Emmanuel Vaillant

4.3k total citations
28 papers, 832 citations indexed

About

Emmanuel Vaillant is a scholar working on Genetics, Surgery and Molecular Biology. According to data from OpenAlex, Emmanuel Vaillant has authored 28 papers receiving a total of 832 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Genetics, 12 papers in Surgery and 11 papers in Molecular Biology. Recurrent topics in Emmanuel Vaillant's work include Pancreatic function and diabetes (12 papers), Genomics and Rare Diseases (6 papers) and Genetic Associations and Epidemiology (6 papers). Emmanuel Vaillant is often cited by papers focused on Pancreatic function and diabetes (12 papers), Genomics and Rare Diseases (6 papers) and Genetic Associations and Epidemiology (6 papers). Emmanuel Vaillant collaborates with scholars based in France, United Kingdom and Canada. Emmanuel Vaillant's co-authors include Philippe Froguel, Vincent Vatin, Jörg Hager, Karine Clément, Séverine Dubois, B Guy-Grand, Christian Dina, Arnaud Basdevant, Nathalie Lorentz and Stephan Francke and has published in prestigious journals such as Nature Genetics, PLoS ONE and Diabetes Care.

In The Last Decade

Emmanuel Vaillant

28 papers receiving 804 citations

Peers

Emmanuel Vaillant
Helen Freeman United Kingdom
K Hummel United States
Deborah A. Goldspink United Kingdom
Anne Gill United States
Emmanuel Vaillant
Citations per year, relative to Emmanuel Vaillant Emmanuel Vaillant (= 1×) peers Anette P. Gjesing

Countries citing papers authored by Emmanuel Vaillant

Since Specialization
Citations

This map shows the geographic impact of Emmanuel Vaillant's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Emmanuel Vaillant with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Emmanuel Vaillant more than expected).

Fields of papers citing papers by Emmanuel Vaillant

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Emmanuel Vaillant. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Emmanuel Vaillant. The network helps show where Emmanuel Vaillant may publish in the future.

Co-authorship network of co-authors of Emmanuel Vaillant

This figure shows the co-authorship network connecting the top 25 collaborators of Emmanuel Vaillant. A scholar is included among the top collaborators of Emmanuel Vaillant based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Emmanuel Vaillant. Emmanuel Vaillant is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Delplanque, Jérôme, Audrey Leloire, Emmanuel Vaillant, et al.. (2024). Monoallelic pathogenic variants in LEPR do not cause obesity. The American Journal of Human Genetics. 111(12). 2668–2674. 3 indexed citations
2.
Baron, Morgane, Emmanuel Vaillant, Aurélie Dechaume, et al.. (2024). Pathogenic, Total Loss-of-Function DYRK1B Variants Cause Monogenic Obesity Associated With Type 2 Diabetes. Diabetes Care. 47(3). 444–451. 6 indexed citations
3.
Kouidrat, Youssef, Martine Vaxillaire, Aurélie Dechaume, et al.. (2023). Dominant PDX1 deficiency causes highly penetrant diabetes at different ages, associated with obesity and exocrine pancreatic deficiency: Lessons for precision medicine. Diabetes & Metabolism. 50(1). 101507–101507. 3 indexed citations
4.
Boutry, Raphaël, Emmanuel Vaillant, Aurélie Dechaume, et al.. (2023). Pathogenic monoallelic variants in GLIS3 increase type 2 diabetes risk and identify a subgroup of patients sensitive to sulfonylureas. Diabetologia. 67(2). 327–332. 4 indexed citations
5.
Baron, Morgane, Emmanuel Vaillant, Mathilde Boissel, et al.. (2023). Contribution of heterozygous PCSK1 variants to obesity and implications for precision medicine: a case-control study. The Lancet Diabetes & Endocrinology. 11(3). 182–190. 17 indexed citations
6.
Delemer, Brigitte, Marta Spodenkiewicz, Emmanuelle Durand, et al.. (2022). Compound genetic etiology in a patient with a syndrome including diabetes, intellectual deficiency and distichiasis. Orphanet Journal of Rare Diseases. 17(1). 1 indexed citations
7.
Cornière, Nicolas, R. Brent Thomson, Bruno O. Villoutreix, et al.. (2022). Dominant negative mutation in oxalate transporter SLC26A6 associated with enteric hyperoxaluria and nephrolithiasis. Journal of Medical Genetics. 59(11). 1035–1043. 10 indexed citations
8.
Lebenthal, Yael, Naama Fisch Shvalb, Yael Gozlan, et al.. (2017). The unique clinical spectrum of maturity onset diabetes of the young type 3. Diabetes Research and Clinical Practice. 135. 18–22. 6 indexed citations
9.
Bonnefond, Amélie, Julien Philippe, Emmanuel Vaillant, et al.. (2017). A novel NEUROG3 mutation in neonatal diabetes associated with a neuro‐intestinal syndrome. Pediatric Diabetes. 19(3). 381–387. 15 indexed citations
10.
Philippe, Julien, Mehdi Derhourhi, Emmanuelle Durand, et al.. (2015). What Is the Best NGS Enrichment Method for the Molecular Diagnosis of Monogenic Diabetes and Obesity?. PLoS ONE. 10(11). e0143373–e0143373. 13 indexed citations
11.
Chandra, Vikash, Olivier Albagli, Benoît Hastoy, et al.. (2014). RFX6 Regulates Insulin Secretion by Modulating Ca2+ Homeostasis in Human β Cells. Cell Reports. 9(6). 2206–2218. 58 indexed citations
12.
Robiou-du-Pont, Sébastien, Loïc Yengo, Emmanuel Vaillant, et al.. (2013). Common variants near BDNF and SH2B1 show nominal evidence of association with snacking behavior in European populations. Journal of Molecular Medicine. 91(9). 1109–1115. 4 indexed citations
13.
Bonnefond, Amélie, Emmanuel Vaillant, Julien Philippe, et al.. (2013). Transcription factor gene MNX1 is a novel cause of permanent neonatal diabetes in a consanguineous family. Diabetes & Metabolism. 39(3). 276–280. 38 indexed citations
14.
Bonnefond, Amélie, Loïc Yengo, Julien Philippe, et al.. (2012). Reassessment of the putative role of BLK-p.A71T loss-of-function mutation in MODY and type 2 diabetes. Diabetologia. 56(3). 492–496. 31 indexed citations
15.
Boutant, Marie, Oscar Henrique Pereira Ramos, Cécile Lecœur, et al.. (2012). Glucose-Dependent Regulation of NR2F2 Promoter and Influence of SNP-rs3743462 on Whole Body Insulin Sensitivity. PLoS ONE. 7(5). e35810–e35810. 10 indexed citations
16.
Robiou-du-Pont, Sébastien, Amélie Bonnefond, Loïc Yengo, et al.. (2012). Contribution of 24 obesity-associated genetic variants to insulin resistance, pancreatic beta-cell function and type 2 diabetes risk in the French population. International Journal of Obesity. 37(7). 980–985. 45 indexed citations
17.
Walley, Andrew J., Peter Jacobson, Mario Falchi, et al.. (2011). Differential coexpression analysis of obesity-associated networks in human subcutaneous adipose tissue. International Journal of Obesity. 36(1). 137–147. 36 indexed citations
18.
Ezzidi, Intissar, Nabil Mtiraoui, Stéphane Cauchi, et al.. (2009). Contribution of type 2 diabetes associated loci in the Arabic population from Tunisia: a case-control study. BMC Medical Genetics. 10(1). 33–33. 68 indexed citations
19.
Eyre, Stephen, Hassen Kamoun, Hajer Ennafaa, et al.. (2008). Effect of ENPP1/PC-1-K121Q and PPARγ-Pro12Ala polymorphisms on the genetic susceptibility to T2D in the Tunisian population. Diabetes Research and Clinical Practice. 81(3). 278–283. 31 indexed citations
20.
Hager, Jörg, Christian Dina, Stephan Francke, et al.. (1998). A genome-wide scan for human obesity genes reveals a major susceptibility locus on chromosome 10. Nature Genetics. 20(3). 304–308. 292 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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