Osamu Hatase
- Molecular Biology top 5%
- Cellular and Molecular Neuroscience top 5%
- Cell Biology top 5%
- Oncology top 10%
- Developmental Neuroscience top 5%
- Co-authors
- Hideki MatsuiMasaaki TokudaToshifumi ItanoKazuhito TomizawaJerry H. WangSeisuke HattoriTakaya GotohMichiyuki Matsuda
- Topics
- Neuroscience and Neuropharmacology Research (12 papers)Signaling Pathways in Disease (9 papers)Mitochondrial Function and Pathology (7 papers)
- Partner nations
- JapanUnited StatesCanada
In The Last Decade
Osamu Hatase
64 papers receiving 1.9k citations
Peers
Comparison fields: 5 of 95
- Molecular Biology 1.3k
- Cellular and Molecular Neuroscience 492
- Cell Biology 396
- Oncology 354
- Developmental Neuroscience 170
Countries citing papers authored by Osamu Hatase
This map shows the geographic impact of Osamu Hatase's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Osamu Hatase with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Osamu Hatase more than expected).
Fields of papers citing papers by Osamu Hatase
This network shows the impact of papers produced by Osamu Hatase. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Osamu Hatase. The network helps show where Osamu Hatase may publish in the future.
Co-authorship network of co-authors of Osamu Hatase
This figure shows the co-authorship network connecting the top 25 collaborators of Osamu Hatase. A scholar is included among the top collaborators of Osamu Hatase based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Osamu Hatase. Osamu Hatase is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 4 | |
| 2 | 16 | |
| 3 | 54 | |
| 4 | 24 | |
| 5 | 33 | |
| 6 | 110 | |
| 7 | 15 | |
| 8 | 21 | |
| 9 | 15 | |
| 10 | 20 | |
| 11 | 310 | |
| 12 | 40 | |
| 13 | 289 | |
| 14 | 48 | |
| 15 | 8 | |
| 16 | 4 | |
| 17 | 2 | |
| 18 | 10 | |
| 19 | 5 | |
| 20 | Mitochondrial sulfhydryl groups. A possible endogenous probe of conformational changes in the mitochondrial membrane. | 3 |
About Osamu Hatase
Osamu Hatase is a scholar working on Developmental Neuroscience, Neurology and Cellular and Molecular Neuroscience, having authored 64 papers that have together received 1.9k indexed citations. Recurring topics across this work include Neuroscience and Neuropharmacology Research (12 papers), Signaling Pathways in Disease (9 papers) and Mitochondrial Function and Pathology (7 papers). The work is most often cited by research in Developmental Neuroscience (170 citations), Cellular and Molecular Neuroscience (492 citations) and Cell Biology (396 citations). Osamu Hatase has collaborated with scholars based in Japan, United States and Canada. Frequent co-authors include Hideki Matsui, Masaaki Tokuda, Toshifumi Itano, Kazuhito Tomizawa, Jerry H. Wang, Seisuke Hattori, Takaya Gotoh, Michiyuki Matsuda, Shun Nakamura and Damu Tang. Their work appears in journals such as Journal of Biological Chemistry, Molecular and Cellular Biology and Hepatology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.