Pamela B. Moore
- Co-authors
- John DedmanEileen R. MulvihillSpencer EmtageRichard D. PalmiterNaomi Kraus-FriedmannKermit L. CarrawayMohammad Shamsul OlaVadivel Ganapathy
- Topics
- Ion channel regulation and function (7 papers)Signaling Pathways in Disease (4 papers)Protein Structure and Dynamics (3 papers)
- Partner nations
- United StatesFrance
In The Last Decade
Pamela B. Moore
31 papers receiving 843 citations
Peers
Comparison fields: 5 of 98
- Molecular Biology 628
- Genetics 146
- Cellular and Molecular Neuroscience 118
- Cell Biology 114
- Physiology 66
Countries citing papers authored by Pamela B. Moore
This map shows the geographic impact of Pamela B. Moore's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Pamela B. Moore with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Pamela B. Moore more than expected).
Fields of papers citing papers by Pamela B. Moore
This network shows the impact of papers produced by Pamela B. Moore. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Pamela B. Moore. The network helps show where Pamela B. Moore may publish in the future.
Co-authorship network of co-authors of Pamela B. Moore
This figure shows the co-authorship network connecting the top 25 collaborators of Pamela B. Moore. A scholar is included among the top collaborators of Pamela B. Moore based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Pamela B. Moore. Pamela B. Moore is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 46 | |
| 2 | Downregulation of reduced-folate transporter by glucose in cultured RPE cells and in RPE of diabetic mice. | 28 |
| 3 | 76 | |
| 4 | Screening rabbit colonies for antibodies to Pasteurella multocida by an ELISA. | 11 |
| 5 | 2 | |
| 6 | 3 | |
| 7 | 5 | |
| 8 | 8 | |
| 9 | Expression of 67,000 MW calcimedin and its binding protein in resident and thioglycolate-elicited macrophages. | 1 |
| 10 | 11 | |
| 11 | 23 | |
| 12 | 42 | |
| 13 | 22 | |
| 14 | 139 | |
| 15 | 3 | |
| 16 | 9 | |
| 17 | 48 | |
| 18 | 110 | |
| 19 | 184 | |
| 20 | 23 |
About Pamela B. Moore
Pamela B. Moore is a scholar working on Cell Biology, Physiology and Molecular Biology, having authored 31 papers that have together received 921 indexed citations. Recurring topics across this work include Ion channel regulation and function (7 papers), Signaling Pathways in Disease (4 papers) and Protein Structure and Dynamics (3 papers). The work is most often cited by research in Molecular Biology (628 citations), Physiology (37 citations) and Cell Biology (114 citations). Pamela B. Moore has collaborated with scholars based in United States and France. Frequent co-authors include John Dedman, Eileen R. Mulvihill, Spencer Emtage, Richard D. Palmiter, Naomi Kraus-Friedmann, Kermit L. Carraway, Mohammad Shamsul Ola, Vadivel Ganapathy, Sylvia B. Smith and Charlotte L. Ownby. Their work appears in journals such as Cell, Journal of Biological Chemistry and Analytical Biochemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.