Milena Hristova

3.7k total citations · 1 hit paper
40 papers, 3.1k citations indexed

About

Milena Hristova is a scholar working on Physiology, Immunology and Molecular Biology. According to data from OpenAlex, Milena Hristova has authored 40 papers receiving a total of 3.1k indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Physiology, 22 papers in Immunology and 21 papers in Molecular Biology. Recurrent topics in Milena Hristova's work include Neutrophil, Myeloperoxidase and Oxidative Mechanisms (15 papers), Nitric Oxide and Endothelin Effects (13 papers) and Redox biology and oxidative stress (10 papers). Milena Hristova is often cited by papers focused on Neutrophil, Myeloperoxidase and Oxidative Mechanisms (15 papers), Nitric Oxide and Endothelin Effects (13 papers) and Redox biology and oxidative stress (10 papers). Milena Hristova collaborates with scholars based in United States, Netherlands and Hungary. Milena Hristova's co-authors include Albert van der Vliet, Jason P. Eiserich, Carroll E. Cross, Bruce Α. Freeman, Barry Halliwell, A. Daniel Jones, Yvonne Janssen‐Heininger, Aida Habibovic, Umadevi V. Wesley and Peter F. Bove and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Milena Hristova

39 papers receiving 3.0k citations

Hit Papers

Formation of nitric oxide-derived inflammatory oxidants b... 1998 2026 2007 2016 1998 400 800 1.2k
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Formation of nitric oxide-derived inflammatory oxidants by myeloperoxidase in neutrophils
    1998 1.3k citations Jason P. Eiserich, Milena Hristova et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Milena Hristova United States 23 1.2k 1.2k 981 423 412 40 3.1k
Roy J. Soberman United States 38 1.3k 1.0× 1.1k 1.0× 1.0k 1.0× 444 1.0× 609 1.5× 77 3.6k
Balu K. Chacko United States 36 2.0k 1.6× 870 0.7× 549 0.6× 309 0.7× 332 0.8× 66 4.0k
Leonor Thomson Uruguay 28 1.3k 1.0× 996 0.8× 746 0.8× 195 0.5× 296 0.7× 42 3.4k
Hans‐Erik Claesson Sweden 34 1.2k 1.0× 1.0k 0.9× 1.0k 1.0× 328 0.8× 527 1.3× 109 3.6k
Simona Zarini United States 30 970 0.8× 656 0.6× 423 0.4× 221 0.5× 416 1.0× 58 2.6k
Rafal R. Nazarewicz United States 21 1.3k 1.1× 763 0.6× 587 0.6× 189 0.4× 244 0.6× 32 3.0k
Being‐Sun Wung Taiwan 31 1.5k 1.2× 627 0.5× 510 0.5× 188 0.4× 218 0.5× 43 3.0k
John F. Parkinson United States 30 809 0.7× 792 0.7× 752 0.8× 221 0.5× 401 1.0× 55 2.9k
Carol R. Gardner United States 30 840 0.7× 522 0.4× 624 0.6× 261 0.6× 279 0.7× 62 3.5k
Hazel Lum United States 30 1.4k 1.2× 690 0.6× 442 0.5× 293 0.7× 155 0.4× 50 3.1k

Countries citing papers authored by Milena Hristova

Since Specialization
Citations

This map shows the geographic impact of Milena Hristova's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Milena Hristova with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Milena Hristova more than expected).

Fields of papers citing papers by Milena Hristova

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Milena Hristova. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Milena Hristova. The network helps show where Milena Hristova may publish in the future.

Co-authorship network of co-authors of Milena Hristova

This figure shows the co-authorship network connecting the top 25 collaborators of Milena Hristova. A scholar is included among the top collaborators of Milena Hristova based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Milena Hristova. Milena Hristova is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Habibovic, Aida, Milena Hristova, Miao-chong J. Lin, et al.. (2023). Diet-induced obesity worsens allergen-induced type 2/type 17 inflammation in airways by enhancing DUOX1 activation. American Journal of Physiology-Lung Cellular and Molecular Physiology. 324(2). L228–L242. 4 indexed citations
2.
Dustin, Christopher M., Aida Habibovic, Milena Hristova, et al.. (2021). Oxidation–Dependent Activation of Src Kinase Mediates Epithelial IL-33 Production and Signaling during Acute Airway Allergen Challenge. The Journal of Immunology. 206(12). 2989–2999. 9 indexed citations
3.
Hristova, Milena, Aida Habibovic, Christopher M. Dustin, et al.. (2020). The Transient Receptor Potential Channel Vanilloid 1 is Critical in Innate Airway Epithelial Responses to Protease Allergens. American Journal of Respiratory Cell and Molecular Biology. 63(2). 198–208. 34 indexed citations
4.
Heppner, David E., Christopher M. Dustin, Chenyi Liao, et al.. (2018). Direct cysteine sulfenylation drives activation of the Src kinase. Nature Communications. 9(1). 4522–4522. 97 indexed citations
5.
Little, Andrew C., Milena Hristova, Karamatullah Danyal, et al.. (2016). DUOX1 silencing in lung cancer promotes EMT, cancer stem cell characteristics and invasive properties. Oncogenesis. 5(10). e261–e261. 52 indexed citations
6.
Heppner, David E., Milena Hristova, Christopher M. Dustin, et al.. (2016). The NADPH Oxidases DUOX1 and NOX2 Play Distinct Roles in Redox Regulation of Epidermal Growth Factor Receptor Signaling. Journal of Biological Chemistry. 291(44). 23282–23293. 51 indexed citations
7.
Habibovic, Aida, Milena Hristova, David E. Heppner, et al.. (2016). DUOX1 mediates persistent epithelial EGFR activation, mucous cell metaplasia, and airway remodeling during allergic asthma. JCI Insight. 1(18). e88811–e88811. 56 indexed citations
8.
Danyal, Karamatullah, David E. Heppner, Andrew C. Little, et al.. (2016). Acrolein and thiol-reactive electrophiles suppress allergen-induced innate airway epithelial responses by inhibition of DUOX1 and EGFR. American Journal of Physiology-Lung Cellular and Molecular Physiology. 311(5). L913–L923. 15 indexed citations
9.
Hristova, Milena, Aida Habibovic, Carmen Veith, et al.. (2015). Airway epithelial dual oxidase 1 mediates allergen-induced IL-33 secretion and activation of type 2 immune responses. Journal of Allergy and Clinical Immunology. 137(5). 1545–1556.e11. 115 indexed citations
10.
11.
Randall, Matthew J., Milena Hristova, & Albert van der Vliet. (2013). Protein alkylation by the α,β‐unsaturated aldehyde acrolein. A reversible mechanism of electrophile signaling?. FEBS Letters. 587(23). 3808–3814. 27 indexed citations
12.
Gorissen, Stefan H. M., et al.. (2012). Dual Oxidase–1 Is Required for Airway Epithelial Cell Migration and Bronchiolar Reepithelialization after Injury. American Journal of Respiratory Cell and Molecular Biology. 48(3). 337–345. 44 indexed citations
13.
Hristova, Milena, et al.. (2011). The Tobacco Smoke Component, Acrolein, Suppresses Innate Macrophage Responses by Direct Alkylation of c-Jun N-Terminal Kinase. American Journal of Respiratory Cell and Molecular Biology. 46(1). 23–33. 34 indexed citations
14.
Olson, Nels C., Milena Hristova, Nicholas H. Heintz, Karen M. Lounsbury, & Albert van der Vliet. (2011). Activation of hypoxia-inducible factor-1 protects airway epithelium against oxidant-induced barrier dysfunction. American Journal of Physiology-Lung Cellular and Molecular Physiology. 301(6). L993–L1002. 66 indexed citations
15.
Olson, Nels C., et al.. (2010). Modulation of NF-κB and Hypoxia-Inducible Factor−1 by S -Nitrosoglutathione Does Not Alter Allergic Airway Inflammation in Mice. American Journal of Respiratory Cell and Molecular Biology. 44(6). 813–823. 13 indexed citations
16.
Olson, Nels C., et al.. (2008). Nitric oxide and airway epithelial barrier function: Regulation of tight junction proteins and epithelial permeability. Archives of Biochemistry and Biophysics. 484(2). 205–213. 22 indexed citations
17.
Hristova, Milena, et al.. (2007). GSH‐dependent regulation of Fas‐mediated caspase‐8 activation by acrolein. FEBS Letters. 581(3). 361–367. 17 indexed citations
18.
Bove, Peter F., et al.. (2006). Nitric Oxide Promotes Airway Epithelial Wound Repair through Enhanced Activation of MMP-9. American Journal of Respiratory Cell and Molecular Biology. 36(2). 138–146. 88 indexed citations
19.
Finkelstein, Erik I., et al.. (2005). Regulation of constitutive neutrophil apoptosis by the α,β-unsaturated aldehydes acrolein and 4-hydroxynonenal. American Journal of Physiology-Lung Cellular and Molecular Physiology. 289(6). L1019–L1028. 58 indexed citations
20.
Vliet, Albert van der, Milena Hristova, Carroll E. Cross, Jason P. Eiserich, & Tzipora Goldkorn. (1998). Peroxynitrite Induces Covalent Dimerization of Epidermal Growth Factor Receptors in A431 Epidermoid Carcinoma Cells. Journal of Biological Chemistry. 273(48). 31860–31866. 89 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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