Javier S. Castresana

3.0k total citations
115 papers, 2.3k citations indexed

About

Javier S. Castresana is a scholar working on Molecular Biology, Neurology and Genetics. According to data from OpenAlex, Javier S. Castresana has authored 115 papers receiving a total of 2.3k indexed citations (citations by other indexed papers that have themselves been cited), including 66 papers in Molecular Biology, 35 papers in Neurology and 28 papers in Genetics. Recurrent topics in Javier S. Castresana's work include Epigenetics and DNA Methylation (29 papers), Glioma Diagnosis and Treatment (26 papers) and Hedgehog Signaling Pathway Studies (23 papers). Javier S. Castresana is often cited by papers focused on Epigenetics and DNA Methylation (29 papers), Glioma Diagnosis and Treatment (26 papers) and Hedgehog Signaling Pathway Studies (23 papers). Javier S. Castresana collaborates with scholars based in Spain, United States and India. Javier S. Castresana's co-authors include Juan A. Rey, Mehdi Hayat Shahi, Carlos Barrios, Andris Kreicbergs, Xing Fan, Mari‐Paz Rubio, Ángel Pestaña, Bárbara Meléndez, Miguel Á. Idoate and José M. de Campos and has published in prestigious journals such as SHILAP Revista de lepidopterología, PLoS ONE and JNCI Journal of the National Cancer Institute.

In The Last Decade

Javier S. Castresana

108 papers receiving 2.3k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Javier S. Castresana Spain 30 1.4k 708 592 514 426 115 2.3k
Erik J. Uhlmann United States 30 1.7k 1.2× 734 1.0× 447 0.8× 383 0.7× 243 0.6× 73 3.4k
Arend Koch Germany 34 1.4k 1.0× 415 0.6× 421 0.7× 848 1.6× 333 0.8× 85 2.6k
Meena Gujrati United States 35 1.5k 1.0× 652 0.9× 1.0k 1.7× 685 1.3× 260 0.6× 87 3.0k
Osamu Tachibana Japan 23 1.2k 0.8× 571 0.8× 401 0.7× 1.1k 2.1× 437 1.0× 105 2.7k
Rajeev Vibhakar United States 33 2.1k 1.5× 660 0.9× 760 1.3× 628 1.2× 230 0.5× 98 2.9k
Wiesława Grajkowska Poland 27 1.9k 1.4× 406 0.6× 650 1.1× 1.0k 2.0× 430 1.0× 174 3.3k
Bárbara Meléndez Spain 22 859 0.6× 439 0.6× 421 0.7× 442 0.9× 228 0.5× 66 1.6k
Brent A. Orr United States 34 1.8k 1.2× 498 0.7× 640 1.1× 1.2k 2.4× 537 1.3× 125 3.1k
J. M. Bruner United States 26 1.0k 0.7× 600 0.8× 492 0.8× 742 1.4× 194 0.5× 48 2.5k
Gianluigi Zona Italy 28 1.1k 0.8× 1.3k 1.8× 498 0.8× 666 1.3× 392 0.9× 95 3.2k

Countries citing papers authored by Javier S. Castresana

Since Specialization
Citations

This map shows the geographic impact of Javier S. Castresana's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Javier S. Castresana with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Javier S. Castresana more than expected).

Fields of papers citing papers by Javier S. Castresana

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Javier S. Castresana. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Javier S. Castresana. The network helps show where Javier S. Castresana may publish in the future.

Co-authorship network of co-authors of Javier S. Castresana

This figure shows the co-authorship network connecting the top 25 collaborators of Javier S. Castresana. A scholar is included among the top collaborators of Javier S. Castresana based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Javier S. Castresana. Javier S. Castresana is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Fox, David A., et al.. (2025). CD6 in Human Disease. Cells. 14(4). 272–272.
2.
Shahi, Mehdi Hayat, et al.. (2024). Mesenchymal-Stem-Cell-Based Therapy against Gliomas. Cells. 13(7). 617–617. 6 indexed citations
3.
Shahi, Mehdi Hayat, et al.. (2024). The role of the adenylate kinase 5 gene in various diseases and cancer. Journal of Clinical and Translational Science. 8(1). e96–e96. 2 indexed citations
4.
Afzal, Mohammad, et al.. (2023). A Comprehensive Review of miRNAs and Their Epigenetic Effects in Glioblastoma. Cells. 12(12). 1578–1578. 13 indexed citations
5.
Castresana, Javier S., et al.. (2022). Role of Circular RNA in Brain Tumor Development. Cells. 11(14). 2130–2130. 18 indexed citations
7.
Castresana, Javier S., et al.. (2021). The Role of Side Population Cells and Hypoxia in Resistance to Chemotherapy in Astrocytoma Cell Lines. Journal of Carcinogenesis & Mutagenesis. 12(3). 1–20.
8.
Blanco‐Luquin, Idoia, et al.. (2021). In Vitro Assessment of the Role of p53 on Chemotherapy Treatments in Neuroblastoma Cell Lines. Pharmaceuticals. 14(11). 1184–1184. 6 indexed citations
9.
Castresana, Javier S., et al.. (2021). Glioblastoma and MiRNAs. Cancers. 13(7). 1581–1581. 32 indexed citations
10.
Castresana, Javier S., et al.. (2021). Is an Integrative Model of Neurotheology Possible?. Religions. 12(4). 277–277.
11.
Rodríguez-Losada, Noela, et al.. (2020). Overexpression of alpha-synuclein promotes both cell proliferation and cell toxicity in human SH-SY5Y neuroblastoma cells. Journal of Advanced Research. 23. 37–45. 28 indexed citations
12.
Meléndez, Bárbara, et al.. (2018). Panobinostat Potentiates Temozolomide Effects and Reverses Epithelial–Mesenchymal Transition in Glioblastoma Cells. Epigenomes. 2(1). 5–5. 7 indexed citations
13.
Ordóñez, Raquel, Gabriel Gallo-Oller, Justine Guégan, et al.. (2014). Genome-Wide Microarray Expression and Genomic Alterations by Array-CGH Analysis in Neuroblastoma Stem-Like Cells. PLoS ONE. 9(11). e113105–e113105. 4 indexed citations
14.
Shahi, Mehdi Hayat, Idoya Zazpe, Mohammad Afzal, et al.. (2014). Epigenetic regulation of human hedgehog interacting protein in glioma cell lines and primary tumor samples. Tumor Biology. 36(4). 2383–2391. 12 indexed citations
15.
Torres‐Martín, Miguel, Luis Lassaletta, José M. de Campos, et al.. (2013). Microarray analysis of gene expression in vestibular schwannomas reveals SPP1/MET signaling pathway and androgen receptor deregulation. International Journal of Oncology. 42(3). 848–862. 47 indexed citations
16.
Taşpınar, Mehmet, Mevci̇ Özdemi̇r, Tülin Özkan, et al.. (2013). Effect of lomeguatrib–temozolomide combination on MGMT promoter methylation and expression in primary glioblastoma tumor cells. Tumor Biology. 34(3). 1935–1947. 15 indexed citations
17.
Shahi, Mehdi Hayat, Subrata Sinha, Mohammad Afzal, & Javier S. Castresana. (2009). Role of Sonic hedgehog signaling pathway in neuroblastoma development. 1(4). 0–0. 4 indexed citations
18.
Shahi, Mehdi Hayat, Mohammad Afzal, Subrata Sinha, & Javier S. Castresana. (2008). Aberrant promoter methylation of SFRP1 (Secreted Frizzled-Related Protein1) gene in medulloblastoma and glioblastoma.. Clinical Cancer Research. 14. 1 indexed citations
19.
Enguita, Mónica, et al.. (2007). Expression of neural stem cell markers in glioblastoma, medulloblastoma and neuroblastoma cell lines. Cancer Research. 67. 1280–1280. 1 indexed citations
20.
Rubio, Mari‐Paz, et al.. (1994). P-glycoprotein, HER-2/neu, and Mutant p53 Expression in Human Gynecologic Tumors. JNCI Journal of the National Cancer Institute. 86(11). 850–855. 56 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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