Peter C. Hollenhorst

3.2k total citations
36 papers, 2.3k citations indexed

About

Peter C. Hollenhorst is a scholar working on Molecular Biology, Pulmonary and Respiratory Medicine and Cancer Research. According to data from OpenAlex, Peter C. Hollenhorst has authored 36 papers receiving a total of 2.3k indexed citations (citations by other indexed papers that have themselves been cited), including 32 papers in Molecular Biology, 16 papers in Pulmonary and Respiratory Medicine and 5 papers in Cancer Research. Recurrent topics in Peter C. Hollenhorst's work include Prostate Cancer Treatment and Research (13 papers), Ubiquitin and proteasome pathways (11 papers) and Genomics and Chromatin Dynamics (10 papers). Peter C. Hollenhorst is often cited by papers focused on Prostate Cancer Treatment and Research (13 papers), Ubiquitin and proteasome pathways (11 papers) and Genomics and Chromatin Dynamics (10 papers). Peter C. Hollenhorst collaborates with scholars based in United States, Canada and Italy. Peter C. Hollenhorst's co-authors include Barbara J. Graves, Lawrence P. McIntosh, Mary W. Ferris, Chris Hopkins, Joshua P. Plotnik, Justin A. Budka, Catherine A. Fox, W. Evan Johnson, Vivekananda Kedage and Bradley R. Cairns and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and Journal of Biological Chemistry.

In The Last Decade

Peter C. Hollenhorst

35 papers receiving 2.3k citations

Peers

Peter C. Hollenhorst
Kiran Mahajan United States
Qiu Wu China
Karl Dykema United States
Stefanie S. Schalm United States
Andrew D. Sharrocks United Kingdom
Yoon Jong Choi United States
Wenjian Gan United States
Andrew S. Venteicher United States
Kienan I. Savage United Kingdom
Rendong Yang United States
Kiran Mahajan United States
Peter C. Hollenhorst
Citations per year, relative to Peter C. Hollenhorst Peter C. Hollenhorst (= 1×) peers Kiran Mahajan

Countries citing papers authored by Peter C. Hollenhorst

Since Specialization
Citations

This map shows the geographic impact of Peter C. Hollenhorst's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Peter C. Hollenhorst with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Peter C. Hollenhorst more than expected).

Fields of papers citing papers by Peter C. Hollenhorst

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Peter C. Hollenhorst. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Peter C. Hollenhorst. The network helps show where Peter C. Hollenhorst may publish in the future.

Co-authorship network of co-authors of Peter C. Hollenhorst

This figure shows the co-authorship network connecting the top 25 collaborators of Peter C. Hollenhorst. A scholar is included among the top collaborators of Peter C. Hollenhorst based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Peter C. Hollenhorst. Peter C. Hollenhorst is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Hollenhorst, Peter C., et al.. (2025). PRC2/FOXO1-Mediated Repression Determines Interchangeability of ETS Oncogenes in Prostate Cancer and Ewing Sarcoma. Molecular Cancer Research. 24(1). 48–59.
2.
Damayanti, Nur P., et al.. (2024). TFE3-Splicing Factor Fusions Represent Functional Drivers and Druggable Targets in Translocation Renal Cell Carcinoma. Cancer Research. 84(8). 1286–1302. 5 indexed citations
3.
Hollenhorst, Peter C., et al.. (2023). Activation of AKT induces EZH2-mediated β-catenin trimethylation in colorectal cancer. iScience. 26(9). 107630–107630. 18 indexed citations
4.
Hollenhorst, Peter C., et al.. (2021). Androgen signaling connects short isoform production to breakpoint formation at Ewing sarcoma breakpoint region 1. NAR Cancer. 3(3). zcab033–zcab033. 7 indexed citations
5.
Jerde, Travis J., et al.. (2021). Ras/ERK and PI3K/AKT signaling differentially regulate oncogenic ERG mediated transcription in prostate cells. PLoS Genetics. 17(7). e1009708–e1009708. 8 indexed citations
6.
Hollenhorst, Peter C., et al.. (2019). Oncogenic ETS Factors in Prostate Cancer. Advances in experimental medicine and biology. 1210. 409–436. 40 indexed citations
7.
Damayanti, Nur P., Justin A. Budka, Heba Khella, et al.. (2018). Therapeutic Targeting of TFE3/IRS-1/PI3K/mTOR Axis in Translocation Renal Cell Carcinoma. Clinical Cancer Research. 24(23). 5977–5989. 61 indexed citations
8.
Clark, Kathleen A., et al.. (2018). Electrostatic repulsion causes anticooperative DNA binding between tumor suppressor ETS transcription factors and JUN–FOS at composite DNA sites. Journal of Biological Chemistry. 293(48). 18624–18635. 10 indexed citations
9.
Plotnik, Joshua P. & Peter C. Hollenhorst. (2017). Interaction with ZMYND11 mediates opposing roles of Ras-responsive transcription factors ETS1 and ETS2. PMC. 5 indexed citations
10.
Kedage, Vivekananda, et al.. (2017). Phosphorylation of the oncogenic transcription factor ERG in prostate cells dissociates polycomb repressive complex 2, allowing target gene activation. Journal of Biological Chemistry. 292(42). 17225–17235. 16 indexed citations
11.
Tomar, Sunil, Joshua P. Plotnik, James Haley, et al.. (2017). ETS1 induction by the microenvironment promotes ovarian cancer metastasis through focal adhesion kinase. Cancer Letters. 414. 190–204. 42 indexed citations
12.
Kedage, Vivekananda, et al.. (2016). An Interaction with Ewing's Sarcoma Breakpoint Protein EWS Defines a Specific Oncogenic Mechanism of ETS Factors Rearranged in Prostate Cancer. PMC. 1 indexed citations
13.
Plotnik, Joshua P. & Peter C. Hollenhorst. (2016). Genome-Wide Analysis of RAS/ERK Signaling Targets. Methods in molecular biology. 1487. 277–288. 1 indexed citations
14.
Kedage, Vivekananda, et al.. (2015). Comparison of MAPK specificity across the ETS transcription factor family identifies a high-affinity ERK interaction required for ERG function in prostate cells. Cell Communication and Signaling. 13(1). 12–12. 60 indexed citations
15.
Plotnik, Joshua P., Justin A. Budka, Mary W. Ferris, & Peter C. Hollenhorst. (2014). ETS1 is a genome-wide effector of RAS/ERK signaling in epithelial cells. Nucleic Acids Research. 42(19). 11928–11940. 100 indexed citations
16.
Hollenhorst, Peter C.. (2012). RAS/ERK pathway transcriptional regulation through ETS/AP-1 binding sites. Small GTPases. 3(3). 154–158. 32 indexed citations
17.
Sankar, Savita, Peter C. Hollenhorst, Michelle Kinsey, et al.. (2008). Microsatellites as EWS/FLI response elements in Ewing's sarcoma. Proceedings of the National Academy of Sciences. 105(29). 10149–10154. 195 indexed citations
18.
Hollenhorst, Peter C., et al.. (2007). Genome-wide analyses reveal properties of redundant and specific promoter occupancy within the ETS gene family. Genes & Development. 21(15). 1882–1894. 231 indexed citations
19.
Hollenhorst, Peter C.. (2004). Expression profiles frame the promoter specificity dilemma of the ETS family of transcription factors. Nucleic Acids Research. 32(18). 5693–5702. 176 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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