Pooja Biswas

416 total citations
25 papers, 232 citations indexed

About

Pooja Biswas is a scholar working on Molecular Biology, Ophthalmology and Genetics. According to data from OpenAlex, Pooja Biswas has authored 25 papers receiving a total of 232 indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Molecular Biology, 13 papers in Ophthalmology and 5 papers in Genetics. Recurrent topics in Pooja Biswas's work include Retinal Development and Disorders (14 papers), Retinal Diseases and Treatments (12 papers) and Lysosomal Storage Disorders Research (4 papers). Pooja Biswas is often cited by papers focused on Retinal Development and Disorders (14 papers), Retinal Diseases and Treatments (12 papers) and Lysosomal Storage Disorders Research (4 papers). Pooja Biswas collaborates with scholars based in United States, India and Pakistan. Pooja Biswas's co-authors include Radha Ayyagari, Jacque L. Duncan, John R. Heckenlively, Sheikh Riazuddin, Kari Branham, Kelly A. Frazer, J. Fielding Hejtmancik, Hiroko Matsui, Pauline Lee and Muhammad Asif Naeem and has published in prestigious journals such as PLoS ONE, Human Molecular Genetics and Investigative Ophthalmology & Visual Science.

In The Last Decade

Pooja Biswas

22 papers receiving 229 citations

Peers

Pooja Biswas
Kaylie Webb-Jones United States
Martha A. Cady United States
Mohammed E. El‐Asrag United Kingdom
Zachry T. Soens United States
Aaron Black United States
Pooja Biswas
Citations per year, relative to Pooja Biswas Pooja Biswas (= 1×) peers Muhammad Asif Naeem

Countries citing papers authored by Pooja Biswas

Since Specialization
Citations

This map shows the geographic impact of Pooja Biswas's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Pooja Biswas with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Pooja Biswas more than expected).

Fields of papers citing papers by Pooja Biswas

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Pooja Biswas. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Pooja Biswas. The network helps show where Pooja Biswas may publish in the future.

Co-authorship network of co-authors of Pooja Biswas

This figure shows the co-authorship network connecting the top 25 collaborators of Pooja Biswas. A scholar is included among the top collaborators of Pooja Biswas based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Pooja Biswas. Pooja Biswas is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Biswas, Pooja, TJ Hollingsworth, Naheed W. Khan, et al.. (2025). Ablation of Htra1 leads to sub-RPE deposits and photoreceptor abnormalities. JCI Insight. 10(3). 1 indexed citations
2.
Biswas, Pooja, Benjamin J. Krajacich, Junhua Zhao, et al.. (2024). Avidity sequencing of whole genomes from retinal degeneration pedigrees identifies causal variants. PLoS ONE. 19(10). e0307266–e0307266. 2 indexed citations
3.
Sangermano, Riccardo, Pooja Biswas, Lori S. Sullivan, et al.. (2022). Identification of a novel large multigene deletion and a frameshift indel inPDE6Bas the underlying cause of early-onset recessive rod–cone degeneration. Molecular Case Studies. 8(7). a006247–a006247.
5.
Khan, Shahid Y., Muhammad Ali, Wendy Lee, et al.. (2020). Whole genome sequencing data of multiple individuals of Pakistani descent. Scientific Data. 7(1). 350–350. 2 indexed citations
6.
Biswas, Pooja, Shyamanga Borooah, Hiroko Matsui, et al.. (2020). Detection and validation of novel mutations inMERTKin a simplex case of retinal degeneration using WGS and hiPSC–RPEs model. Human Mutation. 42(2). 189–199. 3 indexed citations
7.
Biswas, Pooja, Kari Branham, Shyamanga Borooah, et al.. (2018). IFT88 mutations identified in individuals with non-syndromic recessive retinal degeneration result in abnormal ciliogenesis. Human Genetics. 137(6-7). 447–458. 11 indexed citations
8.
Branham, Kari, Igor Kozak, Pooja Biswas, et al.. (2018). Identification of Novel Deletions as the Underlying Cause of Retinal Degeneration in Two Pedigrees. Advances in experimental medicine and biology. 1074. 229–236. 2 indexed citations
9.
Biswas, Pooja, Muhammad Asif Naeem, Muhammad Hassaan Ali, et al.. (2018). Whole-Exome Sequencing Identifies Novel Variants that Co-segregates with Autosomal Recessive Retinal Degeneration in a Pakistani Pedigree. Advances in experimental medicine and biology. 1074. 219–228. 1 indexed citations
10.
Biswas, Pooja, Audrey C. Ko, David B. Granet, et al.. (2018). Transcriptome Analysis of Orbital Adipose Tissue in Active Thyroid Eye Disease Using Next Generation RNA Sequencing Technology. The Open Ophthalmology Journal. 12(1). 41–52. 14 indexed citations
11.
Biswas, Pooja, Jacque L. Duncan, Muhammad Ali, et al.. (2017). A mutation in IFT43 causes non-syndromic recessive retinal degeneration. Human Molecular Genetics. 26(23). 4741–4751. 10 indexed citations
12.
Biswas, Pooja, Jacque L. Duncan, Igor Kozak, et al.. (2017). Genetic analysis of 10 pedigrees with inherited retinal degeneration by exome sequencing and phenotype-genotype association. Physiological Genomics. 49(4). 216–229. 24 indexed citations
13.
Duncan, Jacque L., Pooja Biswas, Angel Soto‐Hermida, et al.. (2017). Whole Genome Sequencing Revealed Mutations in Two Independent Genes as the Underlying Cause of Retinal Degeneration in an Ashkenazi Jewish Pedigree. Genes. 8(9). 210–210. 13 indexed citations
14.
Biswas, Pooja, Pongali B. Raghavendra, Bruno Lamontagne, et al.. (2017). Identification of the genetic determinants responsible for retinal degeneration in families of Mexican descent. Ophthalmic Genetics. 39(1). 73–79. 7 indexed citations
15.
Biswas, Pooja, Venkata Ramana Murthy Chavali, Everett Stone, et al.. (2016). A missense mutation inASRGL1is involved in causing autosomal recessive retinal degeneration. Human Molecular Genetics. 25(12). ddw113–ddw113. 12 indexed citations
16.
Biswas, Pooja, Muhammad Asif Naeem, Justin Chu, et al.. (2015). Investigating the Molecular Basis of Retinal Degeneration in a Familial Cohort of Pakistani Decent by Exome Sequencing. PLoS ONE. 10(9). e0136561–e0136561. 30 indexed citations
17.
Duncan, Jacque L., Pooja Biswas, Igor Kozak, et al.. (2014). Ocular Phenotype of a Family with FAM161A-associated Retinal Degeneration. Ophthalmic Genetics. 37(1). 1–9. 15 indexed citations
18.
Biswas, Pooja, Jacque L. Duncan, Kari Branham, et al.. (2014). exomeSuite: Whole exome sequence variant filtering tool for rapid identification of putative disease causing SNVs/indels. Genomics. 103(2-3). 169–176. 16 indexed citations
19.
Biswas, Pooja, Pauline Lee, Shahid Y. Khan, et al.. (2013). Identification of causative mutations in consanguineous pedigrees from Pakistan with recessive retinal degeneration by whole exome analysis. Investigative Ophthalmology & Visual Science. 54(15). 3349–3349. 3 indexed citations
20.
Biswas, Pooja, et al.. (2011). A Temporal Analysis of Geographical Distances in Computer Science Collaborations. 657–660. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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