R. Boland

1.0k citations
41 papers · 819 indexed · h-index 15
Topics
Vitamin D Research Studies (11 papers)Ion channel regulation and function (6 papers)Muscle Physiology and Disorders (4 papers)

In The Last Decade

R. Boland

40 papers receiving 772 citations

Peers

R. Boland
Comparison fields: 5 of 97
  • Molecular Biology 423
  • Pathology and Forensic Medicine 310
  • Cell Biology 117
  • Physiology 117
  • Nutrition and Dietetics 103
Replace Birgit Bölck with:
Birgit Bölck Germany
Doris Rosenthal Brazil
Karl Heinrich Fritzemeier Germany
Lorena Milanesi Argentina
Samuela Cataldi Italy
A. Ata Alturfan Türkiye
Tomoo Noguchi Japan
Dallas G. Clark Australia
Alba Vecchini Italy
Carmen Aceves Mexico
R. Boland relative to Birgit Bölck Germany Birgit Bölck's profile →
Citations per field
00.5×4.1×
Birgit Bölck · 1×
Citations per year

Countries citing papers authored by R. Boland

Since Specialization
Citations

This map shows the geographic impact of R. Boland's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by R. Boland with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites R. Boland more than expected).

Fields of papers citing papers by R. Boland

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by R. Boland. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by R. Boland. The network helps show where R. Boland may publish in the future.

Co-authorship network of co-authors of R. Boland

This figure shows the co-authorship network connecting the top 25 collaborators of R. Boland. A scholar is included among the top collaborators of R. Boland based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with R. Boland. R. Boland is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 1
2 1
3 23
4 5
5 17
6 12
7
[Changes in bone mass and in glucose homeostasis in subjects with high spontaneous fluoride intake].
17
8 37
9 63
10 14
11 93
12 42
13 97
14 5
15 11
16 12
17 5
18 66
19 138
20
The mechanism of Ca transport and the permeability of sarcoplasmic reticulum membranes.
5

About R. Boland

R. Boland is a scholar working on Pathology and Forensic Medicine, Nephrology and Physiology, having authored 41 papers that have together received 819 indexed citations. Recurring topics across this work include Vitamin D Research Studies (11 papers), Ion channel regulation and function (6 papers) and Muscle Physiology and Disorders (4 papers). The work is most often cited by research in Pathology and Forensic Medicine (310 citations), Cell Biology (117 citations) and Sensory Systems (33 citations). R. Boland has collaborated with scholars based in Argentina, United States and Germany. Frequent co-authors include Ana Russo de Boland, A. Martonosi, S. Morelli, Hiroshi Nakamura, R L Jilka, Dikla G. Roufa, Thomas W. Tillack, Susana B. Zanello, Guillermo Vázquez and M. Huarte. Their work appears in journals such as Journal of Biological Chemistry, Analytical Biochemistry and Biochemical Journal.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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