Muling Mao
- Molecular Biology top 5%
- PI3K/AKT/mTOR signaling in cancer 5
- Protein Kinase Regulation and GTPase Signaling 4
- Sphingolipid Metabolism and Signaling 4
- Cell death mechanisms and regulation 2
- Retinal Development and Disorders 2
- bioluminescence and chemiluminescence research 2
- Peroxisome Proliferator-Activated Receptors 2
- Oncology top 5%
- Colorectal Cancer Treatments and Studies 3
- Biochemistry top 5%
- Cancer Research top 10%
- Cell Biology top 5%
Muling Mao
16 papers receiving 1.7k citations
Peers
Comparison fields: 5 of 94
- Molecular Biology 1.3k
- Oncology 501
- Biochemistry 108
- Cancer Research 222
- Cell Biology 227
Countries citing papers authored by Muling Mao
This map shows the geographic impact of Muling Mao's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Muling Mao with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Muling Mao more than expected).
Fields of papers citing papers by Muling Mao
This network shows the impact of papers produced by Muling Mao. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Muling Mao. The network helps show where Muling Mao may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Muling Mao, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2013 | 99 | |
| 2 | 2012 | 235 | |
| 3 | 2011 | 1 | |
| 4 | 2004 | 23 | |
| 5 | 2003 | 21 | |
| 6 | 2003 | 25 | |
| 7 | 2001 | 22 | |
| 8 | 2001 | 212 | |
| 9 | 2001 | 12 | |
| 10 | 2000 | 44 | |
| 11 | 2000 | 211 | |
| 12 | 2000 | 60 | |
| 13 | 1999 | 236 | |
| 14 | 1999 | 262 | |
| 15 | Lysophosphatidic acid induces urokinase secretion by ovarian cancer cells. | 1999 | 137 |
| 16 | Overexpression of edg-2/vzg-1 induces apoptosis and anoikis in ovarian cancer cells in a lysophosphatidic acid-independent manner. | 1999 | 102 |
About Muling Mao
Muling Mao is a scholar working on Biochemistry, Molecular Biology, Oncology, Reproductive Medicine and Biotechnology, having authored 16 papers that have together received 1.7k indexed citations. Recurring topics across this work include PI3K/AKT/mTOR signaling in cancer (5 papers), Protein Kinase Regulation and GTPase Signaling (4 papers), Sphingolipid Metabolism and Signaling (4 papers), Colorectal Cancer Treatments and Studies (3 papers), Cell death mechanisms and regulation (2 papers), Retinal Development and Disorders (2 papers), bioluminescence and chemiluminescence research (2 papers) and Peroxisome Proliferator-Activated Receptors (2 papers). The work is most often cited by research in Molecular Biology (1.3k citations), Oncology (501 citations), Biochemistry (108 citations), Cancer Research (222 citations) and Cell Biology (227 citations). Muling Mao has collaborated with scholars based in United States, Australia and Canada. Frequent co-authors include Gordon B. Mills, Xianjun Fang, Robert C. Bast, Yiling Lu, Ruth LaPushin, Astrid Eder, Kathy Siminovitch, Bruce D. Cuevas, Tatsuro Furui and Shuangxing Yu. Their work appears in journals such as Journal of Biological Chemistry, Biochemical Journal, Oncogene, Prostaglandins & Other Lipid Mediators and Clinical Cancer Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.