Piotr Grabarczyk

903 total citations
36 papers, 663 citations indexed

About

Piotr Grabarczyk is a scholar working on Molecular Biology, Immunology and Oncology. According to data from OpenAlex, Piotr Grabarczyk has authored 36 papers receiving a total of 663 indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Molecular Biology, 18 papers in Immunology and 13 papers in Oncology. Recurrent topics in Piotr Grabarczyk's work include T-cell and Retrovirus Studies (11 papers), CAR-T cell therapy research (11 papers) and Immune Cell Function and Interaction (7 papers). Piotr Grabarczyk is often cited by papers focused on T-cell and Retrovirus Studies (11 papers), CAR-T cell therapy research (11 papers) and Immune Cell Function and Interaction (7 papers). Piotr Grabarczyk collaborates with scholars based in Germany, Poland and China. Piotr Grabarczyk's co-authors include Grzegorz K. Przybylski, Christian A. Schmidt, Uwe Völker, Jacques J. M. van Dongen, Christian A. Schmidt, Stefan Nagel, Corinna Meyer, Letizia Venturini, Maren Depke and Michaela Scherr and has published in prestigious journals such as Blood, PLoS ONE and Molecular and Cellular Biology.

In The Last Decade

Piotr Grabarczyk

35 papers receiving 659 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Piotr Grabarczyk Germany 16 336 224 191 143 126 36 663
Teresa D’Altri Spain 10 450 1.3× 155 0.7× 99 0.5× 121 0.8× 52 0.4× 15 659
Francesco Boccalatte United States 12 536 1.6× 156 0.7× 193 1.0× 188 1.3× 29 0.2× 21 804
Maria J. Carnicer Spain 11 333 1.0× 148 0.7× 183 1.0× 52 0.4× 40 0.3× 18 611
Junli Yan Singapore 12 536 1.6× 193 0.9× 171 0.9× 195 1.4× 40 0.3× 22 786
Tomas Vilimas United States 8 572 1.7× 202 0.9× 202 1.1× 126 0.9× 193 1.5× 8 829
Munetake Shimabe Japan 11 498 1.5× 182 0.8× 83 0.4× 68 0.5× 64 0.5× 20 862
Brenda Verhaaf Netherlands 10 168 0.5× 161 0.7× 131 0.7× 28 0.2× 59 0.5× 12 460
Laki Buluwela United Kingdom 11 462 1.4× 268 1.2× 107 0.6× 46 0.3× 104 0.8× 16 761
Cyril Šálek Czechia 14 243 0.7× 311 1.4× 275 1.4× 74 0.5× 84 0.7× 47 728
Holger Kohlhammer Germany 9 393 1.2× 129 0.6× 203 1.1× 168 1.2× 58 0.5× 11 721

Countries citing papers authored by Piotr Grabarczyk

Since Specialization
Citations

This map shows the geographic impact of Piotr Grabarczyk's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Piotr Grabarczyk with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Piotr Grabarczyk more than expected).

Fields of papers citing papers by Piotr Grabarczyk

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Piotr Grabarczyk. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Piotr Grabarczyk. The network helps show where Piotr Grabarczyk may publish in the future.

Co-authorship network of co-authors of Piotr Grabarczyk

This figure shows the co-authorship network connecting the top 25 collaborators of Piotr Grabarczyk. A scholar is included among the top collaborators of Piotr Grabarczyk based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Piotr Grabarczyk. Piotr Grabarczyk is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Siebert, Nikolai, et al.. (2025). Affinity Affects the Functional Potency of Anti-GD2 Antibodies by Target-Mediated Drug Disposition. Cancers. 17(15). 2510–2510. 1 indexed citations
2.
Grabarczyk, Piotr, et al.. (2025). Double the Double: Revisiting BCL11B's Multimerization. Proteins Structure Function and Bioinformatics. 93(7). 1205–1211.
3.
4.
Grabarczyk, Piotr, et al.. (2021). Easy Expression and Purification of Fluorescent N-Terminal BCL11B CCHC Zinc Finger Domain. Molecules. 26(24). 7576–7576. 4 indexed citations
5.
Schulig, Lukas, Piotr Grabarczyk, Martin Kulke, et al.. (2021). Unveiling the N-Terminal Homodimerization of BCL11B by Hybrid Solvent Replica-Exchange Simulations. International Journal of Molecular Sciences. 22(7). 3650–3650. 3 indexed citations
6.
Bekeschus, Sander, Ramona Clemen, Lyubomir Haralambiev, et al.. (2020). The Plasma-Induced Leukemia Cell Death is Dictated by the ROS Chemistry and the HO-1/CXCL8 Axis. IEEE Transactions on Radiation and Plasma Medical Sciences. 5(3). 398–411. 9 indexed citations
7.
8.
Brandt, Jens van den, Sandra Lange, Grzegorz K. Przybylski, et al.. (2015). In Vivo Silencing of A20 via TLR9-Mediated Targeted SiRNA Delivery Potentiates Antitumor Immune Response. PLoS ONE. 10(9). e0135444–e0135444. 9 indexed citations
9.
Wu, Xiuli, Xinyu Wang, Zhenyi Jin, et al.. (2014). Specific Gamma Delta T Cells for Cellular Immunotherapy of EBV-Associated Diseases after Allo-HSCT By T-Cell Receptor Gene Modification. Blood. 124(21). 5811–5811. 1 indexed citations
10.
Zawada, M, Piotr Grabarczyk, Markus Möbs, et al.. (2013). Identification of Multiple Complex Rearrangements Associated with Deletions in the 6q23-27 Region in Sézary Syndrome. Journal of Investigative Dermatology. 133(11). 2617–2625. 8 indexed citations
11.
Shen, Qi, Si Chen, Lijian Yang, et al.. (2012). BCL11B suppression does not influence CD34+ cell differentiation and proliferation. Hematology. 17(6). 329–333. 7 indexed citations
12.
Chen, Si, Xin Huang, Shaohua Chen, et al.. (2012). The role of BCL11B in regulating the proliferation of human naive T cells. Human Immunology. 73(5). 456–464. 17 indexed citations
13.
Chen, Si, Qi Shen, Shaohua Chen, et al.. (2011). Down regulation of BCL11B expression inhibits proliferation and induces apoptosis in malignant T cells by BCL11B-935-siRNA. Hematology. 16(4). 236–242. 27 indexed citations
14.
Henrion, Ulrike, Meike Dahlhaus, B. Giese, et al.. (2010). The Activation of the Rat Insulin Gene II by BETA2 and PDX-1 in Rat Insulinoma Cells Is Repressed by Pax6. Molecular Endocrinology. 24(12). 2331–2342. 7 indexed citations
15.
Grabarczyk, Piotr, Viola Nähse, Grzegorz K. Przybylski, et al.. (2010). Increased Expression of Bcl11b Leads to Chemoresistance Accompanied by G1 Accumulation. PLoS ONE. 5(9). e12532–e12532. 25 indexed citations
16.
Nagel, Stefan, Letizia Venturini, Grzegorz K. Przybylski, et al.. (2009). NK-like homeodomain proteins activate NOTCH3-signaling in leukemic T-cells. BMC Cancer. 9(1). 371–371. 23 indexed citations
17.
Nagel, Stefan, Letizia Venturini, Grzegorz K. Przybylski, et al.. (2009). Activation of miR-17-92 by NK-like homeodomain proteins suppresses apoptosis via reduction of E2F1 in T-cell acute lymphoblastic leukemia. Leukemia & lymphoma. 50(1). 101–108. 61 indexed citations
18.
Grabarczyk, Piotr, Grzegorz K. Przybylski, Maren Depke, et al.. (2006). Inhibition of BCL11B expression leads to apoptosis of malignant but not normal mature T cells. Oncogene. 26(26). 3797–3810. 65 indexed citations
19.
Przybylski, Grzegorz K., et al.. (2006). The effect of a novel recombination between the homeobox gene NKX2-5 and the TRD locus in T-cell acute lymphoblastic leukemia on activation of the NKX2-5 gene.. PubMed. 91(3). 317–21. 32 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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