Arthur L. Sagone
- Immunology top 5%
- Molecular Biology
- Physiology top 10%
- Pulmonary and Respiratory Medicine
- Genetics top 5%
- Co-authors
- Stanley P. BalcerzakAlbert F. LoBuglioEarl N. MetzJohn J. RinehartGerald W. KingPaul E. HurtubiseThomas LawrenceW. Bruce Davis
- Topics
- Neutrophil, Myeloperoxidase and Oxidative Mechanisms (9 papers)Nitric Oxide and Endothelin Effects (7 papers)Erythrocyte Function and Pathophysiology (7 papers)
- Cited by
- ImmunologyGeneticsBiochemistry
- Partner nations
- United States
In The Last Decade
Arthur L. Sagone
51 papers receiving 1.4k citations
Peers
Comparison fields: 5 of 118
- Immunology 522
- Molecular Biology 408
- Physiology 289
- Pulmonary and Respiratory Medicine 172
- Genetics 160
Countries citing papers authored by Arthur L. Sagone
This map shows the geographic impact of Arthur L. Sagone's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Arthur L. Sagone with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Arthur L. Sagone more than expected).
Fields of papers citing papers by Arthur L. Sagone
This network shows the impact of papers produced by Arthur L. Sagone. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Arthur L. Sagone. The network helps show where Arthur L. Sagone may publish in the future.
Co-authorship network of co-authors of Arthur L. Sagone
This figure shows the co-authorship network connecting the top 25 collaborators of Arthur L. Sagone. A scholar is included among the top collaborators of Arthur L. Sagone based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Arthur L. Sagone. Arthur L. Sagone is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 9 | |
| 2 | 20 | |
| 3 | 3 | |
| 4 | 10 | |
| 5 | 6 | |
| 6 | 27 | |
| 7 | 37 | |
| 8 | 33 | |
| 9 | 11 | |
| 10 | 2 | |
| 11 | 4 | |
| 12 | Determination of hydroxyl radical production in aqueous solutions irradiated to clinically significant doses. | 10 |
| 13 | 19 | |
| 14 | 3 | |
| 15 | 28 | |
| 16 | 36 | |
| 17 | 60 | |
| 18 | 71 | |
| 19 | 91 | |
| 20 | 34 |
About Arthur L. Sagone
Arthur L. Sagone is a scholar working on Immunology, Biophysics and Physiology, having authored 51 papers that have together received 1.7k indexed citations. Recurring topics across this work include Neutrophil, Myeloperoxidase and Oxidative Mechanisms (9 papers), Nitric Oxide and Endothelin Effects (7 papers) and Erythrocyte Function and Pathophysiology (7 papers). The work is most often cited by research in Immunology (522 citations), Genetics (160 citations) and Biochemistry (82 citations). Arthur L. Sagone has collaborated with scholars based in United States. Frequent co-authors include Stanley P. Balcerzak, Albert F. LoBuglio, Earl N. Metz, John J. Rinehart, Gerald W. King, Paul E. Hurtubise, Thomas Lawrence, W. Bruce Davis, Eric H. Kraut and Gerardo Burton. Their work appears in journals such as New England Journal of Medicine, Journal of Clinical Investigation and Blood.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.