This map shows the geographic impact of K E Davies's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by K E Davies with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites K E Davies more than expected).
This network shows the impact of papers produced by K E Davies. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by K E Davies. The network helps show where K E Davies may publish in the future.
Co-authorship network of co-authors of K E Davies
This figure shows the co-authorship network connecting the top 25 collaborators of K E Davies.
A scholar is included among the top collaborators of K E Davies based on the total number of
citations received by their joint publications. Widths of edges
represent the number of papers authors have co-authored together.
Node borders
signify the number of papers an author published with K E Davies. K E Davies is excluded from
the visualization to improve readability, since they are connected to all nodes in the network.
Ilkovski, Biljana, Kristen L. Nowak, Ana Domazetovska, et al.. (2004). Mutant actins that cause congenital myopathy affect sarcomeric protein expression in patient muscle and produce intranuclear and cytoplasmic aggregates in cultured myoblasts. Neuromuscular Disorders. 14. 562–562.3 indexed citations
6.
Davies, K E, et al.. (2003). Functional studies of Af4 in the robotic mouse.. The American Journal of Human Genetics. 73. 179–179.3 indexed citations
Owen, Nicholas, et al.. (2001). Analysis of mutations in Survival of Motor Neuron (SMN1) gene through model systems.. The American Journal of Human Genetics. 69. 368–368.1 indexed citations
9.
Rybakova, Inna N., et al.. (2001). Dystrophin and utrophin are functionally homologous actin binding proteins but act through distinct modes of filament association. Molecular Biology of the Cell. 12.1 indexed citations
10.
Davies, K E, et al.. (2001). Increased muscle lim protein expression associated with mild muscular dystrophy and with exercise. Circulation. 104. 165–165.6 indexed citations
11.
Cullen, Michael J., Jason Walsh, Jonathon M. Tinsley, Rosie Fisher, & K E Davies. (2001). Confirmation by immunogold labeling that utrophin is localised to the normal position of dystrophin in dystrophin-negative transgenic mouse muscle. Neuromuscular Disorders. 11. 666–666.2 indexed citations
12.
Fisher, Rosie, et al.. (2000). Non-toxic ubiquitous overexpression of utrophin in the mdx mouse.. The American Journal of Human Genetics. 67. 428–428.2 indexed citations
13.
Perkins, Kelly J., Edward A. Burton, Allyson C. Potter, & K E Davies. (2000). Analysis of the transcriptional control of utrophin promoters as a therapeutic strategy for Duchenne muscular dystrophy (DMD).. The American Journal of Human Genetics. 67. 192–192.1 indexed citations
Warren, Stephen T., K E Davies, & S M Tilghman. (1995). Genome rearrangement and stability. The American Journal of Human Genetics. 56(5).6 indexed citations
16.
Davies, K E & Shirley M. Tilghman. (1993). Genome maps and neurological disorders.5 indexed citations
17.
Murray, Joseph A., et al.. (1991). GENETIC AND PHYSICAL MAPPING AROUND THE RETINITIS-PIGMENTOSA (RP2) LOCUS IN XP11.23. UCL Discovery (University College London).1 indexed citations
Davies, K E. (1986). Human genetic diseases : a practical approach.83 indexed citations
20.
Thakker, Rajesh V., Melanie J. Davies, K E Davies, et al.. (1986). LOCALIZATION OF THE GENE CAUSING X-LINKED HYPOPHOSPHATEMIC RICKETS. QJM. 61. 1071–1072.2 indexed citations
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive
bibliographic database. While OpenAlex provides broad and valuable coverage of the global
research landscape, it—like all bibliographic datasets—has inherent limitations. These include
incomplete records, variations in author disambiguation, differences in journal indexing, and
delays in data updates. As a result, some metrics and network relationships displayed in
Rankless may not fully capture the entirety of a scholar's output or impact.