Philip M. Barger

4.4k total citations · 1 hit paper
46 papers, 3.6k citations indexed

About

Philip M. Barger is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Surgery. According to data from OpenAlex, Philip M. Barger has authored 46 papers receiving a total of 3.6k indexed citations (citations by other indexed papers that have themselves been cited), including 28 papers in Molecular Biology, 23 papers in Cardiology and Cardiovascular Medicine and 8 papers in Surgery. Recurrent topics in Philip M. Barger's work include Cardiac Fibrosis and Remodeling (11 papers), Peroxisome Proliferator-Activated Receptors (10 papers) and Mitochondrial Function and Pathology (9 papers). Philip M. Barger is often cited by papers focused on Cardiac Fibrosis and Remodeling (11 papers), Peroxisome Proliferator-Activated Receptors (10 papers) and Mitochondrial Function and Pathology (9 papers). Philip M. Barger collaborates with scholars based in United States, Brazil and Mexico. Philip M. Barger's co-authors include Daniel P. Kelly, Daniel P. Kelly, Attila Kovács, John J. Lehman, Jeffrey E. Saffitz, Denis M. Medeiros, Douglas L. Mann, Carla J. Weinheimer, Teresa C. Leone and Jon M. Brandt and has published in prestigious journals such as Journal of Biological Chemistry, Circulation and Journal of Clinical Investigation.

In The Last Decade

Philip M. Barger

46 papers receiving 3.6k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Peroxisome proliferator–activated receptor γ coactivator-1 promotes cardiac mitochondrial biogenesis

2000 1.1k citations John J. Lehman, Philip M. Barger et al. Journal of Clinical Investigation profile →

Peers

Philip M. Barger

CCM

PHYSI

EPIDE

SURGE

Yi Zhu United States

Attila Kovács United States

Takeshi Marumo Japan

Clifford D.L. Folmes United States

John R. Hagaman United States

Xiangru Lu Canada

Ferhaan Ahmad United States

Yukio Hiroi Japan

Norimichi Koitabashi Japan

Sandrine Horman Belgium

Yi Zhu United States

Philip M. Barger

1.7k ×0.8

659 ×0.5

CCM

1.3k ×1.0

PHYSI

815 ×2.0

EPIDE

496 ×1.3

SURGE

Citations per year, relative to Philip M. Barger Philip M. Barger (= 1×) peers Yi Zhu

Countries citing papers authored by Philip M. Barger

Since Specialization

Citations

This map shows the geographic impact of Philip M. Barger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Philip M. Barger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Philip M. Barger more than expected).

Fields of papers citing papers by Philip M. Barger

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Philip M. Barger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Philip M. Barger. The network helps show where Philip M. Barger may publish in the future.

Co-authorship network of co-authors of Philip M. Barger

This figure shows the co-authorship network connecting the top 25 collaborators of Philip M. Barger. A scholar is included among the top collaborators of Philip M. Barger based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Philip M. Barger. Philip M. Barger is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Evans, Sarah, Carla J. Weinheimer, Attila Kovács, et al.. (2020). Ischemia reperfusion injury provokes adverse left ventricular remodeling in dysferlin-deficient hearts through a pathway that involves TIRAP dependent signaling. Scientific Reports. 10(1). 14129–14129. 8 indexed citations

Verma, Amanda, Diana L. Gray, Jeannie C. Kelly, et al.. (2019). AORTOPATHY AND PREGNANCY: IT TAKES A VILLAGE. Journal of the American College of Cardiology. 73(9). 2527–2527. 1 indexed citations

Lindley, Kathryn J., et al.. (2018). EVALUATION OF CHA2DS2-VASC RISK STRATIFICATION TOOL IN PREGNANT WOMEN WITH ATRIAL FIBRILLATION OR ATRIAL FLUTTER. Journal of the American College of Cardiology. 71(11). A496–A496. 2 indexed citations

Evans, Sarah, Huei‐Ping Tzeng, Deborah J. Veis, et al.. (2018). TNF receptor–activated factor 2 mediates cardiac protection through noncanonical NF-κB signaling. JCI Insight. 3(3). 18 indexed citations

Adamo, Luigi, Cibele Rocha‐Resende, Scot J. Matkovich, et al.. (2018). Modulation of subsets of cardiac B lymphocytes improves cardiac function after acute injury. JCI Insight. 3(11). 71 indexed citations

Topkara, Veli K., Kari T. Chambers, Kai‐Chien Yang, et al.. (2016). Functional significance of the discordance between transcriptional profile and left ventricular structure/function during reverse remodeling. JCI Insight. 1(4). e86038–e86038. 27 indexed citations

Ma, Xiucui, Kai‐Chun Yang, Haiyan Liu, et al.. (2013). Abstract 12198: TRAF2 Coordinates With PARKIN to Mediate Mitochondrial Autophagy in Cardiomyocytes. Circulation. 128. 1 indexed citations

Zhang, Weili, Kory J. Lavine, Slava Epelman, et al.. (2012). Abstract 11268: Damage Associated Molecular Patterns (DAMPs) Activate Cardiac Fibroblasts Through Toll-Like Receptor 4: Implications for Myocardial Fibrosis. Circulation. 126. 1 indexed citations

Mann, Douglas L., Philip M. Barger, & Daniel Burkhoff. (2012). Myocardial Recovery and the Failing Heart. Journal of the American College of Cardiology. 60(24). 2465–2472. 190 indexed citations

10.

Zhang, Weili, Amanda L. Chancey, Huei‐Ping Tzeng, et al.. (2011). The Development of Myocardial Fibrosis in Transgenic Mice With Targeted Overexpression of Tumor Necrosis Factor Requires Mast Cell–Fibroblast Interactions. Circulation. 124(19). 2106–2116. 91 indexed citations

11.

Topkara, Veli K., et al.. (2010). Therapeutic targeting of innate immunity in the failing heart. Journal of Molecular and Cellular Cardiology. 51(4). 594–599. 34 indexed citations

12.

Thakker, Geeta D., Nikolaos G. Frangogiannis, Paweł Zymek, et al.. (2008). Increased Myocardial Susceptibility to Repetitive Ischemia With High‐fat diet‐induced Obesit. Obesity. 16(12). 2593–2600. 29 indexed citations

13.

Sekiguchi, Kenichi, Qi Tian, Masakuni Ishiyama, et al.. (2006). Inhibition of PPAR-α activity in mice with cardiac-restricted expression of tumor necrosis factor: potential role of TGF-β/Smad3. American Journal of Physiology-Heart and Circulatory Physiology. 292(3). H1443–H1451. 32 indexed citations

14.

Barger, Philip M., et al.. (2001). p38 Mitogen-activated Protein Kinase Activates Peroxisome Proliferator-activated Receptor α. Journal of Biological Chemistry. 276(48). 44495–44501. 221 indexed citations

15.

Barger, Philip M., Jon M. Brandt, Teresa C. Leone, Carla J. Weinheimer, & Daniel P. Kelly. (2000). Deactivation of peroxisome proliferator–activated receptor-α during cardiac hypertrophic growth. Journal of Clinical Investigation. 105(12). 1723–1730. 382 indexed citations

16.

Lehman, John J., Philip M. Barger, Attila Kovács, et al.. (2000). Peroxisome proliferator–activated receptor γ coactivator-1 promotes cardiac mitochondrial biogenesis. Journal of Clinical Investigation. 106(7). 847–856. 1058 indexed citations breakdown →

17.

Barger, Philip M. & Daniel P. Kelly. (2000). PPAR Signaling in the Control of Cardiac Energy Metabolism. Trends in Cardiovascular Medicine. 10(6). 238–245. 401 indexed citations

18.

Barger, Philip M. & Daniel P. Kelly. (1999). Fatty Acid Utilization in the Hypertrophied and Failing Heart: Molecular Regulatory Mechanisms. The American Journal of the Medical Sciences. 318(1). 36–36. 149 indexed citations

19.

Cresci, Sharon, et al.. (1996). Transcriptional Control of a Nuclear Gene Encoding a Mitochondrial Fatty Acid Oxidation Enzyme in Transgenic Mice: Role for Nuclear Receptors in Cardiac and Brown Adipose Expression. Molecular and Cellular Biology. 16(8). 4043–4051. 64 indexed citations

20.

Tomlinson, Beianka & Philip M. Barger. (1987). A test of the punctuated-cycling hypothesis in Ambystoma forelimb regenerates: the roles of animal size, limb innervation, and the aneurogenic condition. Differentiation. 35(1). 6–15. 5 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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