Nabil Mtiraoui

1.5k total citations
52 papers, 1.2k citations indexed

About

Nabil Mtiraoui is a scholar working on Hematology, Genetics and Immunology. According to data from OpenAlex, Nabil Mtiraoui has authored 52 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Hematology, 12 papers in Genetics and 12 papers in Immunology. Recurrent topics in Nabil Mtiraoui's work include Blood Coagulation and Thrombosis Mechanisms (11 papers), Reproductive System and Pregnancy (8 papers) and Ovarian function and disorders (8 papers). Nabil Mtiraoui is often cited by papers focused on Blood Coagulation and Thrombosis Mechanisms (11 papers), Reproductive System and Pregnancy (8 papers) and Ovarian function and disorders (8 papers). Nabil Mtiraoui collaborates with scholars based in Tunisia, Bahrain and France. Nabil Mtiraoui's co-authors include Touhami Mahjoub, Wassim Y. Almawi, Intissar Ezzidi, Walid Zammiti, M. Kacem, Molka Chaieb, Amira Turki, Ramzi R. Finan, Jean‐Christophe Gris and Martine Vaxillaire and has published in prestigious journals such as Gene, Fertility and Sterility and International Journal of Environmental Research and Public Health.

In The Last Decade

Nabil Mtiraoui

50 papers receiving 1.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Nabil Mtiraoui Tunisia 24 268 266 240 226 222 52 1.2k
Alfredo Leaños‐Miranda Mexico 23 284 1.1× 127 0.5× 239 1.0× 397 1.8× 113 0.5× 67 1.4k
Rachel T. McGrath Australia 15 153 0.6× 187 0.7× 154 0.6× 168 0.7× 171 0.8× 36 999
Winston W. Bakker Netherlands 19 242 0.9× 76 0.3× 439 1.8× 434 1.9× 49 0.2× 33 1.3k
Malu Zandbergen Netherlands 16 200 0.7× 52 0.2× 352 1.5× 85 0.4× 109 0.5× 22 905
Goichi Honda Japan 17 418 1.6× 721 2.7× 252 1.1× 34 0.2× 121 0.5× 38 1.5k
Christine Kurz Austria 20 147 0.5× 62 0.2× 177 0.7× 405 1.8× 62 0.3× 56 1.2k
Monica Attanasio Italy 20 155 0.6× 224 0.8× 155 0.6× 35 0.2× 207 0.9× 44 1.1k
Erik J. M. Toonen Netherlands 20 234 0.9× 101 0.4× 342 1.4× 19 0.1× 112 0.5× 44 1.0k
Jiřı́ Vácha Czechia 18 211 0.8× 79 0.3× 126 0.5× 39 0.2× 70 0.3× 39 873
Morito Endo Japan 20 252 0.9× 220 0.8× 574 2.4× 33 0.1× 58 0.3× 59 1.1k

Countries citing papers authored by Nabil Mtiraoui

Since Specialization
Citations

This map shows the geographic impact of Nabil Mtiraoui's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nabil Mtiraoui with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nabil Mtiraoui more than expected).

Fields of papers citing papers by Nabil Mtiraoui

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nabil Mtiraoui. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nabil Mtiraoui. The network helps show where Nabil Mtiraoui may publish in the future.

Co-authorship network of co-authors of Nabil Mtiraoui

This figure shows the co-authorship network connecting the top 25 collaborators of Nabil Mtiraoui. A scholar is included among the top collaborators of Nabil Mtiraoui based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nabil Mtiraoui. Nabil Mtiraoui is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Sarray, Sameh, et al.. (2023). Association of matrix metalloproteinase‐2 gene variants with diabetic nephropathy risk. The Journal of Gene Medicine. 25(11). e3553–e3553. 2 indexed citations
2.
Ezzidi, Intissar, et al.. (2023). Interleukin 10 (IL-10) gene variants and haplotypes in Tunisian women with polycystic ovary syndrome (PCOS): a case-control study. Molecular Biology Reports. 50(10). 8089–8096. 1 indexed citations
6.
Zaied, Chiraz, et al.. (2012). Cytochrome P450 ( CYP3A4*18 ) and Glutathione-S-Transferase ( GSTP1 ) Polymorphisms in a Healthy Tunisian Population. Genetic Testing and Molecular Biomarkers. 16(10). 1184–1187. 4 indexed citations
7.
Cauchi, Stéphane, Intissar Ezzidi, Youness El Achhab, et al.. (2012). European genetic variants associated with type 2 diabetes in North African Arabs. Diabetes & Metabolism. 38(4). 316–323. 49 indexed citations
8.
Mtiraoui, Nabil, et al.. (2012). Single-nucleotide polymorphisms and haplotypes in the adiponectin gene contribute to the genetic risk for type 2 diabetes in Tunisian Arabs. Diabetes Research and Clinical Practice. 97(2). 290–297. 39 indexed citations
9.
Turki, Amira, et al.. (2012). Lack of association between genetic polymorphisms within KCNQ1 locus and type 2 diabetes in Tunisian Arabs. Diabetes Research and Clinical Practice. 98(3). 452–458. 18 indexed citations
10.
Cherni, Lotfi, et al.. (2011). Human Alu Insertion Polymorphisms in North African Populations. Human Biology. 83(5). 611–626. 1 indexed citations
11.
Ezzidi, Intissar, Nabil Mtiraoui, Rita Nemr, et al.. (2010). Variants within the calpain-10 gene and relationships with type 2 diabetes (T2DM) and T2DM-related traits among Tunisian Arabs. Diabetes & Metabolism. 36(5). 357–362. 28 indexed citations
12.
Ezzidi, Intissar, Amira Turki, Safia A. Messaoudi, et al.. (2010). Common polymorphisms of calpain-10 and the risk of Type 2 Diabetes in a Tunisian Arab population: a case-control study. BMC Medical Genetics. 11(1). 75–75. 30 indexed citations
13.
Mtiraoui, Nabil, et al.. (2009). Predictive value of interleukin-10 promoter genotypes and haplotypes in determining the susceptibility to nephropathy in type 2 diabetes patients. 1 indexed citations
14.
Ezzidi, Intissar, Nabil Mtiraoui, Stéphane Cauchi, et al.. (2009). Contribution of type 2 diabetes associated loci in the Arabic population from Tunisia: a case-control study. BMC Medical Genetics. 10(1). 33–33. 68 indexed citations
15.
Ezzidi, Intissar, Nabil Mtiraoui, M. Kacem, et al.. (2008). Interleukin‐10–592C/A, –819C/T and –1082A/G promoter variants affect the susceptibility to nephropathy in Tunisian type 2 diabetes (T2DM) patients. Clinical Endocrinology. 70(3). 401–407. 42 indexed citations
16.
Zammiti, Walid, Nabil Mtiraoui, Ramzi R. Finan, Wassim Y. Almawi, & Touhami Mahjoub. (2008). Tumor necrosis factor α and lymphotoxin α haplotypes in idiopathic recurrent pregnancy loss. Fertility and Sterility. 91(5). 1903–1908. 26 indexed citations
17.
Mtiraoui, Nabil, Intissar Ezzidi, Molka Chaieb, et al.. (2006). MTHFR C677T and A1298C gene polymorphisms and hyperhomocysteinemia as risk factors of diabetic nephropathy in type 2 diabetes patients. Diabetes Research and Clinical Practice. 75(1). 99–106. 85 indexed citations
19.
Mtiraoui, Nabil, Walid Zammiti, Lakhdar Ghazouani, et al.. (2006). Methylenetetrahydrofolate reductase C677T and A1298C polymorphism and changes in homocysteine concentrations in women with idiopathic recurrent pregnancy losses. Reproduction. 131(2). 395–401. 74 indexed citations
20.
Mtiraoui, Nabil, Jean‐Christophe Gris, Marc Busson, et al.. (2005). Reduction in coagulation factor VII plasma levels by R353Q but not the −323P0/10 promoter polymorphism in healthy Tunisians. American Journal of Hematology. 79(1). 11–16. 16 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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