Paul E. Hurtubise
- Immunology top 10%
- Oncology top 10%
- Pathology and Forensic Medicine top 5%
- Genetics top 5%
- Pulmonary and Respiratory Medicine top 10%
- Co-authors
- Albert F. LoBuglioGerald W. KingRobert P. BaughmanArthur L. SagoneCarol H. BoskenRobert S. BarWerner HenleJohn F. Hewetson
- Topics
- Lymphoma Diagnosis and Treatment (12 papers)Chronic Lymphocytic Leukemia Research (9 papers)Immunodeficiency and Autoimmune Disorders (7 papers)
- Cited by
- TransplantationGeneticsImmunology
- Partner nations
- United StatesChina
In The Last Decade
Paul E. Hurtubise
59 papers receiving 1.2k citations
Peers
Comparison fields: 5 of 98
- Immunology 407
- Oncology 335
- Pathology and Forensic Medicine 227
- Genetics 225
- Pulmonary and Respiratory Medicine 216
Countries citing papers authored by Paul E. Hurtubise
This map shows the geographic impact of Paul E. Hurtubise's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Paul E. Hurtubise with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Paul E. Hurtubise more than expected).
Fields of papers citing papers by Paul E. Hurtubise
This network shows the impact of papers produced by Paul E. Hurtubise. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Paul E. Hurtubise. The network helps show where Paul E. Hurtubise may publish in the future.
Co-authorship network of co-authors of Paul E. Hurtubise
This figure shows the co-authorship network connecting the top 25 collaborators of Paul E. Hurtubise. A scholar is included among the top collaborators of Paul E. Hurtubise based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Paul E. Hurtubise. Paul E. Hurtubise is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 25 | |
| 2 | 6 | |
| 3 | 44 | |
| 4 | 15 | |
| 5 | Flow cytometry and clinical diagnosis | 15 |
| 6 | 14 | |
| 7 | 27 | |
| 8 | 1 | |
| 9 | 12 | |
| 10 | 36 | |
| 11 | 4 | |
| 12 | 18 | |
| 13 | 49 | |
| 14 | 1 | |
| 15 | 4 | |
| 16 | 5 | |
| 17 | 33 | |
| 18 | 9 | |
| 19 | 2 | |
| 20 | 14 |
About Paul E. Hurtubise
Paul E. Hurtubise is a scholar working on Transplantation, Genetics and Pathology and Forensic Medicine, having authored 60 papers that have together received 1.4k indexed citations. Recurring topics across this work include Lymphoma Diagnosis and Treatment (12 papers), Chronic Lymphocytic Leukemia Research (9 papers) and Immunodeficiency and Autoimmune Disorders (7 papers). The work is most often cited by research in Transplantation (75 citations), Genetics (225 citations) and Immunology (407 citations). Paul E. Hurtubise has collaborated with scholars based in United States and China. Frequent co-authors include Albert F. LoBuglio, Gerald W. King, Robert P. Baughman, Arthur L. Sagone, Carol H. Bosken, Robert S. Bar, Werner Henle, John F. Hewetson, Karen James and Kathryn P. Clausen. Their work appears in journals such as New England Journal of Medicine, Journal of Clinical Investigation and The Journal of Immunology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.