Nicholas H. Heintz

7.7k citations
96 papers · 6.2k indexed · 1 hit paper · h-index 40

Nicholas H. Heintz

96 papers receiving 6.0k citations

Hit Papers

Redox-based regulation of signal transduction: Principles...6262008202620142020200400600

Peers

Nicholas H. Heintz
Comparison fields: 5 of 134
  • Molecular Biology 4.1k
  • Biochemistry 431
  • Cancer Research 665
  • Oncology 840
  • Aging 48
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Alvin M. Malkinson United States
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Citations per field
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Citations per year

Countries citing papers authored by Nicholas H. Heintz

Since Specialization
Citations

This map shows the geographic impact of Nicholas H. Heintz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nicholas H. Heintz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nicholas H. Heintz more than expected).

Fields of papers citing papers by Nicholas H. Heintz

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nicholas H. Heintz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nicholas H. Heintz. The network helps show where Nicholas H. Heintz may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Nicholas H. Heintz, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Nicholas H. Heintz Line = papers co-authored together Nicholas H. Heintz links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 2016175
2 201526
3 201421
4 201437
5 201436
6 201326
7 201166
8 201045
9 200961
10 20043
11 200161
12 199925
13
E2F-4・DP-1 DNA結合複合体の蓄積はG 1 よりS期への遷移中にdhfr発現の誘導と相関する | 文献情報 | J-GLOBAL 科学技術総合リンクセンター
19971
14 199731
15 19961
16 199480
17 199231
18 199048
19 198812
20 19883

About Nicholas H. Heintz

Nicholas H. Heintz is a scholar working on Biochemistry, Cancer Research, Molecular Biology, Pulmonary and Respiratory Medicine and Genetics, having authored 96 papers that have together received 6.2k indexed citations. Recurring topics across this work include DNA Repair Mechanisms (21 papers), Occupational and environmental lung diseases (15 papers), Redox biology and oxidative stress (14 papers), Carcinogens and Genotoxicity Assessment (12 papers), Cancer-related Molecular Pathways (9 papers), Genomics and Chromatin Dynamics (9 papers), Sulfur Compounds in Biology (9 papers) and CRISPR and Genetic Engineering (8 papers). The work is most often cited by research in Molecular Biology (4.1k citations), Biochemistry (431 citations), Cancer Research (665 citations), Oncology (840 citations) and Aging (48 citations). Nicholas H. Heintz has collaborated with scholars based in United States, Japan and South Korea. Frequent co-authors include Brooke T. Mossman, William C. Burhans, Joyce L. Hamlin, Yvonne Janssen‐Heininger, Yvonne M. W. Janssen, Mark S. Caddle, Albert van der Vliet, Jeffrey Milbrandt, Sharon Illenye and Brian Cunniff. Their work appears in journals such as Molecular and Cellular Biology, Journal of Biological Chemistry, Proceedings of the National Academy of Sciences, American Journal of Respiratory Cell and Molecular Biology and Free Radical Biology and Medicine.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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