Pia Rossi

881 total citations
10 papers, 491 citations indexed

About

Pia Rossi is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Neurology. According to data from OpenAlex, Pia Rossi has authored 10 papers receiving a total of 491 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Molecular Biology, 5 papers in Cellular and Molecular Neuroscience and 5 papers in Neurology. Recurrent topics in Pia Rossi's work include Neuroscience and Neuropharmacology Research (4 papers), Cellular transport and secretion (3 papers) and Ion channel regulation and function (2 papers). Pia Rossi is often cited by papers focused on Neuroscience and Neuropharmacology Research (4 papers), Cellular transport and secretion (3 papers) and Ion channel regulation and function (2 papers). Pia Rossi collaborates with scholars based in Italy, United Kingdom and Spain. Pia Rossi's co-authors include Anna Corradi, Fabio Benfenati, Antonella Marte, Flavia Valtorta, Bruno Sterlini, Manuela Fadda, Anna Fassio, Anna Pittaluga, Aldamaria Puliti and Franco Onofri and has published in prestigious journals such as Journal of Biological Chemistry, Brain and Journal of Neurochemistry.

In The Last Decade

Pia Rossi

10 papers receiving 489 citations

Peers

Pia Rossi

MB

CMN

GENET

PMH

NEURO

Bruno Sterlini Italy

Georgia Karadima Greece

Alessandra Folci Italy

Laurie P. Sutton United States

Eva M. Pérez-Villegas Spain

Inge A. Meijer Canada

Benedetta Terragni Italy

Rebecca Brulet United States

Fabrizia Claudia Guarnieri Italy

Bruno Sterlini Italy

Pia Rossi

208 ×1.0

MB

172 ×0.8

CMN

169 ×1.1

GENET

200 ×1.8

PMH

57 ×0.6

NEURO

Citations per year, relative to Pia Rossi Pia Rossi (= 1×) peers Bruno Sterlini

Countries citing papers authored by Pia Rossi

Since Specialization

Citations

This map shows the geographic impact of Pia Rossi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Pia Rossi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Pia Rossi more than expected).

Fields of papers citing papers by Pia Rossi

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Pia Rossi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Pia Rossi. The network helps show where Pia Rossi may publish in the future.

Co-authorship network of co-authors of Pia Rossi

This figure shows the co-authorship network connecting the top 25 collaborators of Pia Rossi. A scholar is included among the top collaborators of Pia Rossi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Pia Rossi. Pia Rossi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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10 of 10 papers shown

1.

Fruscione, Floriana, Pierluigi Valente, Bruno Sterlini, et al.. (2018). PRRT2 controls neuronal excitability by negatively modulating Na+ channel 1.2/1.6 activity. Brain. 141(4). 1000–1016. 91 indexed citations

2.

Rossi, Pia, Bruno Sterlini, Enrico Castroflorio, et al.. (2016). A Novel Topology of Proline-rich Transmembrane Protein 2 (PRRT2). Journal of Biological Chemistry. 291(12). 6111–6123. 52 indexed citations

3.

Valente, Pierluigi, Enrico Castroflorio, Pia Rossi, et al.. (2016). PRRT2 Is a Key Component of the Ca2+-Dependent Neurotransmitter Release Machinery. Cell Reports. 15(1). 117–131. 109 indexed citations

4.

Corradi, Anna, Manuela Fadda, Amélie Piton, et al.. (2013). SYN2 is an autism predisposing gene: loss-of-function mutations alter synaptic vesicle cycling and axon outgrowth. Human Molecular Genetics. 23(1). 90–103. 65 indexed citations

5.

Milanese, Marco, Francesco Giribaldi, Marcello Melone, et al.. (2013). Knocking down metabotropic glutamate receptor 1 improves survival and disease progression in the SOD1G93A mouse model of amyotrophic lateral sclerosis. Neurobiology of Disease. 64. 48–59. 37 indexed citations

6.

Rossi, Pia, Ilaria Musante, Maria Summa, et al.. (2012). Compensatory Molecular and Functional Mechanisms in Nervous System of the Grm1crv4 Mouse Lacking the mGlu1 Receptor: A Model for Motor Coordination Deficits. Cerebral Cortex. 23(9). 2179–2189. 21 indexed citations

7.

Giribaldi, Francesco, Marco Milanese, Tiziana Bonifacino, et al.. (2012). Group I metabotropic glutamate autoreceptors induce abnormal glutamate exocytosis in a mouse model of amyotrophic lateral sclerosis. Neuropharmacology. 66. 253–263. 35 indexed citations

8.

Prisco, Silvia Di, et al.. (2012). RANTES‐mediated control of excitatory amino acid release in mouse spinal cord. Journal of Neurochemistry. 121(3). 428–437. 39 indexed citations

9.

Puliti, Aldamaria, Pia Rossi, Gianluca Caridi, et al.. (2011). Albuminuria and Glomerular Damage in Mice Lacking the Metabotropic Glutamate Receptor 1. American Journal Of Pathology. 178(3). 1257–1269. 25 indexed citations

10.

Rossi, Pia, Antonio Terracciano, Manuela Priolo, et al.. (2009). The metabotropic glutamate receptor 1, GRM1: evaluation as a candidate gene for inherited forms of cerebellar ataxia. Journal of Neurology. 257(4). 598–602. 17 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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