Natasha C. Forrest
- Oncology top 2%
- Molecular Biology top 5%
- Cancer Research top 2%
- Genetics top 5%
- Pulmonary and Respiratory Medicine top 10%
- Co-authors
- Geoffrey J. LindemanJane E. VisvaderMarie-Liesse Asselin-LabatMark ShackletonFrançois VaillantRichard M. ThomasJacqueline van der WeesLynne Hartley
- Topics
- Cancer Cells and Metastasis (4 papers)Wnt/β-catenin signaling in development and cancer (1 paper)Cell death mechanisms and regulation (1 paper)
- Partner nations
- AustraliaUnited StatesNetherlands
In The Last Decade
Natasha C. Forrest
5 papers receiving 2.2k citations
Hit Papers
Peers
Comparison fields: 5 of 75
- Oncology 1.6k
- Molecular Biology 1.3k
- Cancer Research 659
- Genetics 406
- Pulmonary and Respiratory Medicine 375
Countries citing papers authored by Natasha C. Forrest
This map shows the geographic impact of Natasha C. Forrest's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Natasha C. Forrest with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Natasha C. Forrest more than expected).
Fields of papers citing papers by Natasha C. Forrest
This network shows the impact of papers produced by Natasha C. Forrest. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Natasha C. Forrest. The network helps show where Natasha C. Forrest may publish in the future.
Co-authorship network of co-authors of Natasha C. Forrest
This figure shows the co-authorship network connecting the top 25 collaborators of Natasha C. Forrest. A scholar is included among the top collaborators of Natasha C. Forrest based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Natasha C. Forrest. Natasha C. Forrest is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | Aberrant luminal progenitors as the candidate target population for basal tumor development in BRCA1 mutation carriersbreakdown → | 1048 |
| 2 | 255 | |
| 3 | 42 | |
| 4 | Gata-3 is an essential regulator of mammary-gland morphogenesis and luminal-cell differentiationbreakdown → | 659 |
| 5 | 220 |
About Natasha C. Forrest
Natasha C. Forrest is a scholar working on Oncology, Cancer Research and Genetics, having authored 5 papers that have together received 2.2k indexed citations. Recurring topics across this work include Cancer Cells and Metastasis (4 papers), Wnt/β-catenin signaling in development and cancer (1 paper) and Cell death mechanisms and regulation (1 paper). The work is most often cited by research in Oncology (1.6k citations), Cancer Research (659 citations) and Molecular Biology (1.3k citations). Natasha C. Forrest has collaborated with scholars based in Australia, United States and Netherlands. Frequent co-authors include Geoffrey J. Lindeman, Jane E. Visvader, Marie-Liesse Asselin-Labat, Mark Shackleton, François Vaillant, Richard M. Thomas, Jacqueline van der Wees, Lynne Hartley, Holly E. Barker and Lorraine Robb. Their work appears in journals such as Nature Medicine, The EMBO Journal and Nature Cell Biology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.