Motohiko Kadoki

1.7k total citations · 1 hit paper
14 papers, 1.4k citations indexed

About

Motohiko Kadoki is a scholar working on Molecular Biology, Immunology and Genetics. According to data from OpenAlex, Motohiko Kadoki has authored 14 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 7 papers in Immunology and 4 papers in Genetics. Recurrent topics in Motohiko Kadoki's work include HIV Research and Treatment (3 papers), Immune Response and Inflammation (3 papers) and RNA Interference and Gene Delivery (2 papers). Motohiko Kadoki is often cited by papers focused on HIV Research and Treatment (3 papers), Immune Response and Inflammation (3 papers) and RNA Interference and Gene Delivery (2 papers). Motohiko Kadoki collaborates with scholars based in Japan, United States and China. Motohiko Kadoki's co-authors include Yoichiro Iwakura, Shigeru Kakuta, Susumu Nakae, Harumichi Ishigame, Hayato Kotaki, Takeshi Nagai, Katsuko Sudo, Aya Nambu, Chihiro Sasakawa and Noriyuki Fujikado and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Nature Communications.

In The Last Decade

Motohiko Kadoki

14 papers receiving 1.4k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Differential Roles of Interleukin-17A and -17F in Host Defense against Mucoepithelial Bacterial Infection and Allergic Responses

2009 813 citations Harumichi Ishigame, Shigeru Kakuta et al. Immunity profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Motohiko Kadoki Japan	9	852	402	236	163	147	14	1.4k
Toni M.M. van Capel Netherlands	15	915 1.1×	529 1.3×	171 0.7×	175 1.1×	111 0.8×	23	1.8k
Ce Tang Japan	15	511 0.6×	428 1.1×	162 0.7×	119 0.7×	150 1.0×	28	1.1k
Joana F. Neves United Kingdom	15	1.1k 1.3×	571 1.4×	176 0.7×	115 0.7×	189 1.3×	30	1.8k
Meimei Shan United States	8	963 1.1×	663 1.6×	268 1.1×	160 1.0×	138 0.9×	14	1.8k
Leticia Monin United States	15	775 0.9×	288 0.7×	507 2.1×	366 2.2×	99 0.7×	19	1.4k
Carlene L. Zindl United States	14	1.3k 1.5×	426 1.1×	159 0.7×	203 1.2×	219 1.5×	20	1.9k
Zhikang Peng Canada	25	663 0.8×	390 1.0×	164 0.7×	148 0.9×	229 1.6×	53	1.9k
Liliane Martins dos Santos Brazil	16	1.1k 1.3×	568 1.4×	226 1.0×	293 1.8×	193 1.3×	29	1.9k
Natasha Whibley United States	15	795 0.9×	265 0.7×	394 1.7×	320 2.0×	64 0.4×	18	1.3k
Hill Gaston United Kingdom	22	721 0.8×	429 1.1×	217 0.9×	349 2.1×	105 0.7×	38	1.5k

Countries citing papers authored by Motohiko Kadoki

Since Specialization

Citations

This map shows the geographic impact of Motohiko Kadoki's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Motohiko Kadoki with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Motohiko Kadoki more than expected).

Fields of papers citing papers by Motohiko Kadoki

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Motohiko Kadoki. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Motohiko Kadoki. The network helps show where Motohiko Kadoki may publish in the future.

Co-authorship network of co-authors of Motohiko Kadoki

This figure shows the co-authorship network connecting the top 25 collaborators of Motohiko Kadoki. A scholar is included among the top collaborators of Motohiko Kadoki based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Motohiko Kadoki. Motohiko Kadoki is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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14 of 14 papers shown

Lee, Seoung Rak, Motohiko Kadoki, Gleb Pishchany, et al.. (2025). Elucidating the role of Campylobacter concisus –derived indole metabolites in gut inflammation and immune modulation. Proceedings of the National Academy of Sciences. 122(34). e2514071122–e2514071122. 1 indexed citations

Tang, Ce, Haiyang Sun, Motohiko Kadoki, et al.. (2023). Blocking Dectin-1 prevents colorectal tumorigenesis by suppressing prostaglandin E2 production in myeloid-derived suppressor cells and enhancing IL-22 binding protein expression. Nature Communications. 14(1). 1493–1493. 44 indexed citations

Kalam, Haroon, Chih‐Hung Chou, Motohiko Kadoki, et al.. (2023). Identification of host regulators of Mycobacterium tuberculosis phenotypes uncovers a role for the MMGT1-GPR156 lipid droplet axis in persistence. Cell Host & Microbe. 31(6). 978–992.e5. 7 indexed citations

Nakata, Toru, Elizabeth A. Creasey, Motohiko Kadoki, et al.. (2020). A missense variant in SLC39A8 confers risk for Crohn’s disease by disrupting manganese homeostasis and intestinal barrier integrity. Proceedings of the National Academy of Sciences. 117(46). 28930–28938. 32 indexed citations

Varma, Mukund, Motohiko Kadoki, Ariel Lefkovith, et al.. (2020). Cell Type– and Stimulation-Dependent Transcriptional Programs Regulated by Atg16L1 and Its Crohn's Disease Risk Variant T300A. The Journal of Immunology. 205(2). 414–424. 6 indexed citations

Tang, Ce, Shigeru Kakuta, Kenji Shimizu, et al.. (2018). Suppression of IL-17F, but not of IL-17A, provides protection against colitis by inducing Treg cells through modification of the intestinal microbiota. Nature Immunology. 19(7). 755–765. 132 indexed citations

Kamiya, Tomonori, Ce Tang, Motohiko Kadoki, et al.. (2017). β-Glucans in food modify colonic microflora by inducing antimicrobial protein, calprotectin, in a Dectin-1-induced-IL-17F-dependent manner. Mucosal Immunology. 11(3). 763–773. 37 indexed citations

Kadoki, Motohiko, Ashwini Patil, Donald Brooks, et al.. (2017). Organism-Level Analysis of Vaccination Reveals Networks of Protection across Tissues. Cell. 171(2). 398–413.e21. 63 indexed citations

Tang, Ce, Tomonori Kamiya, Yang Liu, et al.. (2015). Inhibition of Dectin-1 Signaling Ameliorates Colitis by Inducing Lactobacillus-Mediated Regulatory T Cell Expansion in the Intestine. Cell Host & Microbe. 18(2). 183–197. 221 indexed citations

10.

Tada, Takuya, Motohiko Kadoki, Yang Liu, Kenzo Tokunaga, & Yoichiro Iwakura. (2013). Transgenic expression of the human LEDGF/p75 gene relieves the species barrier against HIV-1 infection in mouse cells. Frontiers in Microbiology. 4. 377–377. 1 indexed citations

11.

Tang, Ce, Tomonori Kamiya, Motohiko Kadoki, & Yoichiro Iwakura. (2012). O040 A novel role of Dectin-1 signaling in promoting intestinal inflammation. Cytokine. 59(3). 516–516. 1 indexed citations

12.

Kadoki, Motohiko, Byung-il Choi, & Yoichiro Iwakura. (2010). The mechanism of LPS-induced HIV type I activation in transgenic mouse macrophages. International Immunology. 22(6). 469–478. 9 indexed citations

13.

Ishigame, Harumichi, Shigeru Kakuta, Takeshi Nagai, et al.. (2009). Differential Roles of Interleukin-17A and -17F in Host Defense against Mucoepithelial Bacterial Infection and Allergic Responses. Immunity. 30(1). 108–119. 813 indexed citations breakdown →

14.

Tsurutani, Naomi, Jiro Yasuda, Naoki Yamamoto, et al.. (2006). Nuclear Import of the Preintegration Complex Is Blocked upon Infection by Human Immunodeficiency Virus Type 1 in Mouse Cells. Journal of Virology. 81(2). 677–688. 25 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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