Peizhong Mao

4.8k total citations · 1 hit paper
35 papers, 4.0k citations indexed

About

Peizhong Mao is a scholar working on Molecular Biology, Physiology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Peizhong Mao has authored 35 papers receiving a total of 4.0k indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Molecular Biology, 20 papers in Physiology and 10 papers in Cellular and Molecular Neuroscience. Recurrent topics in Peizhong Mao's work include Mitochondrial Function and Pathology (12 papers), Alzheimer's disease research and treatments (12 papers) and Adipose Tissue and Metabolism (5 papers). Peizhong Mao is often cited by papers focused on Mitochondrial Function and Pathology (12 papers), Alzheimer's disease research and treatments (12 papers) and Adipose Tissue and Metabolism (5 papers). Peizhong Mao collaborates with scholars based in United States, China and Japan. Peizhong Mao's co-authors include P. Hemachandra Reddy, Maria Mańczak, Ulziibat Shirendeb, Marcus J. Calkins, Arubala P. Reddy, T. P. Reddy, Byung Park, Anda Cornea, Michael P. Murphy and Hazel H. Szeto and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Neuroscience and SHILAP Revista de lepidopterología.

In The Last Decade

Peizhong Mao

35 papers receiving 3.9k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Impaired mitochondrial biogenesis, defective axonal transport of mitochondria, abnormal mitochondrial dynamics and synaptic degeneration in a mouse model of Alzheimer's disease

2011 531 citations Marcus J. Calkins, Maria Mańczak et al. Human Molecular Genetics profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Peizhong Mao United States	24	2.4k	1.9k	1.0k	409	396	35	4.0k
Rodrigo A. Quintanilla Chile	36	2.4k 1.0×	1.8k 1.0×	1.0k 1.0×	484 1.2×	376 0.9×	80	4.2k
Joanna B. Strosznajder Poland	36	2.4k 1.0×	1.9k 1.0×	1.0k 1.0×	566 1.4×	494 1.2×	165	4.8k
Sandra Moreno Italy	35	1.9k 0.8×	1.0k 0.5×	845 0.8×	362 0.9×	398 1.0×	80	3.6k
Heng Du United States	31	2.7k 1.1×	2.3k 1.2×	756 0.7×	591 1.4×	222 0.6×	73	4.4k
Takashi Morihara Japan	28	2.0k 0.8×	2.1k 1.1×	835 0.8×	500 1.2×	322 0.8×	57	4.9k
Marc Gleichmann United States	27	2.0k 0.8×	1.2k 0.7×	909 0.9×	308 0.8×	341 0.9×	40	3.9k
Wan Sung Choi South Korea	37	2.2k 0.9×	1.0k 0.6×	886 0.9×	405 1.0×	166 0.4×	163	4.9k
Shaharyar M. Khan United States	19	1.7k 0.7×	1.5k 0.8×	394 0.4×	391 1.0×	238 0.6×	29	2.8k
Gyeong Jae Cho South Korea	36	2.0k 0.8×	1.0k 0.5×	603 0.6×	402 1.0×	148 0.4×	122	4.3k
Latha Devi United States	19	1.3k 0.5×	1.6k 0.9×	900 0.9×	427 1.0×	735 1.9×	19	3.0k

Countries citing papers authored by Peizhong Mao

Since Specialization

Citations

This map shows the geographic impact of Peizhong Mao's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Peizhong Mao with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Peizhong Mao more than expected).

Fields of papers citing papers by Peizhong Mao

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Peizhong Mao. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Peizhong Mao. The network helps show where Peizhong Mao may publish in the future.

Co-authorship network of co-authors of Peizhong Mao

This figure shows the co-authorship network connecting the top 25 collaborators of Peizhong Mao. A scholar is included among the top collaborators of Peizhong Mao based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Peizhong Mao. Peizhong Mao is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Mao, Peizhong, et al.. (2018). The catalytic, stem, and transmembrane portions of matriptase-2 are required for suppressing the expression of the iron-regulatory hormone hepcidin. Journal of Biological Chemistry. 294(6). 2060–2073. 9 indexed citations

Rønnekleiv, Oline K., Fang Yuan, Chunguang Zhang, et al.. (2014). Research Resource: Gene Profiling of G Protein–Coupled Receptors in the Arcuate Nucleus of the Female. Molecular Endocrinology. 28(8). 1362–1380. 20 indexed citations

Mao, Peizhong, Maria Mańczak, Ulziibat Shirendeb, & P. Hemachandra Reddy. (2013). MitoQ, a mitochondria-targeted antioxidant, delays disease progression and alleviates pathogenesis in an experimental autoimmune encephalomyelitis mouse model of multiple sclerosis. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1832(12). 2322–2331. 99 indexed citations

Mao, Peizhong. (2012). Recent progress and concerns in dementia: Distinguishing Alzheimer's disease and dementia with Lewy bodies via biochemical markers in the cerebrospinal fluid. Advances in Biological Chemistry. 2(2). 176–190. 8 indexed citations

Mao, Peizhong, Charles K. Meshul, Philippe Thuillier, Natalie R.S. Goldberg, & P. Hemachandra Reddy. (2012). CART Peptide Is a Potential Endogenous Antioxidant and Preferentially Localized in Mitochondria. PLoS ONE. 7(1). e29343–e29343. 46 indexed citations

Mao, Peizhong & P. Hemachandra Reddy. (2011). Aging and amyloid beta-induced oxidative DNA damage and mitochondrial dysfunction in Alzheimer's disease: Implications for early intervention and therapeutics. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1812(11). 1359–1370. 252 indexed citations

Shirendeb, Ulziibat, Arubala P. Reddy, Maria Mańczak, et al.. (2011). Abnormal mitochondrial dynamics, mitochondrial loss and mutant huntingtin oligomers in Huntington's disease: implications for selective neuronal damage. Human Molecular Genetics. 20(7). 1438–1455. 311 indexed citations

Mao, Peizhong, et al.. (2011). A transgenic mouse model for Alzheimer's disease has impaired synaptic gain but normal synaptic dynamics. Neuroscience Letters. 500(3). 212–215. 5 indexed citations

Mao, Peizhong, Patience Gallagher, Maria Mańczak, et al.. (2011). Mitochondrial DNA deletions and differential mitochondrial DNA content in Rhesus monkeys: Implications for aging. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1822(2). 111–119. 20 indexed citations

10.

Calkins, Marcus J., Maria Mańczak, Peizhong Mao, Ulziibat Shirendeb, & P. Hemachandra Reddy. (2011). Impaired mitochondrial biogenesis, defective axonal transport of mitochondria, abnormal mitochondrial dynamics and synaptic degeneration in a mouse model of Alzheimer's disease. Human Molecular Genetics. 20(23). 4515–4529. 531 indexed citations breakdown →

11.

Shirendeb, Ulziibat, Marcus J. Calkins, Maria Mańczak, et al.. (2011). Mutant huntingtin's interaction with mitochondrial protein Drp1 impairs mitochondrial biogenesis and causes defective axonal transport and synaptic degeneration in Huntington's disease. Human Molecular Genetics. 21(2). 406–420. 306 indexed citations

12.

Reddy, P. Hemachandra, Raghav Tripathi, T. P. Reddy, et al.. (2011). Abnormal mitochondrial dynamics and synaptic degeneration as early events in Alzheimer's disease: Implications to mitochondria-targeted antioxidant therapeutics. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1822(5). 639–649. 313 indexed citations

13.

Reddy, P. Hemachandra, T. P. Reddy, Maria Mańczak, et al.. (2010). Dynamin-related protein 1 and mitochondrial fragmentation in neurodegenerative diseases. Brain Research Reviews. 67(1-2). 103–118. 305 indexed citations

14.

Reddy, P. Hemachandra, Maria Mańczak, Peizhong Mao, et al.. (2010). Amyloid-β and Mitochondria in Aging and Alzheimer's Disease: Implications for Synaptic Damage and Cognitive Decline. Journal of Alzheimer s Disease. 20(s2). S499–S512. 209 indexed citations

15.

Mao, Peizhong & P. Hemachandra Reddy. (2009). Is multiple sclerosis a mitochondrial disease?. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1802(1). 66–79. 200 indexed citations

16.

Mańczak, Maria, Peizhong Mao, Kazuhiro Nakamura, et al.. (2009). Neutralization of granulocyte macrophage colony-stimulating factor decreases amyloid beta 1-42 and suppresses microglial activity in a transgenic mouse model of Alzheimer's disease. Human Molecular Genetics. 18(20). 3876–3893. 46 indexed citations

17.

Reddy, P. Hemachandra, Peizhong Mao, & Maria Mańczak. (2009). Mitochondrial structural and functional dynamics in Huntington's disease. Brain Research Reviews. 61(1). 33–48. 131 indexed citations

18.

Mao, Peizhong, Yuan‐Xiang Tao, Masahiro Fukaya, et al.. (2008). Cloning and characterization of E‐dlg, a novel splice variant of mouse homologue of the Drosophila discs large tumor suppressor binds preferentially to SAP102. IUBMB Life. 60(10). 684–692. 2 indexed citations

19.

Tao, Feng, Yuan‐Xiang Tao, Julio A. González, et al.. (2001). Knockdown of PSD-95/SAP90 delays the development of neuropathic pain in rats. Neuroreport. 12(15). 3251–3255. 63 indexed citations

20.

Fang, Xi, et al.. (1992). Evaluation of biological activity of LHRH antagonists by three methods. Contraception. 46(2). 135–137. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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