Pedro C. Redondo

3.2k total citations
78 papers, 2.3k citations indexed

About

Pedro C. Redondo is a scholar working on Molecular Biology, Sensory Systems and Cellular and Molecular Neuroscience. According to data from OpenAlex, Pedro C. Redondo has authored 78 papers receiving a total of 2.3k indexed citations (citations by other indexed papers that have themselves been cited), including 46 papers in Molecular Biology, 32 papers in Sensory Systems and 12 papers in Cellular and Molecular Neuroscience. Recurrent topics in Pedro C. Redondo's work include Ion Channels and Receptors (32 papers), Ion channel regulation and function (22 papers) and Receptor Mechanisms and Signaling (9 papers). Pedro C. Redondo is often cited by papers focused on Ion Channels and Receptors (32 papers), Ion channel regulation and function (22 papers) and Receptor Mechanisms and Signaling (9 papers). Pedro C. Redondo collaborates with scholars based in Spain, United Kingdom and United States. Pedro C. Redondo's co-authors include Juan A. Rosado, Ginés M. Salido, José A. Pariente, José J. López, Isaac Jardín, Stewart O. Sage, Alejandro Berna‐Erro, Carlos Cantonero, A. Rodríguez and Javier Espino and has published in prestigious journals such as Journal of Biological Chemistry, Blood and The Journal of Physiology.

In The Last Decade

Pedro C. Redondo

72 papers receiving 2.3k citations

Peers

Pedro C. Redondo
Baskaran Thyagarajan United States
Fanning Zeng United Kingdom
Derek S. Damron United States
Suk Hyo Suh South Korea
Piruthivi Sukumar United Kingdom
Pedro C. Redondo
Citations per year, relative to Pedro C. Redondo Pedro C. Redondo (= 1×) peers José J. López

Countries citing papers authored by Pedro C. Redondo

Since Specialization
Citations

This map shows the geographic impact of Pedro C. Redondo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Pedro C. Redondo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Pedro C. Redondo more than expected).

Fields of papers citing papers by Pedro C. Redondo

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Pedro C. Redondo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Pedro C. Redondo. The network helps show where Pedro C. Redondo may publish in the future.

Co-authorship network of co-authors of Pedro C. Redondo

This figure shows the co-authorship network connecting the top 25 collaborators of Pedro C. Redondo. A scholar is included among the top collaborators of Pedro C. Redondo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Pedro C. Redondo. Pedro C. Redondo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Pérez‐Gómez, Jorge, Antonio Castillo-Paredes, Pedro C. Redondo, et al.. (2025). Effects of Twelve Weeks of Square Stepping Exercises on Physical and Cognitive Function and Plasma Content of SMP30: A Randomised Control Trial. Geriatrics. 10(1). 22–22. 1 indexed citations
2.
Redondo, Pedro C., et al.. (2025). Notch1 regulates Orai1 and Orai3 expression in breast cancer cells. Scientific Reports. 16(1). 3229–3229.
4.
Berna‐Erro, Alejandro, et al.. (2023). SARAF overexpression impairs thrombin‐induced Ca 2+ homeostasis in neonatal platelets. British Journal of Haematology. 204(3). 988–1004.
5.
Redondo, Pedro C., et al.. (2023). Hypothyroidism: A Peculiar Presentation. Cureus. 15(12). e49819–e49819.
6.
Cantonero, Carlos, Pedro J. Camello, Ginés M. Salido, Juan A. Rosado, & Pedro C. Redondo. (2021). TMEM97 facilitates the activation of SOCE by downregulating the association of cholesterol to Orai1 in MDA-MB-231 cells. Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids. 1866(6). 158906–158906. 12 indexed citations
7.
Sánchez-Collado, José, José J. López, Carlos Cantonero, et al.. (2021). Orai2 Modulates Store-Operated Ca2+ Entry and Cell Cycle Progression in Breast Cancer Cells. Cancers. 14(1). 114–114. 20 indexed citations
8.
Cantonero, Carlos, Pedro J. Camello, Carmen Abate, et al.. (2020). NO1, a New Sigma 2 Receptor/TMEM97 Fluorescent Ligand, Downregulates SOCE and Promotes Apoptosis in the Triple Negative Breast Cancer Cell Lines. Cancers. 12(2). 257–257. 32 indexed citations
9.
López, Esther, Alejandro Berna‐Erro, José J. López, et al.. (2015). Role of mTOR1 and mTOR2 complexes in MEG-01 cell physiology. Thrombosis and Haemostasis. 114(11). 969–981. 6 indexed citations
10.
Berna‐Erro, Alejandro, Pedro C. Redondo, Esther López, Letizia Albarrán, & Juan A. Rosado. (2013). Molecular Interplay between Platelets and the Vascular Wall in Thrombosis and Hemostasis. Current Vascular Pharmacology. 11(4). 409–430. 11 indexed citations
11.
Berna‐Erro, Alejandro, Pedro C. Redondo, & Juan A. Rosado. (2012). Store-Operated Ca2+ Entry. Advances in experimental medicine and biology. 740. 349–382. 43 indexed citations
12.
Rosado, Juan A., et al.. (2011). Immunophilin Dysfunction and Neuropathology. Current Medicinal Chemistry. 18(35). 5398–5407. 2 indexed citations
13.
Cubero, Juan José, Esther López, Pedro C. Redondo, et al.. (2011). Immunossupressant and Organ Transplantation: Immunophilins Targeting Agent and Alternative Therapies. Current Medicinal Chemistry. 18(35). 5324–5332. 2 indexed citations
14.
Rodríguez, A., Ignacio Bejarano, Javier Espino, et al.. (2009). Role of Calcium Signals on Hydrogen Peroxide-Induced Apoptosis in Human Myeloid HL-60 Cells. International Journal of Biomedical Science. 5(3). 246–256. 26 indexed citations
15.
Bejarano, Ignacio, Pedro C. Redondo, Javier Espino, et al.. (2009). Melatonin induces mitochondrial‐mediated apoptosis in human myeloid HL‐60 cells. Journal of Pineal Research. 46(4). 392–400. 128 indexed citations
16.
Redondo, Pedro C., Isaac Jardín, José J. López, Ginés M. Salido, & Juan A. Rosado. (2007). Intracellular Ca2+ store depletion induces the formation of macromolecular complexes involving hTRPC1, hTRPC6, the type II IP3 receptor and SERCA3 in human platelets. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1783(6). 1163–1176. 52 indexed citations
17.
Jardín, Isaac, Pedro C. Redondo, Ginés M. Salido, & Juan A. Rosado. (2007). Phosphatidylinositol 4,5-bisphosphate enhances store-operated calcium entry through hTRPC6 channel in human platelets. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1783(1). 84–97. 68 indexed citations
18.
Redondo, Pedro C., Juan A. Rosado, José A. Pariente, & Ginés M. Salido. (2005). Collaborative effect of SERCA and PMCA in cytosolic calcium homeostasis in human platelets. Journal of Physiology and Biochemistry. 61(4). 507–516. 12 indexed citations
19.
Rosado, Juan A., et al.. (2004). Hydrogen Peroxide Generation Induces pp60 Activation in Human Platelets. Journal of Biological Chemistry. 279(3). 1665–1675. 113 indexed citations
20.
Redondo, Pedro C., Nidhal Ben Amor, Ginés M. Salido, et al.. (2004). Ca2+-independent activation of Bruton's tyrosine kinase is required for store-mediated Ca2+ entry in human platelets. Cellular Signalling. 17(8). 1011–1021. 44 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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