Ping Lv

1.2k total citations
51 papers, 914 citations indexed

About

Ping Lv is a scholar working on Molecular Biology, Sensory Systems and Cancer Research. According to data from OpenAlex, Ping Lv has authored 51 papers receiving a total of 914 indexed citations (citations by other indexed papers that have themselves been cited), including 32 papers in Molecular Biology, 19 papers in Sensory Systems and 13 papers in Cancer Research. Recurrent topics in Ping Lv's work include Hearing, Cochlea, Tinnitus, Genetics (19 papers), Ion channel regulation and function (12 papers) and Cancer-related molecular mechanisms research (6 papers). Ping Lv is often cited by papers focused on Hearing, Cochlea, Tinnitus, Genetics (19 papers), Ion channel regulation and function (12 papers) and Cancer-related molecular mechanisms research (6 papers). Ping Lv collaborates with scholars based in China, United States and United Kingdom. Ping Lv's co-authors include Ebenezer N. Yamoah, Haitao Shen, Hongxia Fan, Xiaopei Zhao, Hyo Jeong Kim, Hua Tang, Min Liu, Dongguang Wei, Jingyi Fan and Xianghong Zhang and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Journal of Neuroscience.

In The Last Decade

Ping Lv

49 papers receiving 904 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Ping Lv China 18 560 336 217 93 85 51 914
Satoe Takahashi United States 15 490 0.9× 92 0.3× 235 1.1× 61 0.7× 118 1.4× 32 990
Xiaoyun Chen China 16 350 0.6× 109 0.3× 138 0.6× 26 0.3× 63 0.7× 32 691
Justin Ainscough United Kingdom 23 1.3k 2.4× 230 0.7× 134 0.6× 68 0.7× 93 1.1× 45 1.8k
Hak Hyun Jung South Korea 14 251 0.4× 52 0.2× 157 0.7× 39 0.4× 44 0.5× 26 549
Ken Hayashi Japan 14 625 1.1× 98 0.3× 55 0.3× 60 0.6× 181 2.1× 24 947
James Partin United States 12 303 0.5× 52 0.2× 108 0.5× 176 1.9× 56 0.7× 16 782
Magali Savignac France 22 565 1.0× 46 0.1× 190 0.9× 220 2.4× 67 0.8× 40 1.3k
Takeshi Kondo Japan 14 429 0.8× 141 0.4× 44 0.2× 52 0.6× 114 1.3× 25 763
David B. West United States 18 551 1.0× 69 0.2× 133 0.6× 67 0.7× 66 0.8× 25 1.2k
Nammi Park South Korea 14 407 0.7× 57 0.2× 77 0.4× 136 1.5× 31 0.4× 29 557

Countries citing papers authored by Ping Lv

Since Specialization
Citations

This map shows the geographic impact of Ping Lv's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ping Lv with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ping Lv more than expected).

Fields of papers citing papers by Ping Lv

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ping Lv. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ping Lv. The network helps show where Ping Lv may publish in the future.

Co-authorship network of co-authors of Ping Lv

This figure shows the co-authorship network connecting the top 25 collaborators of Ping Lv. A scholar is included among the top collaborators of Ping Lv based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ping Lv. Ping Lv is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Wang, Shengnan, Qi Fang, Sai Shi, et al.. (2025). LRRC8A Regulates Outer Hair Cell Volume and Electromotility and is Required for Hearing. Advanced Science. 12(41). e10477–e10477.
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Liang, Xiaoyan, Jiayu Zhang, Delin Zhu, et al.. (2024). Dietary Aflatoxin G1 exposure causes an imbalance between pulmonary tissue-resident alveolar macrophages and monocyte-derived macrophages in both mother and offspring mice. Ecotoxicology and Environmental Safety. 285. 117082–117082. 2 indexed citations
5.
Duan, Chenyang, Jingjing Cao, Delin Zhu, et al.. (2023). AFG1-induced TNF-α-mediated inflammation enhances gastric epithelial cell injury via CYP2E1. Food and Chemical Toxicology. 176. 113756–113756. 8 indexed citations
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Zhang, Haiwei, Shengnan Wang, Fei Wang, et al.. (2022). Downregulation of REST in the cochlea contributes to age-related hearing loss via the p53 apoptosis pathway. Cell Death and Disease. 13(4). 343–343. 10 indexed citations
8.
Yang, Jing, Guoqiang Gu, Qiong Wang, et al.. (2021). Sensorineural Hearing Loss and Mitochondrial Apoptosis of Cochlear Spiral Ganglion Neurons in Fibroblast Growth Factor 13 Knockout Mice. Frontiers in Cellular Neuroscience. 15. 658586–658586. 7 indexed citations
9.
Zhang, Haiwei, Jiaxi Liu, Fei Wang, et al.. (2021). Association between Cav3 channel upregulation in spiral ganglion neurons and age-dependent hearing loss. Experimental Gerontology. 151. 111429–111429. 6 indexed citations
10.
Liu, Xiaoyi, Xiuqing Wang, Wenli Guo, et al.. (2020). High mobility group box-1 protects against Aflatoxin G1-induced pulmonary epithelial cell damage in the lung inflammatory environment. Toxicology Letters. 331. 92–101. 11 indexed citations
11.
Lv, Ping, et al.. (2019). Nobiletin Attenuates Cell Proliferation by Modulating the Activating Protein-1 Signaling Pathway in 7,12-Dimethylbenz[a]anthracene-Induced Mammary Carcinogenesis. Journal of Environmental Pathology Toxicology and Oncology. 39(1). 13–21. 5 indexed citations
12.
Lv, Jing, Hongxia Fan, Xiaopei Zhao, et al.. (2016). Long non-coding RNA Unigene56159 promotes epithelial–mesenchymal transition by acting as a ceRNA of miR-140-5p in hepatocellular carcinoma cells. Cancer Letters. 382(2). 166–175. 112 indexed citations
13.
Shen, Haitao, et al.. (2016). The role of Na+, K+-ATPase in the hypoxic vasoconstriction in isolated rat basilar artery. Vascular Pharmacology. 81. 53–60. 8 indexed citations
14.
Lv, Ping, Hyo Jeong Kim, Jeong-Han Lee, et al.. (2014). Genetic, Cellular, and Functional Evidence for Ca2+Inflow through Cav1.2 and Cav1.3 Channels in Murine Spiral Ganglion Neurons. Journal of Neuroscience. 34(21). 7383–7393. 15 indexed citations
15.
Yang, Juanmei, et al.. (2012). Functional Features of Trans-Differentiated Hair Cells Mediated by Atoh1 Reveals a Primordial Mechanism. Journal of Neuroscience. 32(11). 3712–3725. 24 indexed citations
16.
Shen, Haitao, Ping Lv, Xin Xing, et al.. (2012). Impairment of alveolar type-II cells involved in the toxicity of Aflatoxin G1 in rat lung. Food and Chemical Toxicology. 50(9). 3222–3228. 11 indexed citations
17.
Levic, Snezana, Ping Lv, & Ebenezer N. Yamoah. (2011). The Activity of Spontaneous Action Potentials in Developing Hair Cells Is Regulated by Ca2+-Dependence of a Transient K+ Current. PLoS ONE. 6(12). e29005–e29005. 6 indexed citations
18.
Kim, Hyo Jeong, et al.. (2010). Cellular and Molecular Mechanisms of Autosomal Dominant Form of Progressive Hearing Loss, DFNA2. Journal of Biological Chemistry. 286(2). 1517–1527. 31 indexed citations
19.
Lv, Ping, Dongguang Wei, & Ebenezer N. Yamoah. (2010). Kv7-type Channel Currents in Spiral Ganglion Neurons. Journal of Biological Chemistry. 285(45). 34699–34707. 45 indexed citations
20.
Rodríguez‐Contreras, Adrián, et al.. (2008). Effects of Strontium on the Permeation and Gating Phenotype of Calcium Channels in Hair Cells. Journal of Neurophysiology. 100(4). 2115–2124. 9 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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