Neil P. Rodrigues
- Hematology top 0.5%
- Acute Myeloid Leukemia Research 15
- Hematopoietic Stem Cell Transplantation 10
- Aging top 5%
- Genetics top 2%
- Immunology top 5%
- Immune Cell Function and Interaction 5
- Immune cells in cancer 4
- Molecular Biology top 5%
- Epigenetics and DNA Methylation 11
- CRISPR and Genetic Engineering 3
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- Zebrafish Biomedical Research Applications 3
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- Cancer-related Molecular Pathways 3
- Co-authors
- David T. ScaddenTao ChengDavid DombkowskiHongmei ShenYong‐Guang YangMegan SykesAshleigh S. BoydSebastian Stier
- Cited by
- HematologyAgingGenetics
- Partner nations
- United KingdomUnited StatesSaudi Arabia
In The Last Decade
Neil P. Rodrigues
31 papers receiving 3.0k citations
Hit Papers
Peers
Comparison fields: 5 of 102
- Hematology 1.1k
- Aging 70
- Genetics 374
- Immunology 637
- Molecular Biology 1.8k
Countries citing papers authored by Neil P. Rodrigues
This map shows the geographic impact of Neil P. Rodrigues's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Neil P. Rodrigues with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Neil P. Rodrigues more than expected).
Fields of papers citing papers by Neil P. Rodrigues
This network shows the impact of papers produced by Neil P. Rodrigues. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Neil P. Rodrigues. The network helps show where Neil P. Rodrigues may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Neil P. Rodrigues, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 0 | |
| 2 | 2025 | 1 | |
| 3 | 2024 | 0 | |
| 4 | 2023 | 2 | |
| 5 | 2023 | 7 | |
| 6 | 2022 | 2 | |
| 7 | 2022 | 3 | |
| 8 | 2021 | 10 | |
| 9 | 2019 | 1 | |
| 10 | 2019 | 8 | |
| 11 | 2017 | 11 | |
| 12 | 2013 | 288 | |
| 13 | 2011 | 51 | |
| 14 | 2009 | 81 | |
| 15 | 2007 | 404 | |
| 16 | 2005 | 212 | |
| 17 | Haploinsufficiency of GAtA-2 effects adult stem cell homeostasis. | 2003 | 1 |
| 18 | 2000 | 274 | |
| 19 | Hematopoietic Stem Cell Quiescence Maintained by p21 cip1/waf1breakdown → | 2000 | 1083 |
| 20 | 1998 | 92 |
About Neil P. Rodrigues
Neil P. Rodrigues is a scholar working on Hematology, Immunology and Molecular Biology, having authored 34 papers that have together received 3.0k indexed citations. Recurring topics across this work include Acute Myeloid Leukemia Research (15 papers), Epigenetics and DNA Methylation (11 papers), Hematopoietic Stem Cell Transplantation (10 papers), Immune Cell Function and Interaction (5 papers), Immune cells in cancer (4 papers), CRISPR and Genetic Engineering (3 papers), Zebrafish Biomedical Research Applications (3 papers) and Cancer-related Molecular Pathways (3 papers). The work is most often cited by research in Hematology (1.1k citations), Aging (70 citations) and Genetics (374 citations). Neil P. Rodrigues has collaborated with scholars based in United Kingdom, United States and Saudi Arabia. Frequent co-authors include David T. Scadden, Tao Cheng, David Dombkowski, Hongmei Shen, Yong‐Guang Yang, Megan Sykes, Ashleigh S. Boyd, Sebastian Stier, Tariq Enver and Gustavo Mostoslavsky. Their work appears in journals such as Nature, Science and Nature Medicine.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.