Oréda Boussadia

2.0k total citations · 1 hit paper
10 papers, 1.7k citations indexed

About

Oréda Boussadia is a scholar working on Molecular Biology, Cell Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Oréda Boussadia has authored 10 papers receiving a total of 1.7k indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 2 papers in Cell Biology and 1 paper in Cellular and Molecular Neuroscience. Recurrent topics in Oréda Boussadia's work include Wnt/β-catenin signaling in development and cancer (4 papers), RNA Research and Splicing (4 papers) and Cancer-related gene regulation (3 papers). Oréda Boussadia is often cited by papers focused on Wnt/β-catenin signaling in development and cancer (4 papers), RNA Research and Splicing (4 papers) and Cancer-related gene regulation (3 papers). Oréda Boussadia collaborates with scholars based in France, Germany and Italy. Oréda Boussadia's co-authors include Rolf Kemler, Stefanie Kutsch, Robert H. Moore, Andrew P. McMahon, Lukas Sommer, Mākoto Ishibashi, Véronique Brault, David H. Rowitch, Véronique Delmas and Andreas Hierholzer and has published in prestigious journals such as The Journal of Cell Biology, Development and Journal of Virology.

In The Last Decade

Oréda Boussadia

10 papers receiving 1.7k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Inactivation of the β-catenin gene by Wnt1-Cre-mediated deletion results in dramatic brain malformation and failure of craniofacial development

2001 943 citations Véronique Brault, Robert H. Moore et al. Development profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Oréda Boussadia France	10	1.4k	255	217	186	174	10	1.7k
Stefanie Kutsch Germany	9	1.3k 0.9×	318 1.2×	160 0.7×	134 0.7×	184 1.1×	9	1.8k
Jeffrey A. Spencer United States	14	1.1k 0.8×	305 1.2×	213 1.0×	127 0.7×	84 0.5×	15	1.6k
Clemencia Colmenares United States	20	1.6k 1.2×	520 2.0×	145 0.7×	126 0.7×	266 1.5×	32	2.1k
Uichi Koshimizu Japan	22	900 0.7×	258 1.0×	229 1.1×	147 0.8×	83 0.5×	28	1.4k
Rieko Ajima Japan	17	1.2k 0.8×	291 1.1×	221 1.0×	76 0.4×	196 1.1×	29	1.6k
Koichi Miyake Japan	26	1.2k 0.9×	379 1.5×	93 0.4×	294 1.6×	149 0.9×	73	2.1k
Jeong Kyo Yoon United States	28	1.9k 1.4×	489 1.9×	232 1.1×	167 0.9×	289 1.7×	43	2.3k
Carlos Torroja Spain	18	1.4k 1.0×	248 1.0×	208 1.0×	93 0.5×	104 0.6×	33	1.7k
Keijo Luukko Norway	26	1.4k 1.0×	376 1.5×	260 1.2×	559 3.0×	170 1.0×	52	2.2k
David J. Pennisi Australia	19	1.1k 0.8×	296 1.2×	158 0.7×	69 0.4×	140 0.8×	35	1.6k

Countries citing papers authored by Oréda Boussadia

Since Specialization

Citations

This map shows the geographic impact of Oréda Boussadia's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Oréda Boussadia with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Oréda Boussadia more than expected).

Fields of papers citing papers by Oréda Boussadia

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Oréda Boussadia. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Oréda Boussadia. The network helps show where Oréda Boussadia may publish in the future.

Co-authorship network of co-authors of Oréda Boussadia

This figure shows the co-authorship network connecting the top 25 collaborators of Oréda Boussadia. A scholar is included among the top collaborators of Oréda Boussadia based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Oréda Boussadia. Oréda Boussadia is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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10 of 10 papers shown

Saltel, Frédéric, Alban Giese, Lamia Azzi‐Martin, et al.. (2017). Unr defines a novel class of nucleoplasmic reticulum involved in mRNA translation. Journal of Cell Science. 130(10). 1796–1808. 17 indexed citations

Calı̀, Gaetano, Mariastella Zannini, Patrizia Rubini, et al.. (2007). Conditional Inactivation of the E-Cadherin Gene in Thyroid Follicular Cells Affects Gland Development but Does Not Impair Junction Formation. Endocrinology. 148(6). 2737–2746. 39 indexed citations

Cattelino, Anna, Stefan Liebner, R Gallini, et al.. (2003). The conditional inactivation of the β-catenin gene in endothelial cells causes a defective vascular pattern and increased vascular fragility. The Journal of Cell Biology. 162(6). 1111–1122. 263 indexed citations

Boussadia, Oréda, Michael Niepmann, Laurent Créancier, et al.. (2003). Unr Is Required In Vivo for Efficient Initiation of Translation from the Internal Ribosome Entry Sites of both Rhinovirus and Poliovirus. Journal of Virology. 77(6). 3353–3359. 97 indexed citations

Boussadia, Oréda, Stefanie Kutsch, Andreas Hierholzer, Véronique Delmas, & Rolf Kemler. (2002). E-cadherin is a survival factor for the lactating mouse mammary gland. Mechanisms of Development. 115(1-2). 53–62. 216 indexed citations

Boussadia, Oréda, et al.. (2002). E-cadherin Is Required for the Correct Formation of Autotypic Adherens Junctions of the Outer Mesaxon but Not for the Integrity of Myelinated Fibers of Peripheral Nerves. Molecular and Cellular Neuroscience. 21(2). 341–351. 34 indexed citations

Brault, Véronique, Robert H. Moore, Stefanie Kutsch, et al.. (2001). Inactivation of the β-catenin gene by Wnt1-Cre-mediated deletion results in dramatic brain malformation and failure of craniofacial development. Development. 128(8). 1253–1264. 943 indexed citations breakdown →

Boussadia, Oréda, Franck Amiot, Sylvaine Cases, et al.. (1997). Transcription of unr (pstream of ‐as) down‐modulates N‐ras expression in vivo. FEBS Letters. 420(1). 20–24. 45 indexed citations

Amiot, Franck, Oréda Boussadia, Sylvaine Cases, et al.. (1997). Mice heterozygous for a deletion of the tumor necrosis factor‐α and lymphotoxin‐α genes: biological importance of a nonlinear response of tumor necrosis factor‐α to gene dosage. European Journal of Immunology. 27(4). 1035–1042. 39 indexed citations

10.

Boussadia, Oréda, Hélène Jacquemin‐Sablon, & François Dautry. (1993). Exon skipping in the expression of the gene immediately upstream of N-ras (unr/NRU). Biochimica et Biophysica Acta (BBA) - Gene Structure and Expression. 1172(1-2). 64–72. 19 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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