Prudence N. Gatt

507 total citations
13 papers, 358 citations indexed

About

Prudence N. Gatt is a scholar working on Immunology, Pathology and Forensic Medicine and Neurology. According to data from OpenAlex, Prudence N. Gatt has authored 13 papers receiving a total of 358 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Immunology, 5 papers in Pathology and Forensic Medicine and 2 papers in Neurology. Recurrent topics in Prudence N. Gatt's work include Multiple Sclerosis Research Studies (3 papers), T-cell and B-cell Immunology (3 papers) and Immune Cell Function and Interaction (3 papers). Prudence N. Gatt is often cited by papers focused on Multiple Sclerosis Research Studies (3 papers), T-cell and B-cell Immunology (3 papers) and Immune Cell Function and Interaction (3 papers). Prudence N. Gatt collaborates with scholars based in Australia, United States and Belgium. Prudence N. Gatt's co-authors include Graeme J. Stewart, David R. Booth, Steve Vucic, Grant P. Parnell, Michael Barnett, Fiona C. McKay, Fabienne Brilot, Andrew Henderson, Stephen D. Schibeci and Karrnan Pathmanandavel and has published in prestigious journals such as PLoS ONE, Human Molecular Genetics and Journal of Neuroimmunology.

In The Last Decade

Prudence N. Gatt

13 papers receiving 349 citations

Peers

Countries citing papers authored by Prudence N. Gatt

Since Specialization

Citations

This map shows the geographic impact of Prudence N. Gatt's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Prudence N. Gatt with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Prudence N. Gatt more than expected).

Fields of papers citing papers by Prudence N. Gatt

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Prudence N. Gatt. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Prudence N. Gatt. The network helps show where Prudence N. Gatt may publish in the future.

Co-authorship network of co-authors of Prudence N. Gatt

This figure shows the co-authorship network connecting the top 25 collaborators of Prudence N. Gatt. A scholar is included among the top collaborators of Prudence N. Gatt based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Prudence N. Gatt. Prudence N. Gatt is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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13 of 13 papers shown

#	Work	Indexed citations
1	Indeterminate measurable residual disease by multiparameter flow cytometry is associated with an intermediate risk of clinical relapse in adult patients with acute leukaemia 2024 ·Pathology ·(unknown), (unknown), Deborah Cromer, Prudence N. Gatt, (unknown), Marian A. Fernandez, (unknown), Emily Blyth, Jennifer Curnow, (unknown), David A. Brown, Sarah C. Sasson	1
2	Measurable Residual Disease (MRD) by Flow Cytometry in Adult B-Acute Lymphoblastic Leukaemia (B-ALL) and Acute Myeloid Leukaemia (AML): Correlation with Molecular MRD Testing and Clinical Outcome at One Year 2023 ·Cancers ·(unknown), Prudence N. Gatt, (unknown), Marian A. Fernandez, (unknown), Emily Blyth, Jennifer Curnow, David A. Brown, (unknown), Sarah C. Sasson	4
3	Expression of CYP24A1 and other multiple sclerosis risk genes in peripheral blood indicates response to vitamin D in homeostatic and inflammatory conditions 2021 ·Genes and Immunity ·(unknown), Prudence N. Gatt, Stephen D. Schibeci, Fiona C. McKay, Steve Vucic, Prue H. Hart, Scott N. Byrne, David A. Brown, Graeme J. Stewart, Christopher Liddle, Grant P. Parnell, David R. Booth	2
4	The autoimmune risk gene ZMIZ1 is a vitamin D responsive marker of a molecular phenotype of multiple sclerosis 2017 ·Journal of Autoimmunity ·Nicole Fewings, Prudence N. Gatt, Fiona C. McKay, Grant P. Parnell, Stephen D. Schibeci, Jarem Edwards, (unknown), (unknown), Marzena J. Fabis‐Pedrini, Allan G. Kermode, (unknown), Jacob L. McCauley, Dorothee Nickles, Sergio E. Baranzini, Therese Burke, Steve Vucic, Graeme J. Stewart, David R. Booth	23
5	Data characterizing the ZMIZ1 molecular phenotype of multiple sclerosis 2017 ·Data in Brief ·Nicole Fewings, Prudence N. Gatt, Fiona C. McKay, Grant P. Parnell, Stephen D. Schibeci, Jarem Edwards, (unknown), (unknown), Marzena J. Fabis‐Pedrini, Allan G. Kermode, (unknown), Jacob L. McCauley, Dorothee Nickles, Sergio E. Baranzini, Therese Burke, Steve Vucic, Graeme J. Stewart, David R. Booth	5
6	The low EOMES/TBX21 molecular phenotype in multiple sclerosis reflects CD56+ cell dysregulation and is affected by immunomodulatory therapies 2016 ·Clinical Immunology ·Fiona C. McKay, Prudence N. Gatt, Nicole Fewings, Grant P. Parnell, Stephen D. Schibeci, (unknown), Joseph E. Powell, (unknown), Marzena J. Fabis‐Pedrini, Allan G. Kermode, Therese Burke, Steve Vucic, Graeme J. Stewart, David R. Booth	17
7	The autoimmune disease-associated transcription factors EOMES and TBX21 are dysregulated in multiple sclerosis and define a molecular subtype of disease 2014 ·Clinical Immunology ·Grant P. Parnell, Prudence N. Gatt, Małgorzata Krupa, Dorothee Nickles, Fiona C. McKay, Stephen D. Schibeci, Marcel Batten, Sergio E. Baranzini, Andrew Henderson, Michael Barnett, Mark Slee, Steve Vucic, Graeme J. Stewart, David R. Booth	44
8	Antibodies to myelin oligodendrocyte glycoprotein in bilateral and recurrent optic neuritis 2014 ·Journal of Neuroimmunology ·Sudarshini Ramanathan, Stephen Reddel, Andrew Henderson, John Parratt, Michael Barnett, Prudence N. Gatt, Vera Merheb, (unknown), Karrnan Pathmanandavel, Nese Sinmaz, Mahtab Ghadiri, Con Yiannikas, Steve Vucic, (unknown), Andrew Bleasel, David R. Booth, Victor S.C. Fung, Russell C. Dale, Fabienne Brilot	2
9	Antibodies to myelin oligodendrocyte glycoprotein in bilateral and recurrent optic neuritis 2014 ·Neurology Neuroimmunology & Neuroinflammation ·Sudarshini Ramanathan, Stephen Reddel, Andrew Henderson, John Parratt, Michael Barnett, Prudence N. Gatt, Vera Merheb, (unknown), Karrnan Pathmanandavel, Nese Sinmaz, Mahtab Ghadiri, Con Yiannikas, Steve Vucic, Graeme J. Stewart, Andrew Bleasel, David R. Booth, Victor S.C. Fung, Russell C. Dale, Fabienne Brilot	169
10	IL7Rα Expression and Upregulation by IFNβ in Dendritic Cell Subsets Is Haplotype-Dependent 2013 ·PLoS ONE ·Fiona C. McKay, (unknown), Grant P. Parnell, Prudence N. Gatt, Stephen D. Schibeci, Graeme J. Stewart, David R. Booth	9
11	The CYP27B1 variant associated with an increased risk of autoimmune disease is underexpressed in tolerizing dendritic cells 2013 ·Human Molecular Genetics ·(unknown), Grant P. Parnell, Fiona C. McKay, Prudence N. Gatt, (unknown), Kate O'Connor, Stephen D. Schibeci, Fabienne Brilot, Christopher Liddle, Marcel Batten, Graeme J. Stewart, David R. Booth	38
12	Long‐term reversal of diabetes in non‐obese diabetic mice by liver‐directed gene therapy 2013 ·The Journal of Gene Medicine ·(unknown), Bronwyn A. O’Brien, (unknown), (unknown), Prudence N. Gatt, M. A. Swan, Najah T. Nassif, Wei Ming, Rik Gijsbers, Zeger Debyser, Ann M. Simpson	26
13	Ribosomal protein S6 mRNA is a biomarker upregulated in multiple sclerosis, downregulated by interferon treatment, and affected by season 2013 ·Multiple Sclerosis Journal ·Grant P. Parnell, Prudence N. Gatt, Fiona C. McKay, Stephen D. Schibeci, Małgorzata Krupa, Joseph E. Powell, Peter M. Visscher, Grant W. Montgomery, Jeannette Lechner‐Scott, Simon Broadley, Christopher Liddle, Mark Slee, Steve Vucic, Graeme J. Stewart, David R. Booth	18

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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