R. M. Helliwell

693 total citations
16 papers, 592 citations indexed

About

R. M. Helliwell is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Cellular and Molecular Neuroscience. According to data from OpenAlex, R. M. Helliwell has authored 16 papers receiving a total of 592 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 9 papers in Cardiology and Cardiovascular Medicine and 8 papers in Cellular and Molecular Neuroscience. Recurrent topics in R. M. Helliwell's work include Ion channel regulation and function (10 papers), Cardiac electrophysiology and arrhythmias (9 papers) and Neuroscience and Neuropharmacology Research (4 papers). R. M. Helliwell is often cited by papers focused on Ion channel regulation and function (10 papers), Cardiac electrophysiology and arrhythmias (9 papers) and Neuroscience and Neuropharmacology Research (4 papers). R. M. Helliwell collaborates with scholars based in United Kingdom, Australia and United States. R. M. Helliwell's co-authors include W. A. Large, Iain A. Greenwood, Marcel J. de Groot, Derek J. Leishman, Martin Tristani‐Firouzi, John S. Mitcheson, Matthew Perry, Michael C. Sanguinetti, Leanne Stokes and R. C. Hogg and has published in prestigious journals such as The Journal of Physiology, British Journal of Pharmacology and Molecular Pharmacology.

In The Last Decade

R. M. Helliwell

16 papers receiving 580 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
R. M. Helliwell United Kingdom 12 455 216 187 120 66 16 592
H Ozaki Japan 4 340 0.7× 135 0.6× 104 0.6× 58 0.5× 14 0.2× 7 702
Tsuyoshi Ohmura Japan 15 485 1.1× 121 0.6× 285 1.5× 19 0.2× 22 0.3× 30 846
Toshihito Hiroi Japan 6 215 0.5× 40 0.2× 51 0.3× 385 3.2× 109 1.7× 7 654
Edith Wehage Germany 3 157 0.3× 37 0.2× 58 0.3× 357 3.0× 125 1.9× 4 508
James T. Limberis United States 11 384 0.8× 300 1.4× 169 0.9× 201 1.7× 5 0.1× 21 612
Hannes Reuter Germany 13 381 0.8× 434 2.0× 148 0.8× 23 0.2× 5 0.1× 28 702
Sanjay Danthi United States 13 338 0.7× 102 0.5× 159 0.9× 28 0.2× 7 0.1× 15 466
Sven Kathöfer Germany 18 641 1.4× 603 2.8× 184 1.0× 30 0.3× 4 0.1× 27 865
Xudong Ding China 13 270 0.6× 57 0.3× 67 0.4× 45 0.4× 5 0.1× 36 563
Siaw Wei Ng Singapore 9 197 0.4× 15 0.1× 118 0.6× 174 1.4× 26 0.4× 10 579

Countries citing papers authored by R. M. Helliwell

Since Specialization
Citations

This map shows the geographic impact of R. M. Helliwell's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by R. M. Helliwell with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites R. M. Helliwell more than expected).

Fields of papers citing papers by R. M. Helliwell

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by R. M. Helliwell. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by R. M. Helliwell. The network helps show where R. M. Helliwell may publish in the future.

Co-authorship network of co-authors of R. M. Helliwell

This figure shows the co-authorship network connecting the top 25 collaborators of R. M. Helliwell. A scholar is included among the top collaborators of R. M. Helliwell based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with R. M. Helliwell. R. M. Helliwell is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Helliwell, R. M., et al.. (2023). Ginsenosides enhance P2X7-dependent cytokine secretion from LPS-primed rodent macrophages. Purinergic Signalling. 20(1). 65–71. 4 indexed citations
2.
Bidula, Stefan, et al.. (2019). Positive allosteric modulation of P2X7 promotes apoptotic cell death over lytic cell death responses in macrophages. Cell Death and Disease. 10(12). 882–882. 41 indexed citations
3.
Helliwell, R. M., et al.. (2015). Selected ginsenosides of the protopanaxdiol series are novel positive allosteric modulators ofP2X7 receptors. British Journal of Pharmacology. 172(13). 3326–3340. 40 indexed citations
4.
Shergis, Johannah Linda, et al.. (2014). Therapeutic potential of Panax ginseng and ginsenosides in the treatment of chronic obstructive pulmonary disease. Complementary Therapies in Medicine. 22(5). 944–953. 54 indexed citations
5.
Shen, Bojiang, et al.. (2013). An in vitro study of neuroprotective properties of traditional Chinese herbal medicines thought to promote healthy ageing and longevity. BMC Complementary and Alternative Medicine. 13(1). 373–373. 29 indexed citations
6.
Chen, Mao Xiang, R. M. Helliwell, & Jeff J. Clare. (2009). In vitro profiling against ion channels beyond hERG as an early indicator of cardiac risk.. PubMed. 11(3). 269–81. 5 indexed citations
7.
Helliwell, R. M.. (2008). Recording hERG Potassium Currents and Assessing the Effects of Compounds Using the Whole-Cell Patch-Clamp Technique. Methods in molecular biology. 491. 279–295. 3 indexed citations
8.
Perry, Matthew, Marcel J. de Groot, R. M. Helliwell, et al.. (2004). Structural Determinants of HERG Channel Block by Clofilium and Ibutilide. Molecular Pharmacology. 66(2). 240–249. 135 indexed citations
9.
Helliwell, R. M. & W. A. Large. (1998). Facilitatory effect of Ca2+ on the noradrenaline‐evoked cation current in rabbit portal vein smooth muscle cells. The Journal of Physiology. 512(3). 731–741. 19 indexed citations
10.
Greenwood, Iain A., R. M. Helliwell, & W. A. Large. (1997). Modulation of Ca2+‐activated Cl currents in rabbit portal vein smooth muscle by an inhibitor of mitochondrial Ca2+ uptake. The Journal of Physiology. 505(1). 53–64. 46 indexed citations
11.
Helliwell, R. M. & W. A. Large. (1997). Alpha 1‐adrenoceptor activation of a non‐selective cation current in rabbit portal vein by 1,2‐diacyl‐sn‐glycerol.. The Journal of Physiology. 499(2). 417–428. 92 indexed citations
12.
Toma, Catalin, Iain A. Greenwood, R. M. Helliwell, & W. A. Large. (1996). Activation of potassium currents by inhibitors of calcium‐activated chloride conductance in rabbit portal vein smooth muscle cells. British Journal of Pharmacology. 118(3). 513–520. 29 indexed citations
13.
Helliwell, R. M. & W. A. Large. (1996). Dual effect of external Ca2+ on noradrenaline‐activated cation current in rabbit portal vein smooth muscle cells.. The Journal of Physiology. 492(1). 75–88. 34 indexed citations
14.
Helliwell, R. M. & W. A. Large. (1995). Effect of temperature on spontaneous Ca2+-activated CI? currents in rabbit portal vein cells. Pflügers Archiv - European Journal of Physiology. 431(1). 28–31. 7 indexed citations
15.
Helliwell, R. M., et al.. (1994). Synergistic action of histamine and adenosine triphosphate on the response to noradrenaline in rabbit pulmonary artery smooth muscle cells. Pflügers Archiv - European Journal of Physiology. 426(5). 433–439. 16 indexed citations
16.
Hogg, R. C., Q. Wang, R. M. Helliwell, & W. A. Large. (1993). Properties of spontaneous inward currents in rabbit pulmonary artery smooth muscle cells. Pflügers Archiv - European Journal of Physiology. 425(3-4). 233–240. 38 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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