Nada Pejnović

3.2k total citations
74 papers, 2.5k citations indexed

About

Nada Pejnović is a scholar working on Immunology, Surgery and Rheumatology. According to data from OpenAlex, Nada Pejnović has authored 74 papers receiving a total of 2.5k indexed citations (citations by other indexed papers that have themselves been cited), including 47 papers in Immunology, 18 papers in Surgery and 12 papers in Rheumatology. Recurrent topics in Nada Pejnović's work include Immune Cell Function and Interaction (18 papers), IL-33, ST2, and ILC Pathways (16 papers) and Galectins and Cancer Biology (13 papers). Nada Pejnović is often cited by papers focused on Immune Cell Function and Interaction (18 papers), IL-33, ST2, and ILC Pathways (16 papers) and Galectins and Cancer Biology (13 papers). Nada Pejnović collaborates with scholars based in Serbia, United Kingdom and United States. Nada Pejnović's co-authors include Nebojša Arsenijević, Miodrag L. Lukić, Ivan Jovanović, Gordana Radosavljević, Marija Milovanović, Vladislav Volarević, Jelena Pantić, Woon Ling Chan, Nemanja Jovičić and Ilija Jeftić and has published in prestigious journals such as SHILAP Revista de lepidopterología, The Journal of Immunology and PLoS ONE.

In The Last Decade

Nada Pejnović

74 papers receiving 2.5k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Nada Pejnović Serbia 25 1.5k 731 468 343 277 74 2.5k
Daniela Čiháková United States 31 1.4k 0.9× 548 0.7× 766 1.6× 363 1.1× 206 0.7× 82 3.7k
Céline Lamacchia Switzerland 18 1.5k 1.0× 481 0.7× 742 1.6× 220 0.6× 214 0.8× 40 2.6k
Karel Otero United States 22 2.4k 1.6× 593 0.8× 584 1.2× 174 0.5× 283 1.0× 27 3.3k
Haruo Hanawa Japan 32 958 0.6× 391 0.5× 773 1.7× 253 0.7× 204 0.7× 124 2.9k
Cheryl Sweeney Ireland 18 1.9k 1.3× 437 0.6× 605 1.3× 398 1.2× 180 0.6× 26 3.1k
R. Clive Landis United Kingdom 30 693 0.5× 756 1.0× 749 1.6× 205 0.6× 182 0.7× 69 2.9k
Janette Furuzawa‐Carballeda Mexico 26 653 0.4× 510 0.7× 387 0.8× 275 0.8× 197 0.7× 91 2.0k
Nitza Lahat Israel 34 902 0.6× 492 0.7× 674 1.4× 540 1.6× 198 0.7× 126 3.3k
Atsuhiro Ogaẃa Japan 19 1.2k 0.8× 363 0.5× 591 1.3× 296 0.9× 179 0.6× 56 2.3k
Tom E. Mollnes Norway 30 1.1k 0.7× 302 0.4× 322 0.7× 369 1.1× 134 0.5× 67 2.3k

Countries citing papers authored by Nada Pejnović

Since Specialization
Citations

This map shows the geographic impact of Nada Pejnović's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nada Pejnović with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nada Pejnović more than expected).

Fields of papers citing papers by Nada Pejnović

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nada Pejnović. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nada Pejnović. The network helps show where Nada Pejnović may publish in the future.

Co-authorship network of co-authors of Nada Pejnović

This figure shows the co-authorship network connecting the top 25 collaborators of Nada Pejnović. A scholar is included among the top collaborators of Nada Pejnović based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nada Pejnović. Nada Pejnović is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
2.
Jovičić, Nemanja, Nada Pejnović, Biljana Ljujić, et al.. (2021). Transgenic Overexpression of Galectin-3 in Pancreatic β Cells Attenuates Hyperglycemia in Mice: Synergistic Antidiabetic Effect With Exogenous IL-33. Frontiers in Pharmacology. 12. 714683–714683. 6 indexed citations
3.
Ljujić, Biljana, Srdjan Babić, Vera Maravić-Stojkovic, et al.. (2019). IL-33/IL-33R in various types of carotid artery atherosclerotic lesions. Cytokine. 120. 242–250. 21 indexed citations
4.
Pejnović, Nada, et al.. (2012). Effects of TNF inhibitor on innate inflammatory and Th17 cytokines in stimulated whole blood from rheumatoid arthritis patients. Inflammopharmacology. 20(6). 323–330. 7 indexed citations
5.
Milovanović, Marija, Vladislav Volarević, Gordana Radosavljević, et al.. (2012). IL-33/ST2 axis in inflammation and immunopathology. Immunologic Research. 52(1-2). 89–99. 219 indexed citations
6.
Zdravković, Nemanja, et al.. (2012). ST2 gene-deletion reveals a role of Foxp3+ regulatory T cells in diabetes modulation in BALB/c mice. Translational research. 161(2). 118–129. 7 indexed citations
7.
Radosavljević, Gordana, Vladislav Volarević, Ivan Jovanović, et al.. (2012). The roles of Galectin-3 in autoimmunity and tumor progression. Immunologic Research. 52(1-2). 100–110. 102 indexed citations
8.
Volarević, Vladislav, Marina Mitrović, Marija Milovanović, et al.. (2011). Protective role of IL-33/ST2 axis in Con A-induced hepatitis. Journal of Hepatology. 56(1). 26–33. 128 indexed citations
9.
Pejnović, Nada, Athanassios Vratimos, Sang Eun Lee, et al.. (2009). Increased atherosclerotic lesions and Th17 in interleukin-18 deficient apolipoprotein E-knockout mice fed high-fat diet. Molecular Immunology. 47(1). 37–45. 46 indexed citations
10.
Milić, Vera, Radmila Petrović, Ivan Boričić, et al.. (2009). Diagnostic Value of Salivary Gland Ultrasonographic Scoring System in Primary Sjögren’s Syndrome: A Comparison with Scintigraphy and Biopsy. The Journal of Rheumatology. 36(7). 1495–1500. 70 indexed citations
11.
Pejnović, Nada, et al.. (2008). Longitudinal study of 16 patients with Takayasu’s arteritis: clinical features and therapeutic management. Clinical Rheumatology. 28(2). 179–185. 29 indexed citations
12.
Pejnović, Nada, et al.. (2007). Refractory rapidly progressive Takayasu’s arteritis successfully treated with surgery. Clinical Rheumatology. 26(10). 1787–1789. 2 indexed citations
13.
Chan, Woon Ling, et al.. (2005). Predominance of Th2 response in human abdominal aortic aneurysm: Mistaken identity for IL-4-producing NK and NKT cells?. Cellular Immunology. 233(2). 109–114. 34 indexed citations
14.
Pasic, Srdjan, Predrag Minić, Desa Lilić, et al.. (2003). Long-Term Follow-Up and Prognosis of Chronic Granulomatous Disease in Yugoslavia: Is There a Role for Early Bone Marrow Transplantation?. Journal of Clinical Immunology. 23(1). 55–61. 8 indexed citations
15.
Chan, Woon Ling, et al.. (2001). Human IL-18 Receptor and ST2L Are Stable and Selective Markers for the Respective Type 1 and Type 2 Circulating Lymphocytes. The Journal of Immunology. 167(3). 1238–1244. 71 indexed citations
17.
18.
Kataranovski, Milena, et al.. (1994). Post-traumatic activation of draining lymph node cells II. Proliferative and phenotypic characteristics. Burns. 20(5). 403–408. 4 indexed citations
19.
Čolić, Miodrag, et al.. (1992). Interferon Gamma Alters the Phenotype of Rat Thymic Epithelial Cells in Culture and Increases Interleukin‐6Production. Journal of Immunology Research. 2(2). 151–160. 8 indexed citations
20.
Zloković, Berislav V., Dusanka S. Skundric, M. B. Segal, et al.. (1989). Blood-brain barrier permeability changes during acute allergic encephalomyelitis induced in the guinea pig. Metabolic Brain Disease. 4(1). 33–40. 14 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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