Latha Devi

3.6k total citations · 2 hit papers
19 papers, 3.0k citations indexed

About

Latha Devi is a scholar working on Physiology, Pharmacology and Molecular Biology. According to data from OpenAlex, Latha Devi has authored 19 papers receiving a total of 3.0k indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Physiology, 9 papers in Pharmacology and 6 papers in Molecular Biology. Recurrent topics in Latha Devi's work include Alzheimer's disease research and treatments (16 papers), Cholinesterase and Neurodegenerative Diseases (9 papers) and Mitochondrial Function and Pathology (4 papers). Latha Devi is often cited by papers focused on Alzheimer's disease research and treatments (16 papers), Cholinesterase and Neurodegenerative Diseases (9 papers) and Mitochondrial Function and Pathology (4 papers). Latha Devi collaborates with scholars based in United States and India. Latha Devi's co-authors include Masuo Ohno, Hindupur K. Anandatheerthavarada, Narayan G. Avadhani, Badanavalu M. Prabhu, Vijayendran Raghavendran, Domenico F. Galati, Melissa J. Alldred, Stephen D. Ginsberg, Ryoichi Kimura and T.R. Raju and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Neuroscience and PLoS ONE.

In The Last Decade

Latha Devi

19 papers receiving 2.9k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Mitochondrial Import and Accumulation of α-Synuclein Impair Complex I in Human Dopaminergic Neuronal Cultures and Parkinson Disease Brain

2008 863 citations Latha Devi, Vijayendran Raghavendran et al. Journal of Biological Chemistry profile →
Accumulation of Amyloid Precursor Protein in the Mitochondrial Import Channels of Human Alzheimer’s Disease Brain Is Associated with Mitochondrial Dysfunction

2006 664 citations Latha Devi, Badanavalu M. Prabhu et al. Journal of Neuroscience profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Latha Devi United States	19	1.6k	1.3k	900	735	427	19	3.0k
Esther Dalfó Spain	30	1.3k 0.8×	1.2k 0.9×	828 0.9×	1.3k 1.7×	206 0.5×	42	3.1k
Peizhong Mao United States	24	1.9k 1.1×	2.4k 1.8×	1.0k 1.1×	396 0.5×	409 1.0×	35	4.0k
John J. LaFrancois United States	24	2.6k 1.6×	1.4k 1.1×	1.4k 1.5×	453 0.6×	523 1.2×	36	4.5k
Susan Leight United States	19	2.2k 1.4×	1.2k 0.9×	953 1.1×	1.5k 2.1×	378 0.9×	21	3.9k
Magali Dumont United States	25	1.2k 0.7×	1.2k 0.9×	629 0.7×	353 0.5×	344 0.8×	38	2.5k
Valentina Echeverrı́a United States	36	859 0.5×	1.3k 1.0×	705 0.8×	349 0.5×	531 1.2×	87	3.2k
Giuseppina Amadoro Italy	30	1.4k 0.8×	1.1k 0.8×	822 0.9×	234 0.3×	423 1.0×	65	2.5k
Cora O’Neill Ireland	28	1.5k 0.9×	1.6k 1.2×	958 1.1×	267 0.4×	470 1.1×	65	3.2k
Manuela Polydoro United States	23	1.9k 1.2×	926 0.7×	960 1.1×	514 0.7×	381 0.9×	28	3.2k
Hanna Ksiȩżak-Reding United States	35	2.4k 1.5×	1.7k 1.3×	886 1.0×	856 1.2×	357 0.8×	64	3.8k

Countries citing papers authored by Latha Devi

Since Specialization

Citations

This map shows the geographic impact of Latha Devi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Latha Devi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Latha Devi more than expected).

Fields of papers citing papers by Latha Devi

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Latha Devi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Latha Devi. The network helps show where Latha Devi may publish in the future.

Co-authorship network of co-authors of Latha Devi

This figure shows the co-authorship network connecting the top 25 collaborators of Latha Devi. A scholar is included among the top collaborators of Latha Devi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Latha Devi. Latha Devi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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19 of 19 papers shown

Devi, Latha & Masuo Ohno. (2016). Cognitive benefits of memantine in Alzheimer's 5XFAD model mice decline during advanced disease stages. Pharmacology Biochemistry and Behavior. 144. 60–66. 27 indexed citations

Devi, Latha & Masuo Ohno. (2015). A combination Alzheimer’s therapy targeting BACE1 and neprilysin in 5XFAD transgenic mice. Molecular Brain. 8(1). 19–19. 29 indexed citations

Devi, Latha, Jordan Tang, & Masuo Ohno. (2015). Beneficial Effects of the β-Secretase Inhibitor GRL-8234 in 5XFAD Alzheimer’s Transgenic Mice Lessen During Disease Progression. Current Alzheimer Research. 12(1). 13–21. 28 indexed citations

Devi, Latha & Masuo Ohno. (2014). PERK mediates eIF2α phosphorylation responsible for BACE1 elevation, CREB dysfunction and neurodegeneration in a mouse model of Alzheimer's disease. Neurobiology of Aging. 35(10). 2272–2281. 138 indexed citations

Devi, Latha & Masuo Ohno. (2013). Deletion of the eIF2α Kinase GCN2 Fails to Rescue the Memory Decline Associated with Alzheimer’s Disease. PLoS ONE. 8(10). e77335–e77335. 55 indexed citations

Devi, Latha & Masuo Ohno. (2013). Effects of levetiracetam, an antiepileptic drug, on memory impairments associated with aging and Alzheimer’s disease in mice. Neurobiology of Learning and Memory. 102. 7–11. 42 indexed citations

Devi, Latha, Melissa J. Alldred, Stephen D. Ginsberg, & Masuo Ohno. (2012). Mechanisms Underlying Insulin Deficiency-Induced Acceleration of β-Amyloidosis in a Mouse Model of Alzheimer's Disease. PLoS ONE. 7(3). e32792–e32792. 134 indexed citations

Devi, Latha & Masuo Ohno. (2011). 7,8-Dihydroxyflavone, a Small-Molecule TrkB Agonist, Reverses Memory Deficits and BACE1 Elevation in a Mouse Model of Alzheimer's Disease. Neuropsychopharmacology. 37(2). 434–444. 231 indexed citations

Devi, Latha & Masuo Ohno. (2011). Mitochondrial dysfunction and accumulation of the β-secretase-cleaved C-terminal fragment of APP in Alzheimer's disease transgenic mice. Neurobiology of Disease. 45(1). 417–424. 108 indexed citations

10.

Kimura, Ryoichi, Latha Devi, & Masuo Ohno. (2010). Partial reduction of BACE1 improves synaptic plasticity, recent and remote memories in Alzheimer’s disease transgenic mice. Journal of Neurochemistry. 113(1). 248–261. 113 indexed citations

11.

Devi, Latha & Masuo Ohno. (2010). Phospho-eIF2α Level Is Important for Determining Abilities of BACE1 Reduction to Rescue Cholinergic Neurodegeneration and Memory Defects in 5XFAD Mice. PLoS ONE. 5(9). e12974–e12974. 107 indexed citations

12.

Devi, Latha, Melissa J. Alldred, Stephen D. Ginsberg, & Masuo Ohno. (2010). Sex- and brain region-specific acceleration of β-amyloidogenesis following behavioral stress in a mouse model of Alzheimer's disease. Molecular Brain. 3(1). 34–34. 114 indexed citations

13.

Devi, Latha & Hindupur K. Anandatheerthavarada. (2009). Mitochondrial trafficking of APP and alpha synuclein: Relevance to mitochondrial dysfunction in Alzheimer's and Parkinson's diseases. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1802(1). 11–19. 120 indexed citations

14.

Devi, Latha & Masuo Ohno. (2009). Genetic reductions of β‐site amyloid precursor protein‐cleaving enzyme 1 and amyloid‐β ameliorate impairment of conditioned taste aversion memory in 5XFAD Alzheimer’s disease model mice. European Journal of Neuroscience. 31(1). 110–118. 70 indexed citations

15.

Devi, Latha, Vijayendran Raghavendran, Badanavalu M. Prabhu, Narayan G. Avadhani, & Hindupur K. Anandatheerthavarada. (2008). Mitochondrial Import and Accumulation of α-Synuclein Impair Complex I in Human Dopaminergic Neuronal Cultures and Parkinson Disease Brain. Journal of Biological Chemistry. 283(14). 9089–9100. 863 indexed citations breakdown →

16.

Anandatheerthavarada, Hindupur K. & Latha Devi. (2007). Mitochondrial Translocation of Amyloid Precursor Protein and its Cleaved Products: Relevance to Mitochondrial Dysfunction in Alzheimer's Disease. Reviews in the Neurosciences. 18(5). 343–54. 22 indexed citations

17.

Anandatheerthavarada, Hindupur K. & Latha Devi. (2007). Amyloid Precursor Protein and Mitochondrial Dysfunction in Alzheimer's Disease. The Neuroscientist. 13(6). 626–638. 78 indexed citations

18.

Devi, Latha, Badanavalu M. Prabhu, Domenico F. Galati, Narayan G. Avadhani, & Hindupur K. Anandatheerthavarada. (2006). Accumulation of Amyloid Precursor Protein in the Mitochondrial Import Channels of Human Alzheimer’s Disease Brain Is Associated with Mitochondrial Dysfunction. Journal of Neuroscience. 26(35). 9057–9068. 664 indexed citations breakdown →

19.

Devi, Latha, Latha Diwakar, T.R. Raju, & Bindu M. Kutty. (2003). Selective neurodegeneration of hippocampus and entorhinal cortex correlates with spatial learning impairments in rats with bilateral ibotenate lesions of ventral subiculum. Brain Research. 960(1-2). 9–15. 38 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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