Н. К. Исаев
- Molecular Biology top 5%
- Cellular and Molecular Neuroscience top 2%
- Physiology top 5%
- Developmental Neuroscience top 1%
- Pathology and Forensic Medicine top 2%
- Co-authors
- E. V. StelmashookDmitry B. ZorovUlrich DirnaglЕ. Е. ГенрихсKarsten RuscherAndreas MeiselDirk MegowEgor Y. Plotnikov
- Topics
- Neuroscience and Neuropharmacology Research (44 papers)Mitochondrial Function and Pathology (31 papers)Anesthesia and Neurotoxicity Research (17 papers)
- Journals
- Journal of NeuroscienceSHILAP Revista de lepidopterologíaPLoS ONE
- Partner nations
- RussiaTajikistanGermany
In The Last Decade
Н. К. Исаев
113 papers receiving 3.6k citations
Peers
Comparison fields: 5 of 129
- Molecular Biology 1.5k
- Cellular and Molecular Neuroscience 793
- Physiology 619
- Developmental Neuroscience 508
- Pathology and Forensic Medicine 468
Countries citing papers authored by Н. К. Исаев
This map shows the geographic impact of Н. К. Исаев's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Н. К. Исаев with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Н. К. Исаев more than expected).
Fields of papers citing papers by Н. К. Исаев
This network shows the impact of papers produced by Н. К. Исаев. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Н. К. Исаев. The network helps show where Н. К. Исаев may publish in the future.
Co-authorship network of co-authors of Н. К. Исаев
This figure shows the co-authorship network connecting the top 25 collaborators of Н. К. Исаев. A scholar is included among the top collaborators of Н. К. Исаев based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Н. К. Исаев. Н. К. Исаев is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 3 | |
| 2 | 16 | |
| 3 | 0 | |
| 4 | 27 | |
| 5 | 5 | |
| 6 | 12 | |
| 7 | 56 | |
| 8 | 38 | |
| 9 | 32 | |
| 10 | 14 | |
| 11 | 48 | |
| 12 | 13 | |
| 13 | 19 | |
| 14 | 102 | |
| 15 | 29 | |
| 16 | 185 | |
| 17 | 50 | |
| 18 | 35 | |
| 19 | 86 | |
| 20 | 39 |
About Н. К. Исаев
Н. К. Исаев is a scholar working on Developmental Neuroscience, Cellular and Molecular Neuroscience and Neurology, having authored 118 papers that have together received 3.6k indexed citations. Recurring topics across this work include Neuroscience and Neuropharmacology Research (44 papers), Mitochondrial Function and Pathology (31 papers) and Anesthesia and Neurotoxicity Research (17 papers). The work is most often cited by research in Developmental Neuroscience (508 citations), Neurology (460 citations) and Cellular and Molecular Neuroscience (793 citations). Н. К. Исаев has collaborated with scholars based in Russia, Tajikistan and Germany. Frequent co-authors include E. V. Stelmashook, Dmitry B. Zorov, Ulrich Dirnagl, Е. Е. Генрихс, Karsten Ruscher, Andreas Meisel, Dirk Megow, Egor Y. Plotnikov, Josef Priller and Dorette Freyer. Their work appears in journals such as Journal of Neuroscience, SHILAP Revista de lepidopterología and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.