Pedro A. San-Segundo

1.9k total citations
39 papers, 1.4k citations indexed

About

Pedro A. San-Segundo is a scholar working on Molecular Biology, Cell Biology and Cancer Research. According to data from OpenAlex, Pedro A. San-Segundo has authored 39 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 39 papers in Molecular Biology, 11 papers in Cell Biology and 6 papers in Cancer Research. Recurrent topics in Pedro A. San-Segundo's work include DNA Repair Mechanisms (28 papers), Fungal and yeast genetics research (15 papers) and Genomics and Chromatin Dynamics (14 papers). Pedro A. San-Segundo is often cited by papers focused on DNA Repair Mechanisms (28 papers), Fungal and yeast genetics research (15 papers) and Genomics and Chromatin Dynamics (14 papers). Pedro A. San-Segundo collaborates with scholars based in Spain, United States and United Kingdom. Pedro A. San-Segundo's co-authors include G. Shirleen Roeder, Francisco Conde, Carlos R. Vázquez de Aldana, Francisco del Rey, Livia Pérez-Hidalgo, Raimundo Freire, Avelino Bueno, Sergio Moreno, Fred van Leeuwen and Alan G. Hinnebusch and has published in prestigious journals such as Cell, Nucleic Acids Research and Nature Communications.

In The Last Decade

Pedro A. San-Segundo

39 papers receiving 1.4k citations

Peers

Pedro A. San-Segundo

GENET

Adam M. Bailis United States

Alberto Sánchez‐Díaz Spain

Prakash Arumugam Singapore

Masayoshi Iizuka Japan

Kanji Furuya Japan

Laurent Maillet France

Junko Kanoh Japan

Alexandre Huber Switzerland

Rogier ten Hoopen United Kingdom

Adam M. Bailis United States

Pedro A. San-Segundo

911 ×0.7

219 ×0.7

172 ×0.8

101 ×0.8

GENET

71 ×0.7

Citations per year, relative to Pedro A. San-Segundo Pedro A. San-Segundo (= 1×) peers Adam M. Bailis

Countries citing papers authored by Pedro A. San-Segundo

Since Specialization

Citations

This map shows the geographic impact of Pedro A. San-Segundo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Pedro A. San-Segundo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Pedro A. San-Segundo more than expected).

Fields of papers citing papers by Pedro A. San-Segundo

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Pedro A. San-Segundo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Pedro A. San-Segundo. The network helps show where Pedro A. San-Segundo may publish in the future.

Co-authorship network of co-authors of Pedro A. San-Segundo

This figure shows the co-authorship network connecting the top 25 collaborators of Pedro A. San-Segundo. A scholar is included among the top collaborators of Pedro A. San-Segundo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Pedro A. San-Segundo. Pedro A. San-Segundo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Arter, Meret, et al.. (2021). The Cdc14 Phosphatase Controls Resolution of Recombination Intermediates and Crossover Formation during Meiosis. International Journal of Molecular Sciences. 22(18). 9811–9811. 10 indexed citations

Santos, Beatriz, et al.. (2021). Pch2 orchestrates the meiotic recombination checkpoint from the cytoplasm. PLoS Genetics. 17(7). e1009560–e1009560. 18 indexed citations

Carballo, Jesús A., et al.. (2021). The N-Terminal Region of the Polo Kinase Cdc5 Is Required for Downregulation of the Meiotic Recombination Checkpoint. Cells. 10(10). 2561–2561. 1 indexed citations

Grishok, Alla, et al.. (2020). DOT-1.1-dependent H3K79 methylation promotes normal meiotic progression and meiotic checkpoint function in C. elegans. PLoS Genetics. 16(10). e1009171–e1009171. 10 indexed citations

García‐Rodríguez, Néstor, et al.. (2020). Non‐recombinogenic roles for Rad52 in translesion synthesis during DNA damage tolerance. EMBO Reports. 22(1). e50410–e50410. 20 indexed citations

Gardner, Jennifer M., Zulin Yu, Jonna Heldrich, et al.. (2020). SWR1-Independent Association of H2A.Z to the LINC Complex Promotes Meiotic Chromosome Motion. Frontiers in Cell and Developmental Biology. 8. 594092–594092. 10 indexed citations

Santos, Beatriz, et al.. (2019). Characterization of Pch2 localization determinants reveals a nucleolar-independent role in the meiotic recombination checkpoint. Chromosoma. 128(3). 297–316. 11 indexed citations

Subramanian, Vijayalakshmi V., Xuan Zhu, Tovah E. Markowitz, et al.. (2019). Persistent DNA-break potential near telomeres increases initiation of meiotic recombination on short chromosomes. Nature Communications. 10(1). 970–970. 35 indexed citations

Morillo‐Huesca, Macarena, et al.. (2018). Functional Impact of the H2A.Z Histone Variant During Meiosis in Saccharomyces cerevisiae. Genetics. 209(4). 997–1015. 11 indexed citations

10.

San-Segundo, Pedro A., et al.. (2016). The Pch2 AAA+ ATPase promotes phosphorylation of the Hop1 meiotic checkpoint adaptor in response to synaptonemal complex defects. Nucleic Acids Research. 44(16). 7722–7741. 29 indexed citations

11.

San-Segundo, Pedro A., et al.. (2016). Impact of histone H4K16 acetylation on the meiotic recombination checkpoint in Saccharomyces cerevisiae. Microbial Cell. 3(12). 606–620. 16 indexed citations

12.

Vlaming, Hanneke, Tibor van Welsem, Erik L. de Graaf, et al.. (2014). Flexibility in crosstalk between H2B ubiquitination and H3 methylation in vivo. EMBO Reports. 15(11). 1220–1221. 4 indexed citations

13.

Kauppi, Liisa, et al.. (2013). Dynamics of DOT1L localization and H3K79 methylation during meiotic prophase I in mouse spermatocytes. Chromosoma. 123(1-2). 147–164. 30 indexed citations

14.

Leeuwen, Fred van, et al.. (2013). Dot1-Dependent Histone H3K79 Methylation Promotes Activation of the Mek1 Meiotic Checkpoint Effector Kinase by Regulating the Hop1 Adaptor. PLoS Genetics. 9(1). e1003262–e1003262. 47 indexed citations

15.

San-Segundo, Pedro A., et al.. (2011). The budding yeast polo-like kinase Cdc5 regulates the Ndt80 branch of the meiotic recombination checkpoint pathway. Molecular Biology of the Cell. 22(18). 3478–3490. 24 indexed citations

16.

Farmer, Sarah, Pedro A. San-Segundo, & Luís Aragón. (2011). The Smc5–Smc6 Complex Is Required to Remove Chromosome Junctions in Meiosis. PLoS ONE. 6(6). e20948–e20948. 24 indexed citations

17.

Pérez-Hidalgo, Livia, Sergio Moreno, & Pedro A. San-Segundo. (2008). The fission yeast meiotic checkpoint kinase Mek1 regulates nuclear localization of Cdc25 by phosphorylation. Cell Cycle. 7(23). 3720–3730. 10 indexed citations

18.

Perera, David, Livia Pérez-Hidalgo, Peter B. Møens, et al.. (2004). TopBP1 and ATR Colocalization at Meiotic Chromosomes: Role of TopBP1/Cut5 in the Meiotic Recombination Checkpoint. Molecular Biology of the Cell. 15(4). 1568–1579. 64 indexed citations

19.

San-Segundo, Pedro A., et al.. (2004). Characterization of a Saccharomyces cerevisiae thermosensitive lytic mutant leads to the identification of a new allele of the NUD1 gene. The International Journal of Biochemistry & Cell Biology. 36(11). 2196–2213. 9 indexed citations

20.

San-Segundo, Pedro A. & G. Shirleen Roeder. (1999). Pch2 Links Chromatin Silencing to Meiotic Checkpoint Control. Cell. 97(3). 313–324. 228 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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