (2003). Cited1 Is Required in Trophoblasts for Placental Development and for Embryo Growth and Survival. Molecular and Cellular Biology. 24(1). 228–244. 73 indexed citations
11.
Rakyan, Vardhman K., et al.. (2002). Metastable epialleles in mammals. Trends in Genetics. 18(7). 348–351. 348 indexed citations
12.
Rakyan, Vardhman K., Jost I. Preis, Hugh D. Morgan, & Emma Whitelaw. (2001). The marks, mechanisms and memory of epigenetic states in mammals. Biochemical Journal. 356(1). 1–1. 131 indexed citations
13.
Rakyan, Vardhman K., Jost I. Preis, Hugh D. Morgan, & Emma Whitelaw. (2001). The marks, mechanisms and memory of epigenetic states in mammals. Biochemical Journal. 356(1). 1–10. 89 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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