Tomas Vilimas

1.5k total citations
8 papers, 829 citations indexed

About

Tomas Vilimas is a scholar working on Molecular Biology, Oncology and Immunology. According to data from OpenAlex, Tomas Vilimas has authored 8 papers receiving a total of 829 indexed citations (citations by other indexed papers that have themselves been cited), including 4 papers in Molecular Biology, 4 papers in Oncology and 4 papers in Immunology. Recurrent topics in Tomas Vilimas's work include T-cell and Retrovirus Studies (2 papers), Immune Cell Function and Interaction (2 papers) and Pancreatic and Hepatic Oncology Research (1 paper). Tomas Vilimas is often cited by papers focused on T-cell and Retrovirus Studies (2 papers), Immune Cell Function and Interaction (2 papers) and Pancreatic and Hepatic Oncology Research (1 paper). Tomas Vilimas collaborates with scholars based in United States, Italy and Canada. Tomas Vilimas's co-authors include Iannis Aifantis, Adolfo A. Ferrando, Silvia Buonamici, Teresa Palomero, Benjamin J. Thompson, Maria Luisa Sulis, Giuseppe Basso, Malay Mandal, Christina Spaulding and Barbara L. Kee and has published in prestigious journals such as Nature Medicine, The Journal of Experimental Medicine and Blood.

In The Last Decade

Tomas Vilimas

8 papers receiving 819 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Tomas Vilimas United States	8	572	202	202	193	167	8	829
Carol Wai Canada	4	646 1.1×	178 0.9×	147 0.7×	194 1.0×	174 1.0×	5	879
Todd Ashworth United States	12	622 1.1×	134 0.7×	131 0.6×	117 0.6×	175 1.0×	13	851
Archana Agarwal United States	7	679 1.2×	154 0.8×	103 0.5×	219 1.1×	200 1.2×	18	971
Veena Krishnamoorthy United States	9	424 0.7×	201 1.0×	430 2.1×	178 0.9×	182 1.1×	11	889
Cédric S. Tremblay Australia	15	300 0.5×	122 0.6×	150 0.7×	198 1.0×	213 1.3×	32	584
Piotr Grabarczyk Germany	16	336 0.6×	191 0.9×	224 1.1×	126 0.7×	98 0.6×	36	663
Simon Raffel Germany	12	510 0.9×	145 0.7×	310 1.5×	84 0.4×	414 2.5×	27	920
Tetsuharu Shinjyo Japan	10	453 0.8×	181 0.9×	157 0.8×	46 0.2×	131 0.8×	13	703
Mark Y. Chiang United States	17	699 1.2×	231 1.1×	296 1.5×	231 1.2×	248 1.5×	32	1.2k
Eva Welinder Sweden	11	407 0.7×	107 0.5×	459 2.3×	103 0.5×	134 0.8×	14	790

Countries citing papers authored by Tomas Vilimas

Since Specialization

Citations

This map shows the geographic impact of Tomas Vilimas's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tomas Vilimas with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tomas Vilimas more than expected).

Fields of papers citing papers by Tomas Vilimas

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tomas Vilimas. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tomas Vilimas. The network helps show where Tomas Vilimas may publish in the future.

Co-authorship network of co-authors of Tomas Vilimas

This figure shows the co-authorship network connecting the top 25 collaborators of Tomas Vilimas. A scholar is included among the top collaborators of Tomas Vilimas based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tomas Vilimas. Tomas Vilimas is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Vilimas, Tomas. (2019). Measuring Tumor Mutational Burden Using Whole-Exome Sequencing. Methods in molecular biology. 2055. 63–91. 7 indexed citations

2.

Reddi, Alagarsamy Lakku, Priya Aggarwal, Charuta Ambardekar, et al.. (2010). Sprouty Proteins Inhibit Receptor-mediated Activation of Phosphatidylinositol-specific Phospholipase C. Molecular Biology of the Cell. 21(19). 3487–3496. 42 indexed citations

3.

Aifantis, Iannis, Tomas Vilimas, & Silvia Buonamici. (2007). Notches, NF-kBs and the Making of T Cell Leukemia. Cell Cycle. 6(4). 403–406. 13 indexed citations

4.

Thompson, Benjamin J., Silvia Buonamici, Maria Luisa Sulis, et al.. (2007). The SCFFBW7 ubiquitin ligase complex as a tumor suppressor in T cell leukemia. The Journal of Experimental Medicine. 204(8). 1825–1835. 363 indexed citations

5.

Aifantis, Iannis, Malay Mandal, Catherine M. Sawai, Adolfo A. Ferrando, & Tomas Vilimas. (2006). Regulation of T‐cell progenitor survival and cell‐cycle entry by the pre‐T‐cell receptor. Immunological Reviews. 209(1). 159–169. 83 indexed citations

6.

Reschly, Erica J., Christina Spaulding, Tomas Vilimas, et al.. (2006). Notch1 promotes survival of E2A-deficient T cell lymphomas through pre–T cell receptor–dependent and –independent mechanisms. Blood. 107(10). 4115–4121. 43 indexed citations

7.

Vilimas, Tomas, Joaquina Mascarenhas, Teresa Palomero, et al.. (2006). Targeting the NF-κB signaling pathway in Notch1-induced T-cell leukemia. Nature Medicine. 13(1). 70–77. 261 indexed citations

8.

Vilimas, Tomas, et al.. (2003). An early pharyngeal muscle enhancer from the Caenorhabditis elegans ceh-22 gene is targeted by the Forkhead factor PHA-4. Developmental Biology. 266(2). 388–398. 17 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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