Peter V. DiStefano

1.3k total citations
17 papers, 640 citations indexed

About

Peter V. DiStefano is a scholar working on Molecular Biology, Neurology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Peter V. DiStefano has authored 17 papers receiving a total of 640 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 7 papers in Neurology and 4 papers in Cellular and Molecular Neuroscience. Recurrent topics in Peter V. DiStefano's work include Neuroscience and Neuropharmacology Research (4 papers), Neurotransmitter Receptor Influence on Behavior (4 papers) and Vascular Malformations Diagnosis and Treatment (4 papers). Peter V. DiStefano is often cited by papers focused on Neuroscience and Neuropharmacology Research (4 papers), Neurotransmitter Receptor Influence on Behavior (4 papers) and Vascular Malformations Diagnosis and Treatment (4 papers). Peter V. DiStefano collaborates with scholars based in United States, Canada and Netherlands. Peter V. DiStefano's co-authors include Kenneth R. Case, H.Bruce Bosmann, Angela Glading, Jason E. Fish, Ingrid H. Sarelius, G. Frederick Wooten, Dakota Gustafson, H B Bosmann, Michael D. Wilson and Nadiya Khyzha and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and PLoS ONE.

In The Last Decade

Peter V. DiStefano

17 papers receiving 618 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Peter V. DiStefano United States 12 277 190 166 101 60 17 640
Keiichi Kadoyama Japan 13 272 1.0× 121 0.6× 58 0.3× 49 0.5× 44 0.7× 36 583
Ramanjit Gill Switzerland 12 413 1.5× 201 1.1× 72 0.4× 95 0.9× 61 1.0× 15 779
Hans-Dieter Mennel Germany 15 268 1.0× 209 1.1× 125 0.8× 33 0.3× 29 0.5× 26 711
Antonina J. Kruppa United Kingdom 10 482 1.7× 128 0.7× 89 0.5× 36 0.4× 36 0.6× 12 836
Kazutoshi Nakano Japan 9 495 1.8× 125 0.7× 216 1.3× 132 1.3× 17 0.3× 21 766
Astrid Scheschonka Germany 16 606 2.2× 255 1.3× 121 0.7× 27 0.3× 127 2.1× 33 971
Weizhong Xiao China 15 225 0.8× 74 0.4× 53 0.3× 121 1.2× 14 0.2× 31 537
Saadet Mercimek‐Mahmutoglu Canada 22 720 2.6× 183 1.0× 192 1.2× 31 0.3× 36 0.6× 51 1.7k

Countries citing papers authored by Peter V. DiStefano

Since Specialization
Citations

This map shows the geographic impact of Peter V. DiStefano's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Peter V. DiStefano with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Peter V. DiStefano more than expected).

Fields of papers citing papers by Peter V. DiStefano

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Peter V. DiStefano. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Peter V. DiStefano. The network helps show where Peter V. DiStefano may publish in the future.

Co-authorship network of co-authors of Peter V. DiStefano

This figure shows the co-authorship network connecting the top 25 collaborators of Peter V. DiStefano. A scholar is included among the top collaborators of Peter V. DiStefano based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Peter V. DiStefano. Peter V. DiStefano is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Gustafson, Dakota, Peter V. DiStefano, Ruilin Wu, et al.. (2024). Circulating small extracellular vesicles mediate vascular hyperpermeability in diabetes. Diabetologia. 67(6). 1138–1154. 6 indexed citations
2.
Boudreau, Émilie, Alexander M. Herman, Manuel Cantu Gutierrez, et al.. (2020). Somatic Gain of KRAS Function in the Endothelium Is Sufficient to Cause Vascular Malformations That Require MEK but Not PI3K Signaling. Circulation Research. 127(6). 727–743. 82 indexed citations
3.
Alibhai, Faisal J., Azadeh Yeganeh, Peter V. DiStefano, et al.. (2020). Cellular senescence contributes to age‐dependent changes in circulating extracellular vesicle cargo and function. Aging Cell. 19(3). e13103–e13103. 103 indexed citations
4.
Khyzha, Nadiya, Peter V. DiStefano, Liangxi Wang, et al.. (2019). Regulation of CCL2 expression in human vascular endothelial cells by a neighboring divergently transcribed long noncoding RNA. Proceedings of the National Academy of Sciences. 116(33). 16410–16419. 69 indexed citations
5.
DiStefano, Peter V. & Angela Glading. (2019). VEGF signalling enhances lesion burden in KRIT1 deficient mice. Journal of Cellular and Molecular Medicine. 24(1). 632–639. 20 indexed citations
6.
Ahmed, Abdalla, Peter V. DiStefano, Lijun Chi, et al.. (2018). The transcriptional regulator CCCTC-binding factor limits oxidative stress in endothelial cells. Journal of Biological Chemistry. 293(22). 8449–8461. 18 indexed citations
7.
Glading, Angela, Peter V. DiStefano, Tiehua Chen, Hua Su, & Kevin J. Whitehead. (2018). VEGF is required for the initiation of Cerebral Cavernous Malformations. The FASEB Journal. 32(S1). 1 indexed citations
8.
Goitre, Luca, Peter V. DiStefano, Andrea Moglia, et al.. (2017). Up-regulation of NADPH oxidase-mediated redox signaling contributes to the loss of barrier function in KRIT1 deficient endothelium. Scientific Reports. 7(1). 8296–8296. 48 indexed citations
9.
DiStefano, Peter V., Alan V. Smrcka, & Angela Glading. (2016). Phospholipase Cε Modulates Rap1 Activity and the Endothelial Barrier. PLoS ONE. 11(9). e0162338–e0162338. 5 indexed citations
10.
DiStefano, Peter V., et al.. (2014). KRIT1 Protein Depletion Modifies Endothelial Cell Behavior via Increased Vascular Endothelial Growth Factor (VEGF) Signaling. Journal of Biological Chemistry. 289(47). 33054–33065. 52 indexed citations
11.
Wooten, G. Frederick, et al.. (1982). Regional cerebral glucose utilization during morphine withdrawal in the rat.. Proceedings of the National Academy of Sciences. 79(10). 3360–3364. 51 indexed citations
12.
DiStefano, Peter V., et al.. (1980). Characterization of the clonidine withdrawal syndrome in the normotensive rat.. Journal of Pharmacology and Experimental Therapeutics. 214(2). 263–268. 22 indexed citations
13.
DiStefano, Peter V., et al.. (1979). Increased specific binding of [H]diazepam in rat brain following chronic diazepam administration. Cell Biology International Reports. 3(2). 163–167. 38 indexed citations
14.
Bosmann, H.Bruce, et al.. (1978). Diazepam receptor: Specific binding of [3H]diazepam and [3H]flunitrazepam to rat brain subfractions. FEBS Letters. 87(2). 199–202. 42 indexed citations
15.
DiStefano, Peter V., et al.. (1977). Cell surface changes caused by growth of B16 melanoma cells in bromodeoxyuridine. FEBS Letters. 78(1). 109–112. 11 indexed citations
16.
DiStefano, Peter V. & H B Bosmann. (1977). Erythrocyte membrane abnormality in muscular dystrophy. Cell Biology International Reports. 1(4). 375–378. 3 indexed citations
17.
Bosmann, H.Bruce, Kenneth R. Case, & Peter V. DiStefano. (1977). Diazepam receptor characterization: Specific binding of a benzodiazepine to macromolecules in various areas of rat brain. FEBS Letters. 82(2). 368–372. 69 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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