Raymond G. Booth
- Molecular Biology top 10%
- Cellular and Molecular Neuroscience top 2%
- Organic Chemistry top 10%
- Clinical Psychology top 10%
- Pharmacology top 10%
- Co-authors
- Clinton E. CanalRoss J. BaldessariniSteven D. WyrickDrake MorganNeal CastagnoliHans RollemaNeepa ChoksiTania Córdova
- Topics
- Receptor Mechanisms and Signaling (40 papers)Neurotransmitter Receptor Influence on Behavior (22 papers)Pharmacological Receptor Mechanisms and Effects (16 papers)
- Partner nations
- United StatesNetherlandsChina
In The Last Decade
Raymond G. Booth
68 papers receiving 1.4k citations
Peers
Comparison fields: 5 of 90
- Molecular Biology 810
- Cellular and Molecular Neuroscience 784
- Organic Chemistry 211
- Clinical Psychology 159
- Pharmacology 102
Countries citing papers authored by Raymond G. Booth
This map shows the geographic impact of Raymond G. Booth's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Raymond G. Booth with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Raymond G. Booth more than expected).
Fields of papers citing papers by Raymond G. Booth
This network shows the impact of papers produced by Raymond G. Booth. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Raymond G. Booth. The network helps show where Raymond G. Booth may publish in the future.
Co-authorship network of co-authors of Raymond G. Booth
This figure shows the co-authorship network connecting the top 25 collaborators of Raymond G. Booth. A scholar is included among the top collaborators of Raymond G. Booth based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Raymond G. Booth. Raymond G. Booth is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 2 | |
| 2 | 1 | |
| 3 | 10 | |
| 4 | 6 | |
| 5 | 20 | |
| 6 | 5 | |
| 7 | 22 | |
| 8 | 13 | |
| 9 | 18 | |
| 10 | 20 | |
| 11 | 37 | |
| 12 | 9 | |
| 13 | 38 | |
| 14 | 27 | |
| 15 | 19 | |
| 16 | 15 | |
| 17 | 49 | |
| 18 | 13 | |
| 19 | 20 | |
| 20 | 52 |
About Raymond G. Booth
Raymond G. Booth is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Physiology, having authored 70 papers that have together received 1.4k indexed citations. Recurring topics across this work include Receptor Mechanisms and Signaling (40 papers), Neurotransmitter Receptor Influence on Behavior (22 papers) and Pharmacological Receptor Mechanisms and Effects (16 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (784 citations), Molecular Biology (810 citations) and Toxicology (32 citations). Raymond G. Booth has collaborated with scholars based in United States, Netherlands and China. Frequent co-authors include Clinton E. Canal, Ross J. Baldessarini, Steven D. Wyrick, Drake Morgan, Neal Castagnoli, Hans Rollema, Neepa Choksi, Tania Córdova, Nader H. Moniri and Rob Leurs. Their work appears in journals such as Brain Research, The FASEB Journal and Biochemical and Biophysical Research Communications.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.