Pilar Mur

1.4k total citations
37 papers, 648 citations indexed

About

Pilar Mur is a scholar working on Pathology and Forensic Medicine, Molecular Biology and Cancer Research. According to data from OpenAlex, Pilar Mur has authored 37 papers receiving a total of 648 indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Pathology and Forensic Medicine, 15 papers in Molecular Biology and 15 papers in Cancer Research. Recurrent topics in Pilar Mur's work include Genetic factors in colorectal cancer (17 papers), Cancer Genomics and Diagnostics (14 papers) and DNA Repair Mechanisms (8 papers). Pilar Mur is often cited by papers focused on Genetic factors in colorectal cancer (17 papers), Cancer Genomics and Diagnostics (14 papers) and DNA Repair Mechanisms (8 papers). Pilar Mur collaborates with scholars based in Spain, United States and United Kingdom. Pilar Mur's co-authors include Manuela Mollejo, Bárbara Meléndez, J. Borja, Laura Valle, P. A. Galindo, Feo F, Ángel Rodríguez de Lope, Juan A. Rey, Concepción Fiaño and Juan F. Garcı́a and has published in prestigious journals such as Gut, Scientific Reports and Journal of Allergy and Clinical Immunology.

In The Last Decade

Pilar Mur

36 papers receiving 630 citations

Peers

Pilar Mur
Limin Gao China
Canan Cacına Türkiye
Henry Sánchez United States
Valerie Atizado Saudi Arabia
Yifang Hu China
Hele Everaus Estonia
Ezra Baraban United States
Carl Simon Shelley United States
Pilar Mur
Citations per year, relative to Pilar Mur Pilar Mur (= 1×) peers Karolina Nemes

Countries citing papers authored by Pilar Mur

Since Specialization
Citations

This map shows the geographic impact of Pilar Mur's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Pilar Mur with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Pilar Mur more than expected).

Fields of papers citing papers by Pilar Mur

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Pilar Mur. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Pilar Mur. The network helps show where Pilar Mur may publish in the future.

Co-authorship network of co-authors of Pilar Mur

This figure shows the co-authorship network connecting the top 25 collaborators of Pilar Mur. A scholar is included among the top collaborators of Pilar Mur based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Pilar Mur. Pilar Mur is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Terradas, Mariona, Pilar Mur, Chiara Maria Lavinia Löffler, et al.. (2025). Constitutional epimutations in LTBP4, a component of the TGF-β signaling, and in BRCA1, as potential drivers of early-onset colorectal cancer. Clinical Epigenetics. 17(1). 183–183. 1 indexed citations
2.
Seplyarskiy, Vladimir B., Mariona Terradas, Pilar Mur, et al.. (2024). Discovery of recessive effect of human polymerase δ proofreading deficiency through mutational analysis of POLD1-mutated normal and cancer cells. European Journal of Human Genetics. 32(7). 837–845. 5 indexed citations
3.
Andrake, Mark, Pilar Mur, Joanne Xiu, et al.. (2024). Exploring Co-occurring POLE Exonuclease and Non-exonuclease Domain Mutations and Their Impact on Tumor Mutagenicity. Cancer Research Communications. 4(1). 213–225. 6 indexed citations
4.
Valle, Laura, Lior H. Katz, Andrew Latchford, et al.. (2023). Position statement of the International Society for Gastrointestinal Hereditary Tumours (InSiGHT) on APC I1307K and cancer risk. Journal of Medical Genetics. 60(11). 1035–1043. 12 indexed citations
5.
Mur, Pilar, Sandra García‐Mulero, Lorena Magraner-Pardo, et al.. (2023). Recommendations for the classification of germline variants in the exonuclease domain of POLE and POLD1. Genome Medicine. 15(1). 85–85. 8 indexed citations
6.
Singh, Ashish, Pilar Mur, Rosa Aligué, et al.. (2023). Characterization of POLE c.1373A > T p.(Tyr458Phe), causing high cancer risk. Molecular Genetics and Genomics. 298(3). 555–566. 2 indexed citations
7.
Mur, Pilar, Mariona Terradas, Sandra García‐Mulero, et al.. (2022). Potential Involvement of NSD1, KRT24 and ACACA in the Genetic Predisposition to Colorectal Cancer. Cancers. 14(3). 699–699. 2 indexed citations
8.
Mur, Pilar, Núria Bonifaci, Anna Díez-Villanueva, et al.. (2021). Non-Lynch Familial and Early-Onset Colorectal Cancer Explained by Accumulation of Low-Risk Genetic Variants. Cancers. 13(15). 3857–3857. 5 indexed citations
9.
Daca-Álvarez, María, et al.. (2021). The Inherited and Familial Component of Early-Onset Colorectal Cancer. Cells. 10(3). 710–710. 56 indexed citations
10.
Mur, Pilar, Lorena Magraner-Pardo, Sandra García‐Mulero, et al.. (2021). Solving the enigma of POLD1 p.V295M as a potential cause of increased cancer risk. European Journal of Human Genetics. 30(4). 485–489. 1 indexed citations
11.
Terradas, Mariona, Pilar Mur, Sami Belhadj, et al.. (2020). TP53, a gene for colorectal cancer predisposition in the absence of Li-Fraumeni-associated phenotypes. Gut. 70(6). 1139–1146. 11 indexed citations
12.
Belhadj, Sami, Pilar Mur, M. Henar Alonso, et al.. (2019). NTHL1 biallelic mutations seldom cause colorectal cancer, serrated polyposis or a multi-tumor phenotype, in absence of colorectal adenomas. Scientific Reports. 9(1). 9020–9020. 18 indexed citations
13.
Mur, Pilar, Ann‐Sofie Jemth, Nuno Amaral, et al.. (2018). Germline variation in the oxidative DNA repair genesNUDT1andOGG1is not associated with hereditary colorectal cancer or polyposis. Human Mutation. 39(9). 1214–1225. 5 indexed citations
14.
Mur, Pilar, Susanna Aussó, Gemma Aiza, et al.. (2016). Scarce evidence of the causal role of germline mutations in UNC5C in hereditary colorectal cancer and polyposis. Scientific Reports. 6(1). 20697–20697. 6 indexed citations
15.
Mur, Pilar, Manuela Mollejo, Yolanda Ruano, et al.. (2013). Codeletion of 1p and 19q determines distinct gene methylation and expression profiles in IDH-mutated oligodendroglial tumors. Acta Neuropathologica. 126(2). 277–289. 38 indexed citations
16.
Campos‐Martín, Yolanda, Pilar Mur, Concepción Fiaño, et al.. (2012). Genetic Alterations Associated With Progression and Recurrence in Meningiomas. Journal of Neuropathology & Experimental Neurology. 71(10). 882–893. 44 indexed citations
17.
F, Feo, Pilar Mur, Elisa Gómez, et al.. (2007). Rhinoconjunctivitis and asthma caused by corn plant (Dracaena fragrans). Annals of Allergy Asthma & Immunology. 98(3). 290–293. 2 indexed citations
18.
F, Feo, Pilar Mur, Domingo Barber, et al.. (2002). Occupational rhinoconjunctivitis and asthma in a wool worker caused by Dermestidae spp.. Allergy. 57(12). 1191–1194. 9 indexed citations
19.
F, Feo, Pilar Mur, P. A. Galindo, et al.. (2001). Rhinoconjunctivitis and occupational asthma caused byDiplotaxis erucoides (wall rocket). Journal of Allergy and Clinical Immunology. 108(1). 125–127. 5 indexed citations
20.
Mur, Pilar, Feo F, M. Lombardero, et al.. (2001). Allergy to linden pollen (Tilia cordata). Allergy. 56(5). 457–458. 22 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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