Raúl Peña

1.5k total citations · 1 hit paper
17 papers, 1.0k citations indexed

About

Raúl Peña is a scholar working on Molecular Biology, Oncology and Surgery. According to data from OpenAlex, Raúl Peña has authored 17 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 12 papers in Oncology and 3 papers in Surgery. Recurrent topics in Raúl Peña's work include Cancer Cells and Metastasis (11 papers), Fibroblast Growth Factor Research (3 papers) and Cancer, Hypoxia, and Metabolism (3 papers). Raúl Peña is often cited by papers focused on Cancer Cells and Metastasis (11 papers), Fibroblast Growth Factor Research (3 papers) and Cancer, Hypoxia, and Metabolism (3 papers). Raúl Peña collaborates with scholars based in Spain, United States and Sweden. Raúl Peña's co-authors include Antonio Garcı́a de Herreros, Félix Bonilla, Cristina Peña, Isabel Puig, Ana Belén Cid Álvarez, Manuel Beltrán, Josep Baulida, Mercedes Herrera, Lorena Alba‐Castellón and J. Ignacio Casal and has published in prestigious journals such as Genes & Development, PLoS ONE and Cancer Research.

In The Last Decade

Raúl Peña

17 papers receiving 1.0k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

A natural antisense transcript regulates Zeb2/Sip1 gene expression during Snail1-induced epithelial–mesenchymal transition

2008 511 citations Manuel Beltrán, Isabel Puig et al. Genes & Development profile →

Peers

Raúl Peña

ONCOL

IMMUN

Julia Mitschke Germany

Caroline Moyret‐Lalle France

Deepak Kumar Singh United States

Shi-Juan Mai China

Nadya Dimitrova United States

William K.C. Cheung China

Sung-Suk Suh United States

Eric Sceusi United States

Julia Mitschke Germany

Raúl Peña

545 ×0.7

351 ×0.7

464 ×1.5

ONCOL

93 ×1.1

102 ×1.3

IMMUN

Citations per year, relative to Raúl Peña Raúl Peña (= 1×) peers Julia Mitschke

Countries citing papers authored by Raúl Peña

Since Specialization

Citations

This map shows the geographic impact of Raúl Peña's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Raúl Peña with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Raúl Peña more than expected).

Fields of papers citing papers by Raúl Peña

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Raúl Peña. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Raúl Peña. The network helps show where Raúl Peña may publish in the future.

Co-authorship network of co-authors of Raúl Peña

This figure shows the co-authorship network connecting the top 25 collaborators of Raúl Peña. A scholar is included among the top collaborators of Raúl Peña based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Raúl Peña. Raúl Peña is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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17 of 17 papers shown

Valle‐Pérez, Beatriz del, et al.. (2023). Noncanonical Wnt signaling promotes colon tumor growth, chemoresistance and tumor fibroblast activation. EMBO Reports. 24(4). e54895–e54895. 15 indexed citations

Mazzolini, Rocco, et al.. (2023). Analyzing the role of cancer‐associated fibroblast activation on macrophage polarization. Molecular Oncology. 17(8). 1492–1513. 6 indexed citations

Peña, Raúl, Alessandra Iaconcig, Juan L. Trincado, et al.. (2023). Formation of an invasion-permissive matrix requires TGFβ/SNAIL1-regulated alternative splicing of fibronectin. Breast Cancer Research. 25(1). 143–143. 4 indexed citations

Peña, Raúl, et al.. (2021). Snail1 expression in endothelial cells controls growth, angiogenesis and differentiation of breast tumors. Theranostics. 11(16). 7671–7684. 10 indexed citations

Peña, Raúl, José Rodríguez-Morató, Lorena Alba‐Castellón, et al.. (2020). Glutamine-Directed Migration of Cancer-Activated Fibroblasts Facilitates Epithelial Tumor Invasion. Cancer Research. 81(2). 438–451. 52 indexed citations

Stanisavljević, Jelena, et al.. (2019). Abrogation of myofibroblast activities in metastasis and fibrosis by methyltransferase inhibition. International Journal of Cancer. 145(11). 3064–3077. 23 indexed citations

Herrera, Alberto, Mercedes Herrera, Natalia Guerra‐Pérez, et al.. (2018). Endothelial cell activation on 3D-matrices derived from PDGF-BB-stimulated fibroblasts is mediated by Snail1. Oncogenesis. 7(9). 76–76. 28 indexed citations

Miceli, Martina, Raúl Peña, Boyko S. Atanassov, et al.. (2018). TGFβ-Activated USP27X Deubiquitinase Regulates Cell Migration and Chemoresistance via Stabilization of Snail1. Cancer Research. 79(1). 33–46. 82 indexed citations

Valle‐Pérez, Beatriz del, et al.. (2018). CK1ε and p120‐catenin control Ror2 function in noncanonical Wnt signaling. Molecular Oncology. 12(5). 611–629. 15 indexed citations

10.

Alba‐Castellón, Lorena, Raúl Peña, Mercedes Herrera, et al.. (2016). Snail1-Dependent Activation of Cancer-Associated Fibroblast Controls Epithelial Tumor Cell Invasion and Metastasis. Cancer Research. 76(21). 6205–6217. 69 indexed citations

11.

Mateu-Jiménez, Mercè, Clara Fermoselle, Federico Rojo, et al.. (2016). Pharmacological Approaches in an Experimental Model of Non-Small Cell Lung Cancer: Effects on Tumor Biology. Current Pharmaceutical Design. 22(34). 5300–5310. 7 indexed citations

12.

Loubat‐Casanovas, Jordina, Raúl Peña, Núria Gonzàlez, et al.. (2015). Snail1 is required for the maintenance of the pancreatic acinar phenotype. Oncotarget. 7(4). 4468–4482. 7 indexed citations

13.

Stanisavljević, Jelena, Jordina Loubat‐Casanovas, Mercedes Herrera, et al.. (2014). Snail1-Expressing Fibroblasts in the Tumor Microenvironment Display Mechanical Properties That Support Metastasis. Cancer Research. 75(2). 284–295. 98 indexed citations

14.

Alba‐Castellón, Lorena, Raquel Batlle, Clara Francı́, et al.. (2014). Snail1 Expression Is Required for Sarcomagenesis. Neoplasia. 16(5). 413–421. 24 indexed citations

15.

Toll, Agustí, Emili Masferrer, Carla Ferrándiz‐Pulido, et al.. (2013). Epithelial to mesenchymal transition markers are associated with an increased metastatic risk in primary cutaneous squamous cell carcinomas but are attenuated in lymph node metastases. Journal of Dermatological Science. 72(2). 93–102. 60 indexed citations

16.

Larriba, María Jesús, Juan Casado‐Vela, Natalia Pendás-Franco, et al.. (2010). Novel Snail1 Target Proteins in Human Colon Cancer Identified by Proteomic Analysis. PLoS ONE. 5(4). e10221–e10221. 28 indexed citations

17.

Beltrán, Manuel, Isabel Puig, Cristina Peña, et al.. (2008). A natural antisense transcript regulates Zeb2/Sip1 gene expression during Snail1-induced epithelial–mesenchymal transition. Genes & Development. 22(6). 756–769. 511 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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