Stephen L. Lessnick

8.2k total citations · 2 hit papers
56 papers, 5.1k citations indexed

About

Stephen L. Lessnick is a scholar working on Pulmonary and Respiratory Medicine, Molecular Biology and Pathology and Forensic Medicine. According to data from OpenAlex, Stephen L. Lessnick has authored 56 papers receiving a total of 5.1k indexed citations (citations by other indexed papers that have themselves been cited), including 45 papers in Pulmonary and Respiratory Medicine, 31 papers in Molecular Biology and 23 papers in Pathology and Forensic Medicine. Recurrent topics in Stephen L. Lessnick's work include Sarcoma Diagnosis and Treatment (45 papers), Lymphoma Diagnosis and Treatment (20 papers) and RNA Research and Splicing (11 papers). Stephen L. Lessnick is often cited by papers focused on Sarcoma Diagnosis and Treatment (45 papers), Lymphoma Diagnosis and Treatment (20 papers) and RNA Research and Splicing (11 papers). Stephen L. Lessnick collaborates with scholars based in United States, Canada and Belgium. Stephen L. Lessnick's co-authors include Richard A. Smith, Savita Sankar, Christopher T. Denny, Timothy J. Triche, Poul H. Sorensen, Dolores López‐Terrada, Xian Fang Liu, John M. Peltier, Sudhir Sahasrabudhe and Moritz von Rechenberg and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Nature Genetics.

In The Last Decade

Stephen L. Lessnick

56 papers receiving 5.1k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

RAS–RAF–MEK-dependent oxidative cell death involving voltage-dependent anion channels

2007 1.3k citations Moritz von Rechenberg, Elma Zaganjor et al. Nature profile →
A second Ewing's sarcoma translocation, t(21;22), fuses the EWS gene to another ETS–family transcription factor, ERG

1994 575 citations Poul H. Sorensen, Stephen L. Lessnick et al. Nature Genetics profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Stephen L. Lessnick United States	32	3.3k	2.9k	1.3k	1.0k	716	56	5.1k
Colin S. Cooper United Kingdom	31	3.0k 0.9×	3.9k 1.4×	1.6k 1.2×	1.6k 1.6×	544 0.8×	67	6.7k
Gregory M. Coté United States	35	1.4k 0.4×	1.3k 0.4×	485 0.4×	1.3k 1.2×	411 0.6×	151	3.4k
Helmut E. Gabbert Germany	44	1.7k 0.5×	2.8k 1.0×	954 0.7×	2.2k 2.2×	761 1.1×	126	6.2k
Roland Penzel Germany	40	1.5k 0.5×	1.7k 0.6×	1.3k 1.0×	1.8k 1.7×	771 1.1×	134	4.4k
Baocun Sun China	39	783 0.2×	2.8k 1.0×	1.5k 1.2×	1.7k 1.7×	379 0.5×	145	4.6k
Zhong Jiang United States	42	1.8k 0.6×	2.2k 0.8×	1.2k 0.9×	794 0.8×	379 0.5×	129	4.6k
Keila E. Torres United States	32	1.7k 0.5×	971 0.3×	297 0.2×	1.3k 1.3×	385 0.5×	94	3.3k
David H. Ilson United States	48	4.9k 1.5×	1.3k 0.5×	530 0.4×	3.5k 3.5×	495 0.7×	276	7.9k
Mario Sarbia Germany	40	1.9k 0.6×	2.0k 0.7×	719 0.5×	1.9k 1.9×	487 0.7×	151	6.1k
Davide Melisi Italy	42	1.3k 0.4×	2.4k 0.8×	1.4k 1.1×	3.5k 3.4×	477 0.7×	164	5.9k

Countries citing papers authored by Stephen L. Lessnick

Since Specialization

Citations

This map shows the geographic impact of Stephen L. Lessnick's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Stephen L. Lessnick with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Stephen L. Lessnick more than expected).

Fields of papers citing papers by Stephen L. Lessnick

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Stephen L. Lessnick. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Stephen L. Lessnick. The network helps show where Stephen L. Lessnick may publish in the future.

Co-authorship network of co-authors of Stephen L. Lessnick

This figure shows the co-authorship network connecting the top 25 collaborators of Stephen L. Lessnick. A scholar is included among the top collaborators of Stephen L. Lessnick based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Stephen L. Lessnick. Stephen L. Lessnick is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Vo, Kieuhoa T., C. Lorrie Epling, Elizabeth Sinclair, et al.. (2016). Impact of Two Measures of Micrometastatic Disease on Clinical Outcomes in Patients with Newly Diagnosed Ewing Sarcoma: A Report from the Children's Oncology Group. Clinical Cancer Research. 22(14). 3643–3650. 17 indexed citations

Lerman, Daniel M., Michael J. Monument, Dali Huang, et al.. (2014). Tumoral TP53 and/or CDKN2A alterations are not reliable prognostic biomarkers in patients with localized Ewing sarcoma: A report from the Children's Oncology Group. Pediatric Blood & Cancer. 62(5). 759–765. 36 indexed citations

Wiles, Elizabeth T., et al.. (2013). BCL11B Is Up-Regulated by EWS/FLI and Contributes to the Transformed Phenotype in Ewing Sarcoma. PLoS ONE. 8(3). e59369–e59369. 35 indexed citations

Evans, Christopher H., Fangjun Liu, Ryan M. Porter, et al.. (2012). EWS-FLI-1-Targeted Cytotoxic T-cell Killing of Multiple Tumor Types Belonging to the Ewing Sarcoma Family of Tumors. Clinical Cancer Research. 18(19). 5341–5351. 39 indexed citations

Calvert, George T., R. Lor Randall, Kevin B. Jones, et al.. (2012). At-Risk Populations for Osteosarcoma: The Syndromes and Beyond. Sarcoma. 2012. 1–9. 59 indexed citations

Sankar, Savita, Russell Bell, Bret J. Stephens, et al.. (2012). Mechanism and relevance of EWS/FLI-mediated transcriptional repression in Ewing sarcoma. Oncogene. 32(42). 5089–5100. 118 indexed citations

Görlick, Richard, Katherine A. Janeway, Stephen L. Lessnick, R. Lor Randall, & Neyssa Marina. (2012). Children's Oncology Group's 2013 blueprint for research: Bone tumors. Pediatric Blood & Cancer. 60(6). 1009–1015. 168 indexed citations

Beck, Robert, Michael J. Monument, W. Scott Watkins, et al.. (2012). EWS/FLI-responsive GGAA microsatellites exhibit polymorphic differences between European and African populations. Cancer Genetics. 205(6). 304–312. 24 indexed citations

Chaturvedi, Aashi, Laura Hoffman, Alana L. Welm, Stephen L. Lessnick, & Mary C. Beckerle. (2012). The EWS/FLI Oncogene Drives Changes in Cellular Morphology, Adhesion, and Migration in Ewing Sarcoma. Genes & Cancer. 3(2). 102–116. 71 indexed citations

10.

Sankar, Savita & Stephen L. Lessnick. (2011). Promiscuous partnerships in Ewing's sarcoma. Cancer Genetics. 204(7). 351–365. 163 indexed citations

11.

Ji, Lingyun, Hiroyuki Shimada, Michele R. Wing, et al.. (2010). Current Treatment Protocols Have Eliminated the Prognostic Advantage of Type 1 Fusions in Ewing Sarcoma: A Report From the Children's Oncology Group. Journal of Clinical Oncology. 28(12). 1989–1994. 95 indexed citations

12.

Kinsey, Michelle, Richard A. Smith, Anita K. Iyer, Edward R.B. McCabe, & Stephen L. Lessnick. (2009). EWS/FLI and Its Downstream Target NR0B1 Interact Directly to Modulate Transcription and Oncogenesis in Ewing's Sarcoma. Cancer Research. 69(23). 9047–9055. 74 indexed citations

13.

Sankar, Savita, Peter C. Hollenhorst, Michelle Kinsey, et al.. (2008). Microsatellites as EWS/FLI response elements in Ewing's sarcoma. Proceedings of the National Academy of Sciences. 105(29). 10149–10154. 195 indexed citations

14.

Owen, Leah A., et al.. (2008). EWS/FLI Mediates Transcriptional Repression via NKX2.2 during Oncogenic Transformation in Ewing's Sarcoma. PLoS ONE. 3(4). e1965–e1965. 116 indexed citations

15.

Zhou, Zhichao, et al.. (2007). EWS/FLI1 Regulates Tumor Angiogenesis in Ewing's Sarcoma via Suppression of Thrombospondins. Cancer Research. 67(14). 6675–6684. 32 indexed citations

16.

Rechenberg, Moritz von, Elma Zaganjor, Andras J. Bauer, et al.. (2007). RAS–RAF–MEK-dependent oxidative cell death involving voltage-dependent anion channels. Nature. 447(7146). 865–869. 1301 indexed citations breakdown →

17.

Kinsey, Michelle, Richard A. Smith, & Stephen L. Lessnick. (2006). NR0B1 Is Required for the Oncogenic Phenotype Mediated by EWS/FLI in Ewing's Sarcoma. Molecular Cancer Research. 4(11). 851–859. 160 indexed citations

18.

Smith, Richard A., Leah A. Owen, Jenny Wong, et al.. (2006). Expression profiling of EWS/FLI identifies NKX2.2 as a critical target gene in Ewing's sarcoma. Cancer Cell. 9(5). 405–416. 271 indexed citations

19.

Davis, Ian J., Jessica Kim, Fatih Ozsolak, et al.. (2006). Oncogenic MITF dysregulation in clear cell sarcoma: Defining the MiT family of human cancers. Cancer Cell. 9(6). 473–484. 152 indexed citations

20.

Sorensen, Poul H., Stephen L. Lessnick, Dolores López‐Terrada, et al.. (1994). A second Ewing's sarcoma translocation, t(21;22), fuses the EWS gene to another ETS–family transcription factor, ERG. Nature Genetics. 6(2). 146–151. 575 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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